The carboxy-terminal acidic domain of Rift Valley Fever virus NSs protein is essential for the formation of filamentous structures but not for the nuclear localization of the protein.
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Rift Valley fever virus NSs protein functions and the similarity to other bunyavirus NSs proteinsPhleboviruses and the Type I Interferon ResponseThe S Segment of Punta Toro Virus (Bunyaviridae, Phlebovirus) Is a Major Determinant of Lethality in the Syrian Hamster and Codes for a Type I Interferon AntagonistMechanistic Insight into the Host Transcription Inhibition Function of Rift Valley Fever Virus NSs and Its Importance in VirulenceThe role of interferon antagonist, non-structural proteins in the pathogenesis and emergence of arboviruses.A SAP30 complex inhibits IFN-beta expression in Rift Valley fever virus infected cells.Suppression of the interferon and NF-κB responses by severe fever with thrombocytopenia syndrome virus.Rift Valley fever virus NSs protein promotes post-transcriptional downregulation of protein kinase PKR and inhibits eIF2alpha phosphorylation.Differential Antagonism of Human Innate Immune Responses by Tick-Borne Phlebovirus Nonstructural Proteins.Multi-faceted proteomic characterization of host protein complement of Rift Valley fever virus virions and identification of specific heat shock proteins, including HSP90, as important viral host factorsCountermeasure development for Rift Valley fever: deletion, modification or targeting of major virulence factor NSsThe pathogenesis of Rift Valley fever.Functional analysis of Rift Valley fever virus NSs encoding a partial truncation.Advances in Rift Valley fever research: insights for disease preventionThe consequences of reconfiguring the ambisense S genome segment of Rift Valley fever virus on viral replication in mammalian and mosquito cells and for genome packaging.Dual functions of Rift Valley fever virus NSs protein: inhibition of host mRNA transcription and post-transcriptional downregulation of protein kinase PKR.Middle East Respiratory Syndrome Coronavirus nsp1 Inhibits Host Gene Expression by Selectively Targeting mRNAs Transcribed in the Nucleus while Sparing mRNAs of Cytoplasmic Originβ-Catenin Upregulates the Constitutive and Virus-Induced Transcriptional Capacity of the Interferon Beta Promoter through T-Cell Factor Binding SitesRift Valley fever virus NSs inhibits host transcription independently of the degradation of dsRNA-dependent protein kinase PKR.Dicer-2- and Piwi-mediated RNA interference in Rift Valley fever virus-infected mosquito cells.Presence of Viral RNA and Proteins in Exosomes from Cellular Clones Resistant to Rift Valley Fever Virus Infection.Molecular biology and genetic diversity of Rift Valley fever virusLoss of Tau protein affects the structure, transcription and repair of neuronal pericentromeric heterochromatinDevelopment of a RVFV ELISA that can distinguish infected from vaccinated animals.Non-structural proteins of arthropod-borne bunyaviruses: roles and functions.Restriction of rift valley Fever virus virulence in mosquito cells.NSs protein of rift valley fever virus promotes posttranslational downregulation of the TFIIH subunit p62.The S segment of rift valley fever phlebovirus (Bunyaviridae) carries determinants for attenuation and virulence in mice.Genetic evidence for an interferon-antagonistic function of rift valley fever virus nonstructural protein NSsNonstructural NSs protein of rift valley fever virus interacts with pericentromeric DNA sequences of the host cell, inducing chromosome cohesion and segregation defects.NSs protein of Rift Valley fever virus blocks interferon production by inhibiting host gene transcription.Rift Valley fever phlebovirus NSs protein core domain structure suggests molecular basis for nuclear filaments.Large-scale chromatin immunoprecipitation with promoter sequence microarray analysis of the interaction of the NSs protein of Rift Valley fever virus with regulatory DNA regions of the host genome.The nonstructural protein NSs induces a variable antibody response in domestic ruminants naturally infected with Rift Valley fever virus.Current Status of Rift Valley Fever Vaccine Development.Recent advances in the development of antiviral therapeutics for Rift Valley fever virus infection.SFTS phlebovirus promotes LC3-II accumulation and nonstructural protein of SFTS phlebovirus co-localizes with autophagy proteins.
P2860
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P2860
The carboxy-terminal acidic domain of Rift Valley Fever virus NSs protein is essential for the formation of filamentous structures but not for the nuclear localization of the protein.
description
1999 nî lūn-bûn
@nan
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
The carboxy-terminal acidic do ...... ar localization of the protein
@nl
The carboxy-terminal acidic do ...... r localization of the protein.
@ast
The carboxy-terminal acidic do ...... r localization of the protein.
@en
type
label
The carboxy-terminal acidic do ...... ar localization of the protein
@nl
The carboxy-terminal acidic do ...... r localization of the protein.
@ast
The carboxy-terminal acidic do ...... r localization of the protein.
@en
prefLabel
The carboxy-terminal acidic do ...... ar localization of the protein
@nl
The carboxy-terminal acidic do ...... r localization of the protein.
@ast
The carboxy-terminal acidic do ...... r localization of the protein.
@en
P2093
P2860
P921
P3181
P1433
P1476
The carboxy-terminal acidic do ...... ar localization of the protein
@en
P2093
P2860
P304
P3181
P577
1999-06-01T00:00:00Z