Spatial and temporal profiles of growth factor expression during CNS demyelination reveal the dynamics of repair priming - Wikidata - awgldk - TriplyDB

Spatial and temporal profiles of growth factor expression during CNS demyelination reveal the dynamics of repair priming

Spatial and temporal profiles of growth factor expression during CNS demyelination reveal the dynamics of repair priming

http://www.wikidata.org/entity/Q27330010

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Neurodevelopmental effects of insulin-like growth factor signaling.Effects of murine and human bone marrow-derived mesenchymal stem cells on cuprizone induced demyelination.Short-term cuprizone feeding induces selective amino acid deprivation with concomitant activation of an integrated stress response in oligodendrocytes.Fibroblast growth factor signaling in oligodendrocyte-lineage cells facilitates recovery of chronically demyelinated lesions but is redundant in acute lesions.Astrogliosis during acute and chronic cuprizone demyelination and implications for remyelination Microglial Hv1 proton channel promotes cuprizone-induced demyelination through oxidative damage.After Intracerebral Hemorrhage, Oligodendrocyte Precursors Proliferate and Differentiate Inside White-Matter Tracts in the Rat Striatum Glial response during cuprizone-induced de- and remyelination in the CNS: lessons learned.Oligodendroglia and neurotrophic factors in neurodegeneration.Stroke neuroprotection: oestrogen and insulin-like growth factor-1 interactions and the role of microglia.The effect of glia-glia interactions on oligodendrocyte precursor cell biology during development and in demyelinating diseases Post-ischemic inflammation regulates neural damage and protection.Inflammatory demyelination alters subcortical visual circuits.The role of growth factors as a therapeutic approach to demyelinating disease.Astrocyte-targeted production of interleukin-6 reduces astroglial and microglial activation in the cuprizone demyelination model: Implications for myelin clearance and oligodendrocyte maturation.Synaptophysin Is a Reliable Marker for Axonal Damage.Neural Stem Cell-Based Regenerative Approaches for the Treatment of Multiple Sclerosis.TRPA1 deficiency is protective in cuprizone-induced demyelination-A new target against oligodendrocyte apoptosis.Erythropoietin attenuates neurological and histological consequences of toxic demyelination in mice.Dietary cholesterol promotes repair of demyelinated lesions in the adult brain.Interleukin-13 immune gene therapy prevents CNS inflammation and demyelination via alternative activation of microglia and macrophages.Remyelination After Cuprizone-Induced Demyelination Is Accelerated in Juvenile Mice.Dynamic Changes of Microglia/Macrophage M1 and M2 Polarization in Theiler's Murine Encephalomyelitis.Receptor tyrosine kinases: molecular switches regulating CNS axon regeneration.MicroRNA-185 regulates spinal cord injuries induced by thoracolumbar spine compression fractures by targeting transforming growth factor-β1 TREM2 sustains microglial expansion during aging and response to demyelination.Astrocytes regulate myelin clearance through recruitment of microglia during cuprizone-induced demyelination.Dynamic and Responsive Growth Factor Delivery from Electrospun and Hydrogel Tissue Engineering Materials.Functional antagonism of sphingosine-1-phosphate receptor 1 prevents cuprizone-induced demyelination.Grey matter OPCs are less mature and less sensitive to IFNγ than white matter OPCs: consequences for remyelination.Pivotal role of choline metabolites in remyelination.Long-term impact of neonatal inflammation on demyelination and remyelination in the central nervous system.Minocycline reduces remyelination by suppressing ciliary neurotrophic factor expression after cuprizone-induced demyelination.Iron Availability Compromises Not Only Oligodendrocytes But Also Astrocytes and Microglial Cells.

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Spatial and temporal profiles of growth factor expression during CNS demyelination reveal the dynamics of repair priming

http://www.wikidata.org/entity/Q27330010

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2011 nî lūn-bûn

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Spatial and temporal profiles ...... the dynamics of repair priming

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Spatial and temporal profiles ...... the dynamics of repair priming

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Spatial and temporal profiles ...... the dynamics of repair priming

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Spatial and temporal profiles ...... the dynamics of repair priming

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Darius Moharregh-Khiabani

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Elke Voss

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Kirsten Wissel

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Konstantin Frichert

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Sabine Wolter

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Viktoria Gudi

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Özlem Yildiz

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P2860

Dissecting demyelination

The blood-brain barrier in cortical multiple sclerosis lesions

Role for TGF-beta1, FGF-2 and PDGF-AA in a myelination of CNS aggregate cultures enriched with macrophages

Isolation of murine microglial cells for RNA analysis or flow cytometry.

Understanding CNS remyelination: clues from developmental and regeneration biology.

Oligodendrocytes and the control of myelination in vivo: new insights from the rat anterior medullary velum.

The neurotoxicant, cuprizone, as a model to study demyelination and remyelination in the central nervous system.

FGF modulates the PDGF-driven pathway of oligodendrocyte development.

Extensive cortical remyelination in patients with chronic multiple sclerosis.

A role for TGF-beta in oligodendrocyte differentiation

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Jelena Škuljec

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