The S segment of rift valley fever phlebovirus (Bunyaviridae) carries determinants for attenuation and virulence in mice.
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p53 Activation following Rift Valley fever virus infection contributes to cell death and viral productionPhleboviruses and the Type I Interferon ResponseReverse genetics system for Uukuniemi virus (Bunyaviridae): RNA polymerase I-catalyzed expression of chimeric viral RNAsThe S Segment of Punta Toro Virus (Bunyaviridae, Phlebovirus) Is a Major Determinant of Lethality in the Syrian Hamster and Codes for a Type I Interferon AntagonistRNA polymerase I-mediated expression of viral RNA for the rescue of infectious virulent and avirulent Rift Valley fever virusesRapid Accumulation of Virulent Rift Valley Fever Virus in Mice from an Attenuated Virus Carrying a Single Nucleotide Substitution in the M RNAMetagenomic shotgun sequencing of a Bunyavirus in wild-caught Aedes aegypti from Thailand informs the evolutionary and genomic history of the Phleboviruses.Characterization of the Sandfly fever Naples species complex and description of a new Karimabad species complex (genus Phlebovirus, family Bunyaviridae).A SAP30 complex inhibits IFN-beta expression in Rift Valley fever virus infected cells.Characterization of the Uukuniemi virus group (Phlebovirus: Bunyaviridae): evidence for seven distinct speciesEvolutionary and molecular analysis of the emergent severe fever with thrombocytopenia syndrome virus.Mapping of transcription termination within the S segment of SFTS phlebovirus facilitated the generation of NSs-deletant viruses.Reverse genetics technology for Rift Valley fever virus: current and future applications for the development of therapeutics and vaccines.The NSs protein of tomato spotted wilt virus is required for persistent infection and transmission by Frankliniella occidentalis.Pathogenicity of Hantaan virus in newborn mice: genetic reassortant study demonstrating that a single amino acid change in glycoprotein G1 is related to virulenceVaccination with DNA plasmids expressing Gn coupled to C3d or alphavirus replicons expressing gn protects mice against Rift Valley fever virus.Molecular biology of rift valley Fever virusHost alternation is necessary to maintain the genome stability of rift valley fever virus.Comparative pathogenesis and systems biology for biodefense virus vaccine development.Rift Valley fever virus(Bunyaviridae: Phlebovirus): an update on pathogenesis, molecular epidemiology, vectors, diagnostics and preventionThe pathogenesis of Rift Valley fever.Rift Valley fever virus vaccine lacking the NSs and NSm genes is safe, nonteratogenic, and confers protection from viremia, pyrexia, and abortion following challenge in adult and pregnant sheep.Negative-strand tospoviruses and tenuiviruses carry a gene for a suppressor of gene silencing at analogous genomic positions.Rift Valley Fever Virus MP-12 Vaccine Is Fully Attenuated by a Combination of Partial Attenuations in the S, M, and L Segments.Schmallenberg virus pathogenesis, tropism and interaction with the innate immune system of the host.Development of a novel, single-cycle replicable rift valley Fever vaccine.A ΩXaV motif in the Rift Valley fever virus NSs protein is essential for degrading p62, forming nuclear filaments and virulence.Induction of DNA damage signaling upon Rift Valley fever virus infection results in cell cycle arrest and increased viral replicationMolecular epidemiology of Rift Valley fever virusSingle-dose immunization with virus replicon particles confers rapid robust protection against Rift Valley fever virus challengeThe dominant-negative inhibition of double-stranded RNA-dependent protein kinase PKR increases the efficacy of Rift Valley fever virus MP-12 vaccine.Rift valley fever virus lacking the NSs and NSm genes is highly attenuated, confers protective immunity from virulent virus challenge, and allows for differential identification of infected and vaccinated animals.Dicer-2- and Piwi-mediated RNA interference in Rift Valley fever virus-infected mosquito cells.Molecular biology and genetic diversity of Rift Valley fever virusRift Valley fever virus clearance and protection from neurologic disease are dependent on CD4+ T cell and virus-specific antibody responses.Development of a RVFV ELISA that can distinguish infected from vaccinated animals.Rift valley fever vaccines.Interplay between the Virus and Host in Rift Valley Fever Pathogenesis.Restriction of rift valley Fever virus virulence in mosquito cells.Creation of a recombinant Rift Valley fever virus with a two-segmented genome.
P2860
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P2860
The S segment of rift valley fever phlebovirus (Bunyaviridae) carries determinants for attenuation and virulence in mice.
description
2000 nî lūn-bûn
@nan
2000年の論文
@ja
2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
2000年论文
@zh
2000年论文
@zh-cn
name
The S segment of rift valley f ...... nuation and virulence in mice.
@en
The S segment of rift valley fever phlebovirus
@nl
type
label
The S segment of rift valley f ...... nuation and virulence in mice.
@en
The S segment of rift valley fever phlebovirus
@nl
prefLabel
The S segment of rift valley f ...... nuation and virulence in mice.
@en
The S segment of rift valley fever phlebovirus
@nl
P2093
P2860
P1433
P1476
The S segment of rift valley f ...... nuation and virulence in mice.
@en
P2093
P2860
P304
P356
10.1128/JVI.74.3.1538-1543.2000
P407
P577
2000-02-01T00:00:00Z