Loss of protein inhibitors of activated STAT-3 expression in glioblastoma multiforme tumors: implications for STAT-3 activation and gene expression - Wikidata - awgldk - TriplyDB

Loss of protein inhibitors of activated STAT-3 expression in glioblastoma multiforme tumors: implications for STAT-3 activation and gene expression

Loss of protein inhibitors of activated STAT-3 expression in glioblastoma multiforme tumors: implications for STAT-3 activation and gene expression

http://www.wikidata.org/entity/Q24619748

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SUMOylation of hnRNP-K is required for p53-mediated cell-cycle arrest in response to DNA damage Regulation of Natural Killer Cell Function by STAT3 NF-κB and STAT3 in glioblastoma: therapeutic targets coming of age A role for G-CSF and GM-CSF in nonmyeloid cancers Role of interleukin-6 in Barrett's esophagus pathogenesis Astrocytes: biology and pathology The impact of the Cancer Genome Atlas on lung cancer.Metabolic patterns and biotransformation activities of resveratrol in human glioblastoma cells: relevance with therapeutic efficacies PIAS3 expression in squamous cell lung cancer is low and predicts overall survival.The role of CXC-chemokine receptor CXCR2 and suppressor of cytokine signaling-3 (SOCS-3) in renal cell carcinoma.SOCS1 and SOCS3 in the control of CNS immunity Immunobiological characterization of cancer stem cells isolated from glioblastoma patients.Stat3-positive tumor cells contribute to vessels neoformation in primary central nervous system lymphoma Expression of STAT5, COX-2 and PIAS3 in correlation with NSCLC histhopathological features.Glioblastoma cancer-initiating cells inhibit T-cell proliferation and effector responses by the signal transducers and activators of transcription 3 pathway Short hairpin RNA-mediated fibronectin knockdown delays tumor growth in a mouse glioma model.PIAS3 activates the intrinsic apoptotic pathway in non-small cell lung cancer cells independent of p53 status.Nucleo-cytoplasmic trafficking of TRIM8, a novel oncogene, is involved in positive regulation of TNF induced NF-κB pathway.STAT-Related Profiles Are Associated with Patient Response to Targeted Treatments in Locally Advanced SCCHN.Protein inhibitor of activated STAT3 expression in lung cancer.PIAS1 is a crucial factor for prostate cancer cell survival and a valid target in docetaxel resistant cells Oncostatin-M differentially regulates mesenchymal and proneural signature genes in gliomas via STAT3 signaling.Identification of STAT3-independent regulatory effects for protein inhibitor of activated STAT3 by binding to novel transcription factors.Differential roles of STAT3 in the initiation and growth of lung cancer Transcriptional network analysis reveals that AT1 and AT2 angiotensin II receptors are both involved in the regulation of genes essential for glioma progression.Sorafenib exerts anti-glioma activity in vitro and in vivo.Fad104, a positive regulator of adipocyte differentiation, suppresses invasion and metastasis of melanoma cells by inhibition of STAT3 activity.Therapeutic potential of AZD1480 for the treatment of human glioblastoma Brassinin inhibits STAT3 signaling pathway through modulation of PIAS-3 and SOCS-3 expression and sensitizes human lung cancer xenograft in nude mice to paclitaxel Expression of interleukin-8 receptor CXCR2 and suppressor of cytokine signaling-3 in astrocytic tumors.Human leukocyte antigen-G is frequently expressed in glioblastoma and may be induced in vitro by combined 5-aza-2'-deoxycytidine and interferon-γ treatments: results from a multicentric study.BIRC3 is a novel driver of therapeutic resistance in Glioblastoma.Small ubiquitin-like modifier 1-3 conjugation [corrected] is activated in human astrocytic brain tumors and is required for glioblastoma cell survival.Activation of the NF-κB pathway by the STAT3 inhibitor JSI-124 in human glioblastoma cells.The role of JAK-STAT signaling within the CNS.Sox11 expression in astrocytic gliomas: correlation with nestin/c-Met/IDH1-R132H expression phenotypes, p-Stat-3 and survival NF-κB-induced IL-6 ensures STAT3 activation and tumor aggressiveness in glioblastoma Nuclear factor one B (NFIB) encodes a subtype-specific tumour suppressor in glioblastoma.The association and nuclear translocation of the PIAS3-STAT3 complex is ligand and time dependent.NF-kappaB and STAT3 signaling in glioma: targets for future therapies

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Loss of protein inhibitors of activated STAT-3 expression in glioblastoma multiforme tumors: implications for STAT-3 activation and gene expression

http://www.wikidata.org/entity/Q24619748

description

2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年学术文章

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2008年学术文章

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Clinical Cancer Research

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Loss of protein inhibitors of ...... activation and gene expression

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Cheryl Ann Palmer

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Emily C Brantley

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Etty N Benveniste

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G Yancey Gillespie

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Keith Harrison

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Kevin Roarty

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L Burton Nabors

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Youn-Hee Choi

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P2860

PIAS3 suppresses NF-kappaB-mediated transcription by interacting with the p65/RelA subunit

Principles of interleukin (IL)-6-type cytokine signalling and its regulation

Disruption of parallel and converging signaling pathways contributes to the synergistic antitumor effects of simultaneous mTOR and EGFR inhibition in GBM cells

Identification of a PTEN-regulated STAT3 brain tumor suppressor pathway

Overexpression of PIAS3 suppresses cell growth and restores the drug sensitivity of human lung cancer cells in association with PI3-K/Akt inactivation

Knockdown of STAT3 expression by RNAi induces apoptosis in astrocytoma cells

Nitric oxide destabilizes Pias3 and regulates sumoylation

Distinct effects of PIAS proteins on androgen-mediated gene activation in prostate cancer cells

Differential PIAS3 expression in human malignancy

Regulation of gene-activation pathways by PIAS proteins in the immune system

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15

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