Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells - Wikidata - awgldk - TriplyDB

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

http://www.wikidata.org/entity/Q27480362

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Entry inhibitors: New advances in HCV treatment Impact of lipids and lipoproteins on hepatitis C virus infection and virus neutralization Hepatic serum amyloid A1 aggravates T cell-mediated hepatitis by inducing chemokines via Toll-like receptor 2 in mice Elevation of intact and proteolytic fragments of acute phase proteins constitutes the earliest systemic antiviral response in HIV-1 infection Secretory phospholipase A2, group IIA is a novel serum amyloid A target gene: activation of smooth muscle cell expression by an interleukin-1 receptor-independent mechanism.Structure and Expression of Different Serum Amyloid A (SAA) Variants and their Concentration-Dependent Functions During Host Insults Uropathogenic Escherichia coli induces serum amyloid a in mice following urinary tract and systemic inoculation.Hepatitis C virus experimental model systems and antiviral drug research.Hepatitis C virus entry.Early steps of the hepatitis C virus life cycle.IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.Role of lipid metabolism in hepatitis C virus assembly and entry.Know your enemy: translating insights about the molecular biology of hepatitis C virus into novel therapeutic approaches.Scavenger receptor class B type I and the hypervariable region-1 of hepatitis C virus in cell entry and neutralisation.The role of humoral innate immunity in hepatitis C virus infection.Serum amyloid A immunohistochemical staining patterns in hepatitis.Hepatitis C virus dynamics and cellular gene expression in uPA-SCID chimeric mice with humanized livers during intravenous silibinin monotherapy.Interplay among cellular polarization, lipoprotein metabolism and hepatitis C virus entry.Increased levels of serum amyloid A during the early phase of hepatitis C treatment with interferon are associated with sustained virologic response - a pilot study.Serum amyloid A3 is required for normal weight and immunometabolic function in mice.Matrix Metalloproteinase-9-Generated COOH-, but Not NH2-Terminal Fragments of Serum Amyloid A1 Retain Potentiating Activity in Neutrophil Migration to CXCL8, With Loss of Direct Chemotactic and Cytokine-Inducing Capacity.Blocking HCV entry as potential antiviral therapy Serum Amyloid A3 is required for normal lung development and survival following influenza infection

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P2860

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

http://www.wikidata.org/entity/Q27480362

description

2007 nî lūn-bûn

@nan

2007 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2007 թվականի հունիսին հրատարակված գիտական հոդված

@hy

2007年の論文

@ja

2007年論文

@yue

2007年論文

@zh-hant

2007年論文

@zh-hk

2007年論文

@zh-mo

2007年論文

@zh-tw

2007年论文

@wuu

name

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

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Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

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Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

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type

label

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

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Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

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Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

@nl

prefLabel

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

@ast

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

@en

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

@nl

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P1433

Journal of Virology

P1476

Human Serum Amyloid A Protein Inhibits Hepatitis C Virus Entry into Cells

@en

P2093

Deneys R van der Westhuyzen

http://www.w3.org/2001/XMLSchema#string

Guangxiang Luo

http://www.w3.org/2001/XMLSchema#string

Jieyun Jiang

http://www.w3.org/2001/XMLSchema#string

Kyung-Soo Chang

http://www.w3.org/2001/XMLSchema#string

Lei Cai

http://www.w3.org/2001/XMLSchema#string

Zhaohui Cai

http://www.w3.org/2001/XMLSchema#string

P2860

Binding of hepatitis C virus to CD81

The human scavenger receptor class B type I is a novel candidate receptor for the hepatitis C virus.

Initiation of hepatitis C virus infection is dependent on cholesterol and cooperativity between CD81 and scavenger receptor B type I

Scavenger receptor class B type I and hepatitis C virus infection of primary tupaia hepatocytes.

Robust hepatitis C virus infection in vitro.

Robust production of infectious hepatitis C virus (HCV) from stably HCV cDNA-transfected human hepatoma cells.

Highly permissive cell lines for subgenomic and genomic hepatitis C virus RNA replication.

Association between Hepatitis C Virus and Very-Low-Density Lipoprotein (VLDL)/LDL Analyzed in Iodixanol Density Gradients

Hepatitis C virus and other flaviviridae viruses enter cells via low density lipoprotein receptor

Production of infectious hepatitis C virus in tissue culture from a cloned viral genome

P304

6128-33

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scholarly article

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10.1128/JVI.02627-06

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11

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P478

81

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P577

2007-06-01T00:00:00Z

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17329325

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P932

1900255

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