X-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cells - Wikidata - awgldk - TriplyDB

X-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cells

X-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cells

http://www.wikidata.org/entity/Q24676168

about

Characterization of SH2D1A missense mutations identified in X-linked lymphoproliferative disease patients Structural basis for the interaction of the free SH2 domain EAT-2 with SLAM receptors in hematopoietic cells Early expression of triggering receptors and regulatory role of 2B4 in human natural killer cell precursors undergoing in vitro differentiation.Progress and problems in understanding and managing primary Epstein-Barr virus infections NTB-A [correction of GNTB-A], a novel SH2D1A-associated surface molecule contributing to the inability of natural killer cells to kill Epstein-Barr virus-infected B cells in X-linked lymphoproliferative disease The molecular pathology of primary immunodeficiencies The interplay between Epstein-Barr virus and B lymphocytes: implications for infection, immunity, and disease Structure of Natural Killer Receptor 2B4 Bound to CD48 Reveals Basis for Heterophilic Recognition in Signaling Lymphocyte Activation Molecule Family X-linked lymphoproliferative syndromes: brothers or distant cousins?NK Cell Influence on the Outcome of Primary Epstein-Barr Virus Infection Influence of CRACC, a SLAM family receptor coupled to the adaptor EAT-2, on natural killer cell function Regulation of SLAM-mediated signal transduction by SAP, the X-linked lymphoproliferative gene product Dual functional roles for the X-linked lymphoproliferative syndrome gene product SAP/SH2D1A in signaling through the signaling lymphocyte activation molecule (SLAM) family of immune receptors A spectrum of mutations in SH2D1A that causes X-linked lymphoproliferative disease and other Epstein-Barr virus-associated illnesses Association of the X-linked lymphoproliferative disease gene product SAP/SH2D1A with 2B4, a natural killer cell-activating molecule, is dependent on phosphoinositide 3-kinase Negative regulation of natural killer cell function by EAT-2, a SAP-related adaptor Kinome analysis of receptor-induced phosphorylation in human natural killer cells Up on the tightrope: natural killer cell activation and inhibition Occurrence of nodular lymphocyte-predominant hodgkin lymphoma in hermansky-pudlak type 2 syndrome is associated to natural killer and natural killer T cell defects.NK cell cytotoxicity mediated by 2B4 and NTB-A is dependent on SAP acting downstream of receptor phosphorylation.Tonsilar NK cells restrict B cell transformation by the Epstein-Barr virus via IFN-gamma.Expansion of murine gammaherpesvirus latently infected B cells requires T follicular help.CD244 maintains the proliferation ability of leukemia initiating cells through SHP-2/p27kip1 signaling.Crosstalk between decidual NK and CD14+ myelomonocytic cells results in induction of Tregs and immunosuppression.Molecular pathogenesis of EBV susceptibility in XLP as revealed by analysis of female carriers with heterozygous expression of SAP.On guard--activating NK cell receptors.TGF-β1 down-regulation of NKG2D/DAP10 and 2B4/SAP expression on human NK cells contributes to HBV persistence A dual activation and inhibition role for the paired immunoglobulin-like receptor B in eosinophils Molecular dissection of 2B4 signaling: implications for signal transduction by SLAM-related receptors.Pathogenesis of haemophagocytic lymphohistiocytosis.From Burkitt's lymphoma to chronic active Epstein-Barr virus (EBV) infection: an expanding spectrum of EBV-associated diseases.XMEN disease: a new primary immunodeficiency affecting Mg2+ regulation of immunity against Epstein-Barr virus.Natural killer cells: a mystery no more.Genetic control of innate immune responses against cytomegalovirus: MCMV meets its match.X-linked lymphoproliferative disease: clinical, diagnostic and molecular perspective.Natural killer cells from patients with chronic rhinosinusitis have impaired effector functions Molecular and immunological basis of X-linked lymphoproliferative disease.New approaches in the immunotherapy of haematological malignancies.Negative Regulation of Humoral Immunity Due to Interplay between the SLAMF1, SLAMF5, and SLAMF6 Receptors.Unravelling natural killer cell function: triggering and inhibitory human NK receptors.

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X-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cells

http://www.wikidata.org/entity/Q24676168

description

2000 nî lūn-bûn

@nan

2000 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

@hyw

2000 թվականի օգոստոսին հրատարակված գիտական հոդված

@hy

2000年の論文

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2000年論文

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2000年論文

@zh-hant

2000年論文

@zh-hk

2000年論文

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2000年論文

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2000年论文

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name

X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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Journal of Experimental Medicine

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X-linked lymphoproliferative d ...... tein-Barr virus-infected cells

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A Moretta

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H D Ochs

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H Wolf

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R Franceschini

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S Giliani

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P2860

The X-linked lymphoproliferative-disease gene product SAP regulates signals induced through the co-receptor SLAM

NKp44, a triggering receptor involved in tumor cell lysis by activated human natural killer cells, is a novel member of the immunoglobulin superfamily

Cutting edge: human 2B4, an activating NK cell receptor, recruits the protein tyrosine phosphatase SHP-2 and the adaptor signaling protein SAP

Molecular and functional characterization of IRp60, a member of the immunoglobulin superfamily that functions as an inhibitory receptor in human NK cells

Molecular cloning of NKp46: a novel member of the immunoglobulin superfamily involved in triggering of natural cytotoxicity

Identification of a novel signal transduction surface molecule on human cytotoxic lymphocytes

A novel receptor involved in T-cell activation

Down-regulation of class I HLA antigens and of the Epstein-Barr virus-encoded latent membrane protein in Burkitt lymphoma lines

2B4, the natural killer and T cell immunoglobulin superfamily surface protein, is a ligand for CD48

NKp44, a novel triggering surface molecule specifically expressed by activated natural killer cells, is involved in non-major histocompatibility complex-restricted tumor cell lysis

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1368734

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10.1084/JEM.192.3.337

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3

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192

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Roberto Biassoni

Cristina Bottino

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12386897

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Epstein–Barr virus

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2193227

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