Biology of MET: a double life between normal tissue repair and tumor progression - Wikidata - awgldk - TriplyDB

Biology of MET: a double life between normal tissue repair and tumor progression

Biology of MET: a double life between normal tissue repair and tumor progression

http://www.wikidata.org/entity/Q28085247

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Chronic arsenic trioxide exposure leads to enhanced aggressiveness via Met oncogene addiction in cancer cells.Effects of HGF gene polymorphisms and protein expression on transhepatic arterial chemotherapeutic embolism efficacy and prognosis in patients with primary liver cancer.MET tyrosine kinase receptor expression and amplification as prognostic biomarkers of survival in gastroesophageal adenocarcinoma.MET expression and copy number status in clear-cell renal cell carcinoma: prognostic value and potential predictive marker.Hepatocyte growth factor/MET in cancer progression and biomarker discovery Inhibition of the ubiquitin ligase activity improves the production of biologically active fusion protein HSA-HGF in Chinese hamster ovary cells.Role and Therapeutic Targeting of the HGF/MET Pathway in Glioblastoma.In vitro significance of SOCS-3 and SOCS-4 and potential mechanistic links to wound healing.Signaling by MET Targeted therapy of gastroenteropancreatic neuroendocrine tumours: preclinical strategies and future targets.Candidate Biomarkers of Liver Fibrosis: A Concise, Pathophysiology-oriented Review Cellular signaling and gene expression profiles evoked by a bivalent macrocyclic peptide that serves as an artificial MET receptor agonist

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P2860

Biology of MET: a double life between normal tissue repair and tumor progression

http://www.wikidata.org/entity/Q28085247

description

2015 nî lūn-bûn

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2015 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2015 թվականի ապրիլին հրատարակված գիտական հոդված

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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Biology of MET: a double life between normal tissue repair and tumor progression

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J.ISSN.2305-5839.2015.03.58

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Annals of translational medicine

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Biology of MET: a double life between normal tissue repair and tumor progression

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Anna-Barbara Moscicki

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Dorothy J. Wiley

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Elizabeth A. Stier

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Elizabeth Chiao

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J. Michael Berry-Lawhorn

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Jennifer M. Roberts

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Joel M. Palefsky

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Lori A. Boardman

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Mark H. Einstein

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Mark L. Welton

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P2860

An integrated map of genetic variation from 1,092 human genomes

Use of signal specific receptor tyrosine kinase oncoproteins reveals that pathways downstream from Grb2 or Shc are sufficient for cell transformation and metastasis

Crystal structure of the HGF beta-chain in complex with the Sema domain of the Met receptor

Hepatocyte growth factor receptor tyrosine kinase met is a substrate of the receptor protein-tyrosine phosphatase DEP-1

An uncleavable form of pro-scatter factor suppresses tumor growth and dissemination in mice

An overview of the c-MET signaling pathway

MET amplification occurs with or without T790M mutations in EGFR mutant lung tumors with acquired resistance to gefitinib or erlotinib

Essential role of Gab1 for signaling by the c-Met receptor in vivo

Hepatocyte growth factor/scatter factor activates the ETS1 transcription factor by a RAS-RAF-MEK-ERK signaling pathway

The semaphorin 4D receptor controls invasive growth by coupling with Met

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82

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5032676

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10.3978/J.ISSN.2305-5839.2015.03.58

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