Islet inflammation: a unifying target for diabetes treatment? - Wikidata - awgldk - TriplyDB

Islet inflammation: a unifying target for diabetes treatment?

Islet inflammation: a unifying target for diabetes treatment?

http://www.wikidata.org/entity/Q26860847

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Potential roles of GPR120 and its agonists in the management of diabetes Considerations for Defining Cytokine Dose, Duration, and Milieu That Are Appropriate for Modeling Chronic Low-Grade Inflammation in Type 2 Diabetes Stress-induced dissociations between intracellular calcium signaling and insulin secretion in pancreatic islets Minireview: 12-Lipoxygenase and Islet β-Cell Dysfunction in Diabetes.The expression of the beta cell-derived autoimmune ligand for the killer receptor nkp46 is attenuated in type 2 diabetes.Oxidative stress and susceptibility to mitochondrial permeability transition precedes the onset of diabetes in autoimmune non-obese diabetic mice.Polyamine biosynthesis is critical for growth and differentiation of the pancreas.Low Level Pro-inflammatory Cytokines Decrease Connexin36 Gap Junction Coupling in Mouse and Human Islets through Nitric Oxide-mediated Protein Kinase Cδ.Lipids and immunoinflammatory pathways of beta cell destruction.Molecular and genetic inflammation networks in major human diseases.Interaction between cytokines and inflammatory cells in islet dysfunction, insulin resistance and vascular disease Assessment of the link between in utero exposure to 2-aminoanthracene (2AA) and type-1 diabetes (T1D).Association of proinflammatory cytokines and islet resident leucocytes with islet dysfunction in type 2 diabetes Neutralizing Th2 inflammation in neonatal islets prevents β-cell failure in adult IUGR rats.Iron: the hard player in diabetes pathophysiology.TNFα-induced DLK activation contributes to apoptosis in the beta-cell line HIT.APPL1 prevents pancreatic beta cell death and inflammation by dampening NFκB activation in a mouse model of type 1 diabetes.Lipid signaling and lipotoxicity in metaflammation: indications for metabolic disease pathogenesis and treatment.Coumarins as potential antidiabetic agents.Expression pattern of 12-lipoxygenase in human islets with type 1 diabetes and type 2 diabetes Aspirin, diabetes, and amyloid: re-examination of the inhibition of amyloid formation by aspirin and ketoprofen Deriving at candidate genes of metabolic stress from pancreas of WNIN/GR-Ob mutant rats.12-Lipoxygenase Inhibitor Improves Functions of Cytokine-Treated Human Islets and Type 2 Diabetic Islets.Endoplasmic reticulum stress induced by lipopolysaccharide is involved in the association between inflammation and autophagy in INS‑1 cells.Misfolded proinsulin in the endoplasmic reticulum during development of beta cell failure in diabetes.Effects of 1α,25 Dihydroxyvitamin D3 on Pro-inflammatory Cytokines of Palmitic Acid Treated Thp-1 Cells.The impact of cell surface PEGylation and short-course immunotherapy on islet graft survival in an allogeneic murine model.High-Throughput Yeast-Based Reporter Assay to Identify Compounds with Anti-inflammatory Potential.Vitamin D Switches BAF Complexes to Protect β Cells.The Role of Extracellular Adenosine Generation in the Development of Autoimmune Diseases.Inhibition of TBK1/IKKε Promotes Regeneration of Pancreatic β-cells

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P2860

Islet inflammation: a unifying target for diabetes treatment?

http://www.wikidata.org/entity/Q26860847

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2013 nî lūn-bûn

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2013 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2013 թվականի հուլիսին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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Islet inflammation: a unifying target for diabetes treatment?

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J.TEM.2013.01.007

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Trends in Endocrinology and Metabolism

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Islet inflammation: a unifying target for diabetes treatment?

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Anca D. Dobrian

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Jerry L. Nadler

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Margaret A. Morris

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Yumi Imai

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P2860

The ancient drug salicylate directly activates AMP-activated protein kinase

Endoplasmic reticulum stress and the inflammatory basis of metabolic disease

The role of adipokines in β-cell failure of type 2 diabetes

Functional and pathological roles of the 12- and 15-lipoxygenases

Deficit of tRNA(Lys) modification by Cdkal1 causes the development of type 2 diabetes in mice

Histone deacetylases 1 and 3 but not 2 mediate cytokine-induced beta cell apoptosis in INS-1 cells and dispersed primary islets from rats and are differentially regulated in the islets of type 1 diabetic children

Cytokines tumor necrosis factor-α and interferon-γ induce pancreatic β-cell apoptosis through STAT1-mediated Bim protein activation

Activation of the NLRP3 inflammasome by islet amyloid polypeptide provides a mechanism for enhanced IL-1β in type 2 diabetes.

Functional interactions between the gut microbiota and host metabolism

Type 2 diabetes as an inflammatory disease

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10.1016/J.TEM.2013.01.007

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3686848

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