Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
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Salmonella enterica serovars Typhimurium and Typhi as model organisms: revealing paradigm of host-pathogen interactionsThe Dynamic Interactions between Salmonella and the Microbiota, within the Challenging Niche of the Gastrointestinal TractIntraspecies competition for niches in the distal gut dictate transmission during persistent Salmonella infectionTranscriptional priming of Salmonella Pathogenicity Island-2 precedes cellular invasionRole of T3SS-1 SipD Protein in Protecting Mice against Non-typhoidal Salmonella TyphimuriumThe SseC translocon component in Salmonella enterica serovar Typhimurium is chaperoned by SscAA novel secretion pathway of Salmonella enterica acts as an antivirulence modulator during salmonellosisA naturally occurring single nucleotide polymorphism in the Salmonella SPI-2 type III effector srfH/sseI controls early extraintestinal disseminationSystemic translocation of Salmonella enterica serovar Dublin in cattle occurs predominantly via efferent lymphatics in a cell-free niche and requires type III secretion system 1 (T3SS-1) but not T3SS-2Coordinated regulation of virulence during systemic infection of Salmonella enterica serovar Typhimurium.RpoE fine tunes expression of a subset of SsrB-regulated virulence factors in Salmonella enterica serovar Typhimurium.Identification of the regulatory logic controlling Salmonella pathoadaptation by the SsrA-SsrB two-component system.Muc2 protects against lethal infectious colitis by disassociating pathogenic and commensal bacteria from the colonic mucosa.The SdiA-regulated gene srgE encodes a type III secreted effector.Global analysis of the eukaryotic pathways and networks regulated by Salmonella typhimurium in mouse intestinal infection in vivo.Analysis of the expression, secretion and translocation of the Salmonella enterica type III secretion system effector SteAEffect of the Salmonella pathogenicity island 2 type III secretion system on Salmonella survival in activated chicken macrophage-like HD11 cells.A fundamental regulatory mechanism operating through OmpR and DNA topology controls expression of Salmonella pathogenicity islands SPI-1 and SPI-2Polyamines are required for virulence in Salmonella enterica serovar Typhimurium.Nramp1 expression by dendritic cells modulates inflammatory responses during Salmonella Typhimurium infection.Salmonella phage ST64B encodes a member of the SseK/NleB effector familyIntegration of a complex regulatory cascade involving the SirA/BarA and Csr global regulatory systems that controls expression of the Salmonella SPI-1 and SPI-2 virulence regulons through HilDPopulation dynamics of Salmonella enterica serotypes in commercial egg and poultry productionSalmonella typhimurium disseminates within its host by manipulating the motility of infected cells.A comprehensive study of the contribution of Salmonella enterica serovar Typhimurium SPI2 effectors to bacterial colonization, survival, and replication in typhoid fever, macrophage, and epithelial cell infection models.Bistable expression of CsgD in Salmonella enterica serovar Typhimurium connects virulence to persistence.SseL is a salmonella-specific translocated effector integrated into the SsrB-controlled salmonella pathogenicity island 2 type III secretion system.Environmental mimics and the Lvh type IVA secretion system contribute to virulence-related phenotypes of Legionella pneumophila.Altered levels of Salmonella DNA adenine methylase are associated with defects in gene expression, motility, flagellar synthesis, and bile resistance in the pathogenic strain 14028 but not in the laboratory strain LT2.RNA-seq Brings New Insights to the Intra-Macrophage Transcriptome of Salmonella TyphimuriumThe two-component system CpxR/A represses the expression of Salmonella virulence genes by affecting the stability of the transcriptional regulator HilDNet replication of Salmonella enterica serovars Typhimurium and Choleraesuis in porcine intestinal mucosa and nodes is associated with their differential virulence.Host transmission of Salmonella enterica serovar Typhimurium is controlled by virulence factors and indigenous intestinal microbiota.Red Seaweeds Sarcodiotheca gaudichaudii and Chondrus crispus down Regulate Virulence Factors of Salmonella Enteritidis and Induce Immune Responses in Caenorhabditis elegans.HilD-mediated transcriptional cross-talk between SPI-1 and SPI-2.Genomic islands: tools of bacterial horizontal gene transfer and evolutionSalmonella enterica serovar typhimurium exploits Toll-like receptor signaling during the host-pathogen interaction.Spatial segregation of virulence gene expression during acute enteric infection with Salmonella enterica serovar TyphimuriumType III effector-mediated processes in Salmonella infection.Toll-like receptor-deficient mice reveal how innate immune signaling influences Salmonella virulence strategies
P2860
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P2860
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
description
2005 nî lūn-bûn
@nan
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine
@nl
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@ast
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@en
type
label
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine
@nl
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@ast
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@en
prefLabel
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine
@nl
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@ast
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@en
P2093
P2860
P50
P3181
P1433
P1476
Salmonella pathogenicity island 2 is expressed prior to penetrating the intestine.
@en
P2093
B Brett Finlay
Nat F Brown
Yanet Valdez
P2860
P3181
P356
10.1371/JOURNAL.PPAT.0010032
P407
P577
2005-11-18T00:00:00Z