Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways. - Wikidata - awgldk - TriplyDB

Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways.

Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways.

http://www.wikidata.org/entity/Q34134217

about

CCR5 ameliorates Japanese encephalitis via dictating the equilibrium of regulatory CD4+Foxp3+ T and IL-17+CD4+ Th17 cells Interactions of human microglia cells with Japanese encephalitis virus Zika virus disrupts molecular fingerprinting of human neurospheres Exacerbation of Japanese Encephalitis by CD11chi Dendritic Cell Ablation Is Associated with an Imbalance in Regulatory Foxp3+ and IL-17+CD4+ Th17 Cells and in Ly-6Chi and Ly-6Clo Monocytes.A Japanese encephalitis virus genotype 5 molecular clone is highly neuropathogenic in a mouse model: impact of the structural protein region on virulence Amelioration of Japanese encephalitis by blockage of 4-1BB signaling is coupled to divergent enhancement of type I/II IFN responses and Ly-6C(hi) monocyte differentiation.CD11c(hi) Dendritic Cells Regulate Ly-6C(hi) Monocyte Differentiation to Preserve Immune-privileged CNS in Lethal Neuroinflammation.Blockage of indoleamine 2,3-dioxygenase regulates Japanese encephalitis via enhancement of type I/II IFN innate and adaptive T-cell responses.Encephalitic Arboviruses: Emergence, Clinical Presentation, and Neuropathogenesis.Diversity in a honey bee pathogen: first report of a third master variant of the Deformed Wing Virus quasispecies Polymorphisms of toll-like receptors 2 and 9 and severity and prognosis of bacterial meningitis in Chinese children.Mesenchymal stem cells alleviate Japanese encephalitis virus-induced neuroinflammation and mortality.TLR7 Deficiency Leads to TLR8 Compensative Regulation of Immune Response against JEV in Mice MLKL Mediated Necroptosis Accelerates JEV-Induced Neuroinflammation in Mice.Viral sensing at the blood-brain barrier: new roles for innate immunity at the CNS vasculature.CCL2, but not its receptor, is essential to restrict immune privileged central nervous system-invasion of Japanese encephalitis virus via regulating accumulation of CD11b(+) Ly-6C(hi) monocytes.Regulation of inflammation in Japanese encephalitis.Dengue virus NS1 protein activates cells via Toll-like receptor 4 and disrupts endothelial cell monolayer integrity.TLR2/MyD88 pathway-dependent regulation of dendritic cells by dengue virus promotes antibody-dependent enhancement via Th2-biased immunity.Infection with Classical Swine Fever Virus Induces Expression of Type III Interferons and Activates Innate Immune Signaling.Structural study of the C-terminal domain of non-structural protein 1 from Japanese encephalitis virus.Dual TLR2/9 Recognition of Herpes Simplex Virus Infection Is Required for Recruitment and Activation of Monocytes and NK Cells and Restriction of Viral Dissemination to the Central Nervous System.Toll-Like Receptors and RIG-I-Like Receptors Play Important Roles in Resisting Flavivirus.

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Distinct dictation of Japanese encephalitis virus-induced neuroinflammation and lethality via triggering TLR3 and TLR4 signal pathways.

http://www.wikidata.org/entity/Q34134217

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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JOURNAL.PPAT.1004319

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Distinct dictation of Japanese ...... TLR3 and TLR4 signal pathways.

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Bum Seok Kim

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Erdenebelig Uyangaa

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Jin Hyoung Kim

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Jin Young Choi

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Koanhoi Kim

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Seong Bum Kim

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Seong Kug Eo

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Young Woo Han

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P2860

Japanese encephalitis-a pathological and clinical perspective

Novel signaling interactions between proteinase-activated receptor 2 and Toll-like receptors in vitro and in vivo

The atypical PKCs in inflammation: NF-κB and beyond

Toll-like receptors: key players in antiviral immunity

Differential Expression of Interferon (IFN) Regulatory Factors and IFN-Stimulated Genes at Early Times after West Nile Virus Infection of Mouse Embryo Fibroblasts

Distinct RIG-I and MDA5 Signaling by RNA Viruses in Innate Immunity

Ly6c+ "inflammatory monocytes" are microglial precursors recruited in a pathogenic manner in West Nile virus encephalitis

Toll-Like Receptor 3 Has a Protective Role against West Nile Virus Infection

Toll-like Receptor 7 Mitigates Lethal West Nile Encephalitis via Interleukin 23-Dependent Immune Cell Infiltration and Homing

Tregs control the development of symptomatic West Nile virus infection in humans and mice

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9

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Japanese encephalitis virus

Toll-like receptor

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