Molecular cloning and biological characterization of NK cell activation-inducing ligand, a counterstructure for CD48
about
Identification of PVR (CD155) and Nectin-2 (CD112) as cell surface ligands for the human DNAM-1 (CD226) activating moleculeEarly expression of triggering receptors and regulatory role of 2B4 in human natural killer cell precursors undergoing in vitro differentiation.NTB-A [correction of GNTB-A], a novel SH2D1A-associated surface molecule contributing to the inability of natural killer cells to kill Epstein-Barr virus-infected B cells in X-linked lymphoproliferative diseaseX-linked lymphoproliferative disease. 2B4 molecules displaying inhibitory rather than activating function are responsible for the inability of natural killer cells to kill Epstein-Barr virus-infected cellsA spectrum of mutations in SH2D1A that causes X-linked lymphoproliferative disease and other Epstein-Barr virus-associated illnessesHuman natural killer cell receptors: insights into their molecular function and structureIdentification of a polyI:C-inducible membrane protein that participates in dendritic cell-mediated natural killer cell activationRequirement of homotypic NK-cell interactions through 2B4(CD244)/CD48 in the generation of NK effector functionsOn guard--activating NK cell receptors.Human natural killer cell receptors and signal transduction.Natural killer cells: a mystery no more.Natural killer cell inhibitory receptors block actin cytoskeleton-dependent recruitment of 2B4 (CD244) to lipid rafts.Evolving role of 2B4/CD244 in T and NK cell responses during virus infection.Dissecting natural killer cell activation pathways through analysis of genetic mutations in human and mouse.Human natural killer cell receptor functions and their implication in diseases.Involvement of activating NK cell receptors and their modulation in pathogen immunityMultiparametric and longitudinal MRI characterization of mild traumatic brain injury in rats.The switch from latent to productive infection in epstein-barr virus-infected B cells is associated with sensitization to NK cell killing.Increased cytotoxicity against B-chronic lymphocytic leukemia by cellular manipulations: potentials for therapeutic use.Enhancement of antigen-specific immunoglobulin G responses by anti-CD48.Protein kinase C is involved in 2B4 (CD244)-mediated cytotoxicity and AP-1 activation in natural killer cells.CD2 is a dominant target for allogeneic responses.
P2860
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P2860
Molecular cloning and biological characterization of NK cell activation-inducing ligand, a counterstructure for CD48
description
1999 nî lūn-bûn
@nan
1999 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
1999年の論文
@ja
1999年論文
@yue
1999年論文
@zh-hant
1999年論文
@zh-hk
1999年論文
@zh-mo
1999年論文
@zh-tw
1999年论文
@wuu
name
Molecular cloning and biologic ...... d, a counterstructure for CD48
@ast
Molecular cloning and biologic ...... d, a counterstructure for CD48
@en
Molecular cloning and biologic ...... d, a counterstructure for CD48
@nl
type
label
Molecular cloning and biologic ...... d, a counterstructure for CD48
@ast
Molecular cloning and biologic ...... d, a counterstructure for CD48
@en
Molecular cloning and biologic ...... d, a counterstructure for CD48
@nl
prefLabel
Molecular cloning and biologic ...... d, a counterstructure for CD48
@ast
Molecular cloning and biologic ...... d, a counterstructure for CD48
@en
Molecular cloning and biologic ...... d, a counterstructure for CD48
@nl
P2093
P2860
P1476
Molecular cloning and biologic ...... d, a counterstructure for CD48
@en
P2093
B M Macduff
D L Parshley
G Trinchieri
L Cassiano
M Petersen
R G Goodwin
P2860
P304
P356
10.1002/(SICI)1521-4141(199911)29:11<3466::AID-IMMU3466>3.0.CO;2-9
P407
P577
1999-11-01T00:00:00Z