Research priorities in sarcomeric cardiomyopathies - Wikidata - awgldk - TriplyDB

Research priorities in sarcomeric cardiomyopathies

Research priorities in sarcomeric cardiomyopathies

http://www.wikidata.org/entity/Q27694507

about

Novel myosin-based therapies for congenital cardiac and skeletal myopathies MicroRNAs Based Therapy of Hypertrophic Cardiomyopathy: The Road Traveled So Far INHERIT (INHibition of the renin angiotensin system in hypertrophic cardiomyopathy and the Effect on hypertrophy-a Randomised Intervention Trial with losartan)Atomic resolution probe for allostery in the regulatory thin filament.Proteomic analysis of physiological versus pathological cardiac remodeling in animal models expressing mutations in myosin essential light chains.Hypercontractile mutant of ventricular myosin essential light chain leads to disruption of sarcomeric structure and function and results in restrictive cardiomyopathy in mice.Genetics of hypertrophic cardiomyopathy: A review of current state.Contribution of Post-translational Phosphorylation to Sarcomere-Linked Cardiomyopathy Phenotypes.Life-long tailoring of management for patients with hypertrophic cardiomyopathy : Awareness and decision-making in changing scenarios.Commentary: Next Generation Sequencing and Linkage Analysis for the Molecular Diagnosis of a Novel Overlapping Syndrome Characterized by Hypertrophic Cardiomyopathy and Typical Electrical Instability of Brugada Syndrome.Ranolazine antagonizes catecholamine-induced dysfunction in isolated cardiomyocytes, but lacks long-term therapeutic effects in vivo in a mouse model of hypertrophic cardiomyopathy.Sarcomeric cardiomyopathies: from bedside to bench and back.Editorial highlights from Cardiovascular Research.Calcium in Brugada Syndrome: Questions for Future Research Impact of the Myosin Modulator Mavacamten on Force Generation and Cross-Bridge Behavior in a Murine Model of Hypercontractility Altered Ca and Na Homeostasis in Human Hypertrophic Cardiomyopathy: Implications for Arrhythmogenesis

P2860

Q26740449-0242CC17-9A0E-4948-94E1-C7AE9181FDBE Q26778555-029C0F69-1F32-4326-9B52-FADBB39C173C Q35217983-14F1D7B4-5411-4B27-B463-D9B90CF0AA5C Q36742843-A3AA5429-B3C0-4984-9D5B-AB565A49A970 Q36843579-E8455BDE-FC05-43D6-ACFF-C8341D332B3E Q38735729-1742BA27-C725-4229-A1D2-281AE07A6F7C Q38736063-8C387CE4-96A8-49BB-9A8B-F380F77190D7 Q38816583-0ED87E6F-9FF1-45CB-BF22-A277883D16A9 Q42320941-EB018499-37A5-4D90-B049-869A8196E29B Q47192340-B78FD1C8-1EAA-4863-A9B7-A681521B5CB6 Q48747768-4BD79E85-C107-4F3E-8802-E4FA4F0E1A85 Q48750044-B36074D1-993A-4B9A-BB56-13B9939041E9 Q49549470-18EF5E42-A994-4555-AEFD-A2C3CE1640F5 Q57169914-E3A4222A-68FE-4B15-AF78-8126E39247CB Q58127428-F4B958CC-D2A1-43E6-B8A9-72FD7D22FFC7 Q58699044-2FB2E2C0-E930-4524-B8FC-855948721751

P2860

Research priorities in sarcomeric cardiomyopathies

http://www.wikidata.org/entity/Q27694507

description

2015 nî lūn-bûn

@nan

2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2015 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

name

Research priorities in sarcomeric cardiomyopathies

@ast

Research priorities in sarcomeric cardiomyopathies

@en

Research priorities in sarcomeric cardiomyopathies

@nl

type

label

Research priorities in sarcomeric cardiomyopathies

@ast

Research priorities in sarcomeric cardiomyopathies

@en

Research priorities in sarcomeric cardiomyopathies

@nl

prefLabel

Research priorities in sarcomeric cardiomyopathies

@ast

Research priorities in sarcomeric cardiomyopathies

@en

Research priorities in sarcomeric cardiomyopathies

@nl

P1433

Q27694507-4D60C2FE-4D1E-48D2-B186-A6FB5CA28A79

P1476

Q27694507-438CAA3A-D183-4668-A28C-720657696385

P2093

Q27694507-93766247-975E-4561-90B9-294A0B8E96CF

Q27694507-A15CB4B6-E02C-4E31-9988-78D71D3ACA89

Q27694507-AAC18ACB-8E03-4A03-BA5C-E1B696712CAE

Q27694507-F860FCB5-B4AC-4CD9-9F5A-9483830D70E3

P2860

Q27694507-053F771B-DFC0-4D0C-8B19-6185EC5E1D3C

Q27694507-09F92C7D-79F0-4BD7-BA8B-8C4F30C409F3

Q27694507-0B79292A-D523-4FB9-A293-55136B35A012

Q27694507-0BB473F7-1E5E-4D3B-ACD7-9EEFA5A4620C

Q27694507-0C46FBC7-3B60-4D48-BFA3-659FFCF5DF77

Q27694507-0DBF8FAB-1343-4A41-90B8-E959E23CAEBC

Q27694507-16335A8B-D1DB-4BCE-BA2C-54AD63178317

Q27694507-1BCD4E81-7C1A-432F-8A9C-211D0A5FDF29

Q27694507-1DD202C7-AABA-4EBB-94D2-A81350BB8AD5

Q27694507-204C67C7-AE49-46F2-AFE1-B4EB66A45F09

P304

Q27694507-1EE75316-5F5A-45F1-99D8-D3AB713036C7

P31

Q27694507-94E9DF09-67E1-451D-BEA6-8000A94F0549

P3181

Q27694507-10916EAA-D17F-40ED-ABE7-F118B06D9E26

P356

Q27694507-D6724EA2-5315-4170-BEA3-EEC8F06325AF

P407

Q27694507-31BE6297-127B-4DF5-9C77-CE8A90282BA1

P433

Q27694507-083D1413-2BAE-4E89-97FB-0173E25559B4

P478

Q27694507-10598F2E-892A-470B-808A-7321C195F983

P50

Q27694507-D60771A9-B875-4F9A-8138-457D8C5CDDDF

Q27694507-F1171476-D974-428A-9818-98482C196D89

P577

Q27694507-F62B6831-5744-4BD2-8BDC-994C23C9DE02

P5875

Q27694507-442FF6C9-BF75-4E9C-A226-0B205C8AD06F

P698

Q27694507-586A9D70-AB33-4CA0-BA43-77B88112BA05

P932

Q27694507-E5881C5B-359D-413D-B2EC-EA3C49A9D93B

P3181

P356

P1433

Cardiovascular Research

P1476

Research priorities in sarcomeric cardiomyopathies

@en

P2093

Carolyn Y Ho

http://www.w3.org/2001/XMLSchema#string

Iacopo Olivotto

http://www.w3.org/2001/XMLSchema#string

Jil C Tardiff

http://www.w3.org/2001/XMLSchema#string

Lucie Carrier

http://www.w3.org/2001/XMLSchema#string

P2860

Alterations in thin filament regulation induced by a human cardiac troponin T mutant that causes dilated cardiomyopathy are distinct from those induced by troponin T mutants that cause hypertrophic cardiomyopathy

Dilated cardiomyopathy mutant tropomyosin mice develop cardiac dysfunction with significantly decreased fractional shortening and myofilament calcium sensitivity

Severe disease expression of cardiac troponin C and T mutations in patients with idiopathic dilated cardiomyopathy

Mutations in fast skeletal troponin I, troponin T, and beta-tropomyosin that cause distal arthrogryposis all increase contractile function

Allele-specific silencing of mutant Myh6 transcripts in mice suppresses hypertrophic cardiomyopathy

Animal and in silico models for the study of sarcomeric cardiomyopathies

Targets for therapy in sarcomeric cardiomyopathies

Mutations in sarcomere protein genes as a cause of dilated cardiomyopathy

Hypertrophic cardiomyopathy: distribution of disease genes, spectrum of mutations, and implications for a molecular diagnosis strategy

Cardiac myosin-binding protein C mutations and hypertrophic cardiomyopathy: haploinsufficiency, deranged phosphorylation, and cardiomyocyte dysfunction

P304

449-456

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P3181

3527339

http://www.w3.org/2001/XMLSchema#string

P356

10.1093/CVR/CVV019

http://www.w3.org/2001/XMLSchema#string

P407

P433

4

http://www.w3.org/2001/XMLSchema#string

P478

105

http://www.w3.org/2001/XMLSchema#string

P50

Jolanda van der Velden

Bjorn C. Knollmann

P577

2015-04-01T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

271596802

http://www.w3.org/2001/XMLSchema#string

P698

25631582

http://www.w3.org/2001/XMLSchema#string

P932

4375392

http://www.w3.org/2001/XMLSchema#string