IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
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Aerobic Exercise and Pharmacological Therapies for Skeletal Myopathy in Heart Failure: Similarities and DifferencesNonmuscle Tissues Contribution to Cancer CachexiaInterleukin-6 myokine signaling in skeletal muscle: a double-edged sword?Current status of bicarbonate in CKDInflammation-induced acute phase response in skeletal muscle and critical illness myopathyUremic myopathy: is oxidative stress implicated in muscle dysfunction in uremia?Tumor necrosis factor-α regulates distinct molecular pathways and gene networks in cultured skeletal muscle cellsGhrelin prevents tumour- and cisplatin-induced muscle wasting: characterization of multiple mechanisms involved.Satellite cell dysfunction and impaired IGF-1 signaling cause CKD-induced muscle atrophyPotential involvement of P2Y2 receptor in diuresis of postobstructive uropathy in rats.Combined effects of ghrelin and higher food intake enhance skeletal muscle mitochondrial oxidative capacity and AKT phosphorylation in rats with chronic kidney disease.IGF-1 prevents ANG II-induced skeletal muscle atrophy via Akt- and Foxo-dependent inhibition of the ubiquitin ligase atrogin-1 expression.Clinical development of ghrelin axis-derived molecules for cancer cachexia treatmentAtrogin-1 affects muscle protein synthesis and degradation when energy metabolism is impaired by the antidiabetes drug berberine.Glucocorticoids increase adipocytes in muscle by affecting IL-4 regulated FAP activity.Shorter telomeres in peripheral blood mononuclear cells from older persons with sarcopenia: results from an exploratory studyAngiotensin type 2 receptor signaling in satellite cells potentiates skeletal muscle regeneration.Macrophage-stimulated cardiac fibroblast production of IL-6 is essential for TGF β/Smad activation and cardiac fibrosis induced by angiotensin IIAngiotensin II modulates mouse skeletal muscle resting conductance to chloride and potassium ions and calcium homeostasis via the AT1 receptor and NADPH oxidaseIL-6 signaling blockade increases inflammation but does not affect muscle function in the mdx mouse.Melanocortin antagonism ameliorates muscle wasting and inflammation in chronic kidney disease.Akt1-mediated fast/glycolytic skeletal muscle growth attenuates renal damage in experimental kidney disease.Myostatin suppression of Akirin1 mediates glucocorticoid-induced satellite cell dysfunction.Mechanisms of muscle wasting in chronic kidney diseaseSerelaxin reduces oxidative stress and asymmetric dimethylarginine in angiotensin II-induced hypertension.Pharmacological inhibition of myostatin suppresses systemic inflammation and muscle atrophy in mice with chronic kidney disease.Protein phosphatase 2C-alpha knockdown reduces angiotensin II-mediated skeletal muscle wasting via restoration of mitochondrial recycling and functionAngiotensin II induced catabolic effect and muscle atrophy are redox dependent.Delving into disability in Crohn's disease: dysregulation of molecular pathways may explain skeletal muscle loss in Crohn's disease.Low-frequency electrical stimulation attenuates muscle atrophy in CKD--a potential treatment strategyXIAP reduces muscle proteolysis induced by CKD.Angiotensin II upregulates protein phosphatase 2Cα and inhibits AMP-activated protein kinase signaling and energy balance leading to skeletal muscle wastingInhibition of Stat3 activation suppresses caspase-3 and the ubiquitin-proteasome system, leading to preservation of muscle mass in cancer cachexiaDecreased miR-29 suppresses myogenesis in CKD.Cachexia and protein-energy wasting in children with chronic kidney disease.The TWEAK-Fn14 system: breaking the silence of cytokine-induced skeletal muscle wasting.Targeting the myostatin signaling pathway to treat muscle wasting diseasesAngiotensin II reduces food intake by altering orexigenic neuropeptide expression in the mouse hypothalamus.Role of integrin-β3 protein in macrophage polarization and regeneration of injured muscleSerum Amyloid A Induces Toll-Like Receptor 2-Dependent Inflammatory Cytokine Expression and Atrophy in C2C12 Skeletal Muscle Myotubes.
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P2860
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 21 January 2009
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@en
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@nl
type
label
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@en
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@nl
prefLabel
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@en
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@nl
P2093
P2860
P356
P1476
IL-6 and serum amyloid A synergy mediates angiotensin II-induced muscle wasting.
@en
P2093
Gabriela Garcia
Guofeng Han
Liping Zhang
William E Mitch
Zhaoyong Hu
P2860
P304
P356
10.1681/ASN.2008060628
P577
2009-01-21T00:00:00Z