Tregs control the development of symptomatic West Nile virus infection in humans and mice - Wikidata - awgldk - TriplyDB

Tregs control the development of symptomatic West Nile virus infection in humans and mice

Tregs control the development of symptomatic West Nile virus infection in humans and mice

http://www.wikidata.org/entity/Q27490259

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Regulatory T cell suppressive potency dictates the balance between bacterial proliferation and clearance during persistent Salmonella infection Expanding Regulatory T Cells Alleviates Chikungunya Virus-Induced Pathology in Mice West Nile Virus Infection in the Central Nervous System West Nile Virus: biology, transmission, and human infection Systems analysis of West Nile virus infection Oncolytic viruses as anticancer vaccines Age-Dependent Cell Trafficking Defects in Draining Lymph Nodes Impair Adaptive Immunity and Control of West Nile Virus Infection Tregs Modulate Lymphocyte Proliferation, Activation, and Resident-Memory T-Cell Accumulation within the Brain during MCMV Infection T regulatory cells control susceptibility to invasive pneumococcal pneumonia in mice CCR5 ameliorates Japanese encephalitis via dictating the equilibrium of regulatory CD4+Foxp3+ T and IL-17+CD4+ Th17 cells IPS-1 Is Essential for the Control of West Nile Virus Infection and Immunity A Mouse Model of Chronic West Nile Virus Disease Origin and functions of pro-inflammatory cytokine producing Foxp3+ regulatory T cells Immune Surveillance of the CNS following Infection and Injury Interferon regulatory factor-1 (IRF-1) shapes both innate and CD8(+) T cell immune responses against West Nile virus infection Extrinsic MAVS signaling is critical for Treg maintenance of Foxp3 expression following acute flavivirus infection Chemokine Receptor Ccr7 Restricts Fatal West Nile Virus Encephalitis CD11c-Expressing Cells Affect Regulatory T Cell Behavior in the Meninges during Central Nervous System Infection.Persistence of virus-specific immune responses in the central nervous system of mice after West Nile virus infection.Viral encephalomyelitis.Protective and detrimental roles for regulatory T cells in a viral model for multiple sclerosis T cell epitope specificity and pathogenesis of mouse hepatitis virus-1-induced disease in susceptible and resistant hosts.Association between HLA class I and class II alleles and the outcome of West Nile virus infection: an exploratory study Virus-specific regulatory T cells ameliorate encephalitis by repressing effector T cell functions from priming to effector stages Identification of genes critical for resistance to infection by West Nile virus using RNA-Seq analysis.Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways.NIAID workshop on Flavivirus immunity.Epitope-specific regulatory CD4 T cells reduce virus-induced illness while preserving CD8 T-cell effector function at the site of infection.CD4+ T cells are not required for the induction of dengue virus-specific CD8+ T cell or antibody responses but contribute to protection after vaccination.Impaired virus clearance, compromised immune response and increased mortality in type 2 diabetic mice infected with West Nile virus.Suppression of innate immune pathology by regulatory T cells during Influenza A virus infection of immunodeficient mice.An analysis of regulatory T-cell and Th-17 cell dynamics during cytomegalovirus replication in solid organ transplant recipients CD4(+)CD25(+)Foxp3(+) regulatory T cells promote Th17 cells in vitro and enhance host resistance in mouse Candida albicans Th17 cell infection model.How tolerogenic dendritic cells induce regulatory T cells West Nile virus nucleic acid persistence in whole blood months after clearance in plasma: implication for transfusion and transplantation safety.IL-21 restricts virus-driven Treg cell expansion in chronic LCMV infection.Systems immunology reveals markers of susceptibility to West Nile virus infection.Macrophage IL-12p70 signaling prevents HSV-1-induced CNS autoimmunity triggered by autoaggressive CD4+ Tregs.Impaired interferon signaling in dendritic cells from older donors infected in vitro with West Nile virus.Virally expressed interleukin-10 ameliorates acute encephalomyelitis and chronic demyelination in coronavirus-infected mice.

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Tregs control the development of symptomatic West Nile virus infection in humans and mice

http://www.wikidata.org/entity/Q27490259

description

2009 nî lūn-bûn

@nan

2009 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Tregs control the development of symptomatic West Nile virus infection in humans and mice

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Tregs control the development of symptomatic West Nile virus infection in humans and mice

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Tregs control the development of symptomatic West Nile virus infection in humans and mice

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type

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Tregs control the development of symptomatic West Nile virus infection in humans and mice

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Tregs control the development of symptomatic West Nile virus infection in humans and mice

@en

Tregs control the development of symptomatic West Nile virus infection in humans and mice

@nl

prefLabel

Tregs control the development of symptomatic West Nile virus infection in humans and mice

@ast

Tregs control the development of symptomatic West Nile virus infection in humans and mice

@en

Tregs control the development of symptomatic West Nile virus infection in humans and mice

@nl

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Journal of Clinical Investigation

P1476

Tregs control the development of symptomatic West Nile virus infection in humans and mice

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Alexander Y Rudensky

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Brian Custer

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Dale F Hirschkorn

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David J Kelvin

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Hany T Kamel

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Harry E Prince

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Jennifer M Lund

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John W Heitman

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Katie M O'Brien

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Leslie H Tobler

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P2860

Development and persistence of West Nile virus-specific immunoglobulin M (IgM), IgA, and IgG in viremic blood donors.

Virology, pathology, and clinical manifestations of West Nile virus disease

Risk factors for West Nile virus neuroinvasive disease, California, 2005

Complement activation is required for induction of a protective antibody response against West Nile virus infection

CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

B cells and antibody play critical roles in the immediate defense of disseminated infection by West Nile encephalitis virus.

Resistance to Alpha/Beta Interferon Is a Determinant of West Nile Virus Replication Fitness and Virulence

Pathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral Evasion

CD4+ T-Cell Responses Are Required for Clearance of West Nile Virus from the Central Nervous System

Abrogation of macrophage migration inhibitory factor decreases West Nile virus lethality by limiting viral neuroinvasion

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3266-77

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2253246

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10.1172/JCI39387

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11

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119

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Michael S. Diamond

Douglas F. Nixon

Marion C. Lanteri

Michael P. Busch

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2009-11-01T00:00:00Z

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19855131

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West Nile virus

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2769173

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