Functional dissection of Caenorhabditis elegans CLK-2/TEL2 cell cycle defects during embryogenesis and germline development
about
A role for the malignant brain tumour (MBT) domain protein LIN-61 in DNA double-strand break repair by homologous recombinationLEM-3 - A LEM domain containing nuclease involved in the DNA damage response in C. elegansBisphenol A impairs the double-strand break repair machinery in the germline and causes chromosome abnormalities.The Caenorhabditis elegans homolog of Gen1/Yen1 resolvases links DNA damage signaling to DNA double-strand break repairPhylogeny and function of the invertebrate p53 superfamilyThe Caenorhabditis elegans Werner syndrome protein functions upstream of ATR and ATM in response to DNA replication inhibition and double-strand DNA breaks.The CRL2LRR-1 ubiquitin ligase regulates cell cycle progression during C. elegans development.Cancer models in Caenorhabditis elegans.Genetic and physical interactions between Tel2 and the Med15 Mediator subunit in Saccharomyces cerevisiae.Drosophila Tel2 is expressed as a translational fusion with EpsinR and is a regulator of wingless signalingThe 53BP1 homolog in C. elegans influences DNA repair and promotes apoptosis in response to ionizing radiationReproductive toxicity and meiotic dysfunction following exposure to the pesticides Maneb, Diazinon and FenarimolDNA damage response and spindle assembly checkpoint function throughout the cell cycle to ensure genomic integrity.Pro-crossover factors regulate damage-dependent apoptosis in the Caenorhabditis elegans germ line.METT-10, a putative methyltransferase, inhibits germ cell proliferative fate in Caenorhabditis elegans.LINC complexes promote homologous recombination in part through inhibition of nonhomologous end joining.Beyond ATM: the protein kinase landscape of the DNA damage response.Caenorhabditis elegans as a powerful alternative model organism to promote research in genetic toxicology and biomedicine.Alterations in Caenorhabditis elegans and Cronobacter sakazakii lipopolysaccharide during interaction.DNA damage in germ cells induces an innate immune response that triggers systemic stress resistance.Caenorhabditis elegans BUB-3 and SAN-1/MAD3 Spindle Assembly Checkpoint Components Are Required for Genome Stability in Response to Treatment with Ionizing Radiation.
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P2860
Functional dissection of Caenorhabditis elegans CLK-2/TEL2 cell cycle defects during embryogenesis and germline development
description
2009 nî lūn-bûn
@nan
2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2009年の論文
@ja
2009年論文
@yue
2009年論文
@zh-hant
2009年論文
@zh-hk
2009年論文
@zh-mo
2009年論文
@zh-tw
2009年论文
@wuu
name
Functional dissection of Caeno ...... nesis and germline development
@ast
Functional dissection of Caeno ...... nesis and germline development
@en
Functional dissection of Caeno ...... nesis and germline development
@nl
type
label
Functional dissection of Caeno ...... nesis and germline development
@ast
Functional dissection of Caeno ...... nesis and germline development
@en
Functional dissection of Caeno ...... nesis and germline development
@nl
prefLabel
Functional dissection of Caeno ...... nesis and germline development
@ast
Functional dissection of Caeno ...... nesis and germline development
@en
Functional dissection of Caeno ...... nesis and germline development
@nl
P2093
P2860
P1433
P1476
Functional dissection of Caeno ...... nesis and germline development
@en
P2093
Ingo Büssing
Ralf Schnabel
Sandra C Moser
Sophie von Elsner
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P304
P356
10.1371/JOURNAL.PGEN.1000451
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P577
2009-04-01T00:00:00Z