Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures
about
Adult and embryonic GAD transcripts are spatiotemporally regulated during postnatal development in the rat brainHypercapnic ventilatory response in mice lacking the 65 kDa isoform of Glutamic Acid Decarboxylase (GAD65)Limbic Encephalitis: Potential Impact of Adaptive Autoimmune Inflammation on Neuronal Circuits of the AmygdalaCortical parvalbumin interneurons and cognitive dysfunction in schizophreniaAltered cortical GABA neurotransmission in schizophrenia: insights into novel therapeutic strategiesGABA production by glutamic acid decarboxylase is regulated by a dynamic catalytic loopIncreased expression of GABA(A) receptor beta-subunits in the hippocampus of patients with temporal lobe epilepsyGABA and glutamate in the human brainCortical inhibitory neurons and schizophreniaDevelopmental PCB Exposure Increases Audiogenic Seizures and Decreases Glutamic Acid Decarboxylase in the Inferior Colliculus.Critical period for acoustic preference in mice.Reduced glutamate decarboxylase 65 protein within primary auditory cortex inhibitory boutons in schizophrenia.Deficits in emotional learning and memory in an animal model of schizophreniaCritical role of the 65-kDa isoform of glutamic acid decarboxylase in consolidation and generalization of Pavlovian fear memoryA theory of the transition to critical period plasticity: inhibition selectively suppresses spontaneous activity.Deletion of mouse FXR gene disturbs multiple neurotransmitter systems and alters neurobehaviorStructural and functional analysis of cysteine residues in human glutamate decarboxylase 65 (GAD65) and GAD67.Metabolic distinction between vesicular and cytosolic GABA in cultured GABAergic neurons using 13C magnetic resonance spectroscopy.Role of the proteasome in excitotoxicity-induced cleavage of glutamic acid decarboxylase in cultured hippocampal neurons.Epilepsy in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase.Associations of glutamate decarboxylase genes with initial sensitivity and age-at-onset of alcohol dependence in the Irish Affected Sib Pair Study of Alcohol Dependence.Reduced level of glutamic acid decarboxylase-67 kDa in the prefrontal cortex in major depression.Glutamic acid decarboxylase 65: a link between GABAergic synaptic plasticity in the lateral amygdala and conditioned fear generalizationAn examination of aspartate decarboxylase and glutamate decarboxylase activity in mosquitoes.Permissive proteolytic activity for visual cortical plasticity.Two distinct mechanisms target GAD67 to vesicular pathways and presynaptic clustersCalpain cleavage of brain glutamic acid decarboxylase 65 is pathological and impairs GABA neurotransmission.The physiological roles of vesicular GABA transporter during embryonic development: a study using knockout mice.Mice lacking Gad2 show altered behavioral effects of ethanol, flurazepam and gabaxadol.Lower glutamic acid decarboxylase 65-kDa isoform messenger RNA and protein levels in the prefrontal cortex in schizoaffective disorder but not schizophreniaThe chandelier neuron in schizophrenia.Knockout of GAD65 has major impact on synaptic GABA synthesized from astrocyte-derived glutamine.A factor analysis of global GABAergic gene expression in human brain identifies specificity in response to chronic alcohol and cocaine exposure.Increased anxiety and altered responses to anxiolytics in mice deficient in the 65-kDa isoform of glutamic acid decarboxylase.NF-kappaB/Rel regulates inhibitory and excitatory neuronal function and synaptic plasticity.Tonic excitation or inhibition is set by GABA(A) conductance in hippocampal interneurons.Altered GABA signaling in early life epilepsies.Differential distribution of proteins regulating GABA synthesis and reuptake in axon boutons of subpopulations of cortical interneurons.GAD67-GFP knock-in mice have normal sleep-wake patterns and sleep homeostasis.Neonatal loss of gamma-aminobutyric acid pathway expression after human perinatal brain injury
P2860
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P2860
Mice lacking the 65 kDa isoform of glutamic acid decarboxylase (GAD65) maintain normal levels of GAD67 and GABA in their brains but are susceptible to seizures
description
1996 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
1996 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1996
@ast
im Dezember 1996 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1996/12/24)
@sk
vědecký článek publikovaný v roce 1996
@cs
wetenschappelijk artikel (gepubliceerd op 1996/12/24)
@nl
наукова стаття, опублікована в грудні 1996
@uk
научни чланак (објављен 1996/12/24)
@sr
name
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@ast
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@en
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@nl
type
label
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@ast
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@en
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@nl
prefLabel
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@ast
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@en
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@nl
P2093
P921
P3181
P356
P1476
Mice lacking the 65 kDa isofor ...... ut are susceptible to seizures
@en
P2093
K. Maruyama
N. Kanbara
P304
P3181
P356
10.1006/BBRC.1996.1898
P407
P577
1996-12-24T00:00:00Z