Overexpression of PIAS3 suppresses cell growth and restores the drug sensitivity of human lung cancer cells in association with PI3-K/Akt inactivation - Wikidata - awgldk - TriplyDB

Overexpression of PIAS3 suppresses cell growth and restores the drug sensitivity of human lung cancer cells in association with PI3-K/Akt inactivation

Overexpression of PIAS3 suppresses cell growth and restores the drug sensitivity of human lung cancer cells in association with PI3-K/Akt inactivation

http://www.wikidata.org/entity/Q24678516

about

Loss of protein inhibitors of activated STAT-3 expression in glioblastoma multiforme tumors: implications for STAT-3 activation and gene expression Suppressor of cytokine signaling (SOCS)-1 is expressed in human prostate cancer and exerts growth-inhibitory function through down-regulation of cyclins and cyclin-dependent kinases Neoplasia: Where We Have Been and Where We Are Going Metabolic patterns and biotransformation activities of resveratrol in human glioblastoma cells: relevance with therapeutic efficacies The PINIT domain of PIAS3: structure-function analysis of its interaction with STAT3 Neoplasia: the second decade.Expression of STAT5, COX-2 and PIAS3 in correlation with NSCLC histhopathological features.Coactivation of SF-1-mediated transcription of steroidogenic enzymes by Ubc9 and PIAS1 Protein inhibitor of activated STAT3 expression in lung cancer.Brassinin inhibits STAT3 signaling pathway through modulation of PIAS-3 and SOCS-3 expression and sensitizes human lung cancer xenograft in nude mice to paclitaxel Prognostic value of protein inhibitor of activated STAT3 in breast cancer patients receiving hormone therapy.Epigenetic regulation of signal transducer and activator of transcription 3 in acute myeloid leukemia Signal transducer and activator of transcription-3: a molecular hub for signaling pathways in gliomas The association and nuclear translocation of the PIAS3-STAT3 complex is ligand and time dependent.STAT3 in Cancer-Friend or Foe?A STAT inhibitor patent review: progress since 2011.3-Formylchromone inhibits proliferation and induces apoptosis of multiple myeloma cells by abrogating STAT3 signaling through the induction of PIAS3.Cucurmin, anticancer, & antitumor perspectives: A comprehensive review.Lung Adenocarcinomas and Lung Cancer Cell Lines Show Association of MMP-1 Expression With STAT3 Activation.Utility of network integrity methods in therapeutic target identification.Neoplasia: An Anniversary of Progress.STAT3/PIAS3 levels serve as "early signature" genes in the development of high-grade serous carcinoma from the fallopian tube.Nuclear Smad6 promotes gliomagenesis by negatively regulating PIAS3-mediated STAT3 inhibition.

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P2860

Overexpression of PIAS3 suppresses cell growth and restores the drug sensitivity of human lung cancer cells in association with PI3-K/Akt inactivation

http://www.wikidata.org/entity/Q24678516

description

2006 nî lūn-bûn

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2006 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2006 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Overexpression of PIAS3 suppre ...... on with PI3-K/Akt inactivation

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Ichiro Kawase

http://www.w3.org/2001/XMLSchema#string

Isao Tachibana

http://www.w3.org/2001/XMLSchema#string

Izumi Nagatomo

http://www.w3.org/2001/XMLSchema#string

Kota Iwahori

http://www.w3.org/2001/XMLSchema#string

Mitsugi Furukawa

http://www.w3.org/2001/XMLSchema#string

Mitsuhiro Yoshida

http://www.w3.org/2001/XMLSchema#string

Tadashi Osaki

http://www.w3.org/2001/XMLSchema#string

Takashi Kijima

http://www.w3.org/2001/XMLSchema#string

Tetsuji Naka

http://www.w3.org/2001/XMLSchema#string

Toru Kumagai

http://www.w3.org/2001/XMLSchema#string

P2860

The role of STATs in apoptosis

A family of cytokine-inducible inhibitors of signalling

A new protein containing an SH2 domain that inhibits JAK kinases

Activating mutations in the epidermal growth factor receptor underlying responsiveness of non-small-cell lung cancer to gefitinib

Interleukin-3-induced phosphorylation of BAD through the protein kinase Akt

Specific inhibition of Stat3 signal transduction by PIAS3

Differential PIAS3 expression in human malignancy

Gefitinib-sensitizing EGFR mutations in lung cancer activate anti-apoptotic pathways

Regulation of cell death protease caspase-9 by phosphorylation

Structure and function of a new STAT-induced STAT inhibitor

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817-25

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scholarly article

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10.1593/NEO.06409

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10

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8

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6761766

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17032498

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1715929

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