Oxidized cholesterol as the driving force behind the development of Alzheimer's disease - Wikidata - awgldk - TriplyDB

Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Therapies for Prevention and Treatment of Alzheimer's Disease Redox Imbalance and Viral Infections in Neurodegenerative Diseases Protective Effects of α-Tocopherol, γ-Tocopherol and Oleic Acid, Three Compounds of Olive Oils, and No Effect of Trolox, on 7-Ketocholesterol-Induced Mitochondrial and Peroxisomal Dysfunction in Microglial BV-2 Cells Glycosyl chains and 25-hydroxycholesterol contribute to the intracellular transport of amyloid beta (Aβ-42) in Jurkat T cells Lipid profiles in schizophrenia associated with clinical traits: a five year follow-up study.Changes in brain oxysterols at different stages of Alzheimer's disease: Their involvement in neuroinflammation Updating the Evidence on the Association between Serum Cholesterol and Risk of Late-Life Dementia: Review and Meta-Analysis.Rosuvastatin ameliorates cognitive impairment in rats fed with high-salt and cholesterol diet via inhibiting acetylcholinesterase activity and amyloid beta peptide aggregation.Nuclear Lipids in the Nervous System: What they do in Health and Disease.Amyloid precursor protein and endosomal-lysosomal dysfunction in Alzheimer's disease: inseparable partners in a multifactorial disease.Intracellular Cholesterol Trafficking and Impact in Neurodegeneration.Tempol and perindopril protect against lipopolysaccharide-induced cognition impairment and amyloidogenesis by modulating brain-derived neurotropic factor, neuroinflammation and oxido-nitrosative stress.Increased Levels of 27-Hydroxycholesterol Induced by Dietary Cholesterol in Brain Contribute to Learning and Memory Impairment in Rats.Effects of Plasma Lipids and Statins on Cognitive Function.Characterizing serum metabolic alterations of Alzheimer's disease and intervention of Shengmai-San by ultra-performance liquid chromatography/electrospray ionization quadruple time-of-flight mass spectrometry.Dapsone protects brain microvascular integrity from high-fat diet induced LDL oxidation.Pro- and Anti-inflammatory Effects of High Cholesterol Diet on Aged Brain.A silver lining for 24-hydroxycholesterol in Alzheimer's disease: The involvement of the neuroprotective enzyme sirtuin 1.Strikingly different effects of cholesterol and 7-ketocholesterol on lipid bilayer-mediated aggregation of amyloid beta (1-42).

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

http://www.wikidata.org/entity/Q27023315

description

2015 nî lūn-bûn

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2015 թուականին հրատարակուած գիտական յօդուած

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2015 թվականին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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name

Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

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FNAGI.2015.00119

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Frontiers in Aging Neuroscience

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Oxidized cholesterol as the driving force behind the development of Alzheimer's disease

@en

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Erica Staurenghi

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Giuseppe Poli

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Simona Gargiulo

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P2860

Cytokines and chemokines at the crossroads of neuroinflammation, neurodegeneration, and neuropathic pain

Alzheimer's Disease

Role of Genes and Environments for Explaining Alzheimer Disease

Liver X receptors in the central nervous system: from lipid homeostasis to neuronal degeneration

Pathways towards and away from Alzheimer's disease

Apolipoprotein E and its receptors in Alzheimer's disease: pathways, pathogenesis and therapy

Clearance of amyloid-beta by circulating lipoprotein receptors

Lipoprotein receptors and cholesterol in APP trafficking and proteolytic processing, implications for Alzheimer's disease

ApoE promotes the proteolytic degradation of Abeta

Amyloid precursor protein regulates brain apolipoprotein E and cholesterol metabolism through lipoprotein receptor LRP1

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scholarly article

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3485027

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10.3389/FNAGI.2015.00119

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7

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Gabriella Leonarduzzi

Gabriella Testa

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2015-01-01T00:00:00Z

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279862155

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26150787

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Alzheimer's disease

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4473000

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