Salmonella typhimurium disseminates within its host by manipulating the motility of infected cells.
about
Evolution of Salmonella enterica virulence via point mutations in the fimbrial adhesin.Exploitation of eukaryotic subcellular targeting mechanisms by bacterial effectorsInteractions of Salmonella enterica with dendritic cellsThe Salmonella SPI2 effector SseI mediates long-term systemic infection by modulating host cell migrationSelective infection of antigen-specific B lymphocytes by Salmonella mediates bacterial survival and systemic spreading of infectionStructure of the catalytic domain of theSalmonellavirulence factor SseIA bacterial toxin catalyzing tyrosine glycosylation of Rho and deamidation of Gq and Gi proteinsInter-kingdom Signaling by the Legionella Quorum Sensing Molecule LAI-1 Modulates Cell Migration through an IQGAP1-Cdc42-ARHGEF9-Dependent PathwaySteC is a Salmonella kinase required for SPI-2-dependent F-actin remodellingA microfluidic-based genetic screen to identify microbial virulence factors that inhibit dendritic cell migrationA naturally occurring single nucleotide polymorphism in the Salmonella SPI-2 type III effector srfH/sseI controls early extraintestinal disseminationMotile invaded neutrophils in the small intestine of Toxoplasma gondii-infected mice reveal a potential mechanism for parasite spread.Subcellular targeting of Salmonella virulence proteins by host-mediated S-palmitoylation.Diverse secreted effectors are required for Salmonella persistence in a mouse infection modelSystemic translocation of Salmonella enterica serovar Dublin in cattle occurs predominantly via efferent lymphatics in a cell-free niche and requires type III secretion system 1 (T3SS-1) but not T3SS-2SpvC is a Salmonella effector with phosphothreonine lyase activity on host mitogen-activated protein kinasesCoordinated regulation of virulence during systemic infection of Salmonella enterica serovar Typhimurium.Development of protective immunity to Salmonella, a mucosal pathogen with a systemic agendaAdaptations to climate-mediated selective pressures in humans.Human genome-wide RNAi screen for host factors that modulate intracellular Salmonella growth.Salmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Change is good: variations in common biological mechanisms in the epsilonproteobacterial genera Campylobacter and HelicobacterDichotomous role of the macrophage in early Mycobacterium marinum infection of the zebrafishDiscovery of Salmonella virulence factors translocated via outer membrane vesicles to murine macrophages.TRIP6: an adaptor protein that regulates cell motility, antiapoptotic signaling and transcriptional activity.Tandem translation generates a chaperone for the Salmonella type III secretion system protein SsaQSrfJ, a Salmonella type III secretion system effector regulated by PhoP, RcsB, and IolR.Human Genome-Wide RNAi Screen for Host Factors That Facilitate Salmonella Invasion Reveals a Role for Potassium Secretion in Promoting Internalization.Identification of salmonella pathogenicity island-2 type III secretion system effectors involved in intramacrophage replication of S. enterica serovar typhimurium: implications for rational vaccine design.Restriction to Fos family members of Trip6-dependent coactivation and glucocorticoid receptor-dependent trans-repression of activator protein-1.The Salmonella-containing vacuole: moving with the times.Type III secretion systems and disease.The LIM domain protein nTRIP6 acts as a co-repressor for the transcription factor MEF2C in myoblasts.Interleukin-15 and NK1.1+ cells provide innate protection against acute Salmonella enterica serovar Typhimurium infection in the gut and in systemic tissuesA method for investigating protein-protein interactions related to salmonella typhimurium pathogenesisUse of high-throughput mass spectrometry to elucidate host-pathogen interactions in Salmonella.Identification of Novel Host Interactors of Effectors Secreted by Salmonella and Citrobacter.Innate immune control of Salmonella enterica serovar Typhimurium: mechanisms contributing to combating systemic Salmonella infection.Pathogens and polymers: microbe-host interactions illuminate the cytoskeleton.Defining the role of TRIP6 in cell physiology and cancer.
P2860
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P2860
Salmonella typhimurium disseminates within its host by manipulating the motility of infected cells.
description
2006 nî lūn-bûn
@nan
2006 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Salmonella typhimurium dissemi ...... he motility of infected cells.
@ast
Salmonella typhimurium dissemi ...... he motility of infected cells.
@en
type
label
Salmonella typhimurium dissemi ...... he motility of infected cells.
@ast
Salmonella typhimurium dissemi ...... he motility of infected cells.
@en
prefLabel
Salmonella typhimurium dissemi ...... he motility of infected cells.
@ast
Salmonella typhimurium dissemi ...... he motility of infected cells.
@en
P2093
P2860
P356
P1476
Salmonella typhimurium dissemi ...... he motility of infected cells.
@en
P2093
Fred Heffron
George S Nieman
Kaoru Geddes
Micah J Worley
P2860
P304
17915-17920
P356
10.1073/PNAS.0604054103
P407
P577
2006-11-09T00:00:00Z