Immunology: hepatitis A virus link to atopic disease - Wikidata - awgldk - TriplyDB

Immunology: hepatitis A virus link to atopic disease

Immunology: hepatitis A virus link to atopic disease

http://www.wikidata.org/entity/Q28208230

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Too clean, or not too clean: the hygiene hypothesis and home hygiene Large-scale candidate gene analysis of spontaneous clearance of hepatitis C virus Immunoglobulin A (IgA) is a natural ligand of hepatitis A virus cellular receptor 1 (HAVCR1), and the association of IgA with HAVCR1 enhances virus-receptor interactions Children's Inter-Individual Variability and Asthma Development TIM polymorphisms--genetics and function The hygiene hypothesis: an explanation for the increased frequency of insulin-dependent diabetes Structures of T Cell Immunoglobulin Mucin Protein 4 Show a Metal-Ion-Dependent Ligand Binding Site where Phosphatidylserine Binds T Cell/Transmembrane, Ig, and Mucin-3 Allelic Variants Differentially Recognize Phosphatidylserine and Mediate Phagocytosis of Apoptotic Cells TIM-1 induces T cell activation and inhibits the development of peripheral tolerance Apoptotic cells activate NKT cells through T cell Ig-like mucin-like-1 resulting in airway hyperreactivity Interactions between commensal intestinal bacteria and the immune system Tim-2 regulates T helper type 2 responses and autoimmunity Soothing signals: transplacental transmission of resistance to asthma and allergy.Growth of hepatitis A virus in a mouse liver cell line Chronic virus infections supress atopy but not asthma in a set of children from a large Latin American city: a cross-section study.Antagonism of TIM-1 blocks the development of disease in a humanized mouse model of allergic asthma.TIM-family proteins inhibit HIV-1 release.TIM-4 expressed on APCs induces T cell expansion and survival.Asthma genetics 2006: the long and winding road to gene discovery.A polymorphism in TIM1 is associated with susceptibility to severe hepatitis A virus infection in humans Association between polymorphisms in the promoter region of T cell immunoglobulin and mucin domain-3 and myasthenia gravis-associated thymoma.Tim-1 stimulation of dendritic cells regulates the balance between effector and regulatory T cells.Time to draw breath: asthma-susceptibility genes are identified.Future research directions in asthma: an NHLBI Working Group report.Th1/Th2 cells in inflammatory disease states: therapeutic implications.The immunogenetics of asthma and eczema: a new focus on the epithelium.Defect in regulatory B-cell function and development of systemic autoimmunity in T-cell Ig mucin 1 (Tim-1) mucin domain-mutant mice.Distinct Trafficking of Cell Surface and Endosomal TIM-1 to the Immune Synapse.Differential engagement of Tim-1 during activation can positively or negatively costimulate T cell expansion and effector function.The expanding realm of heterologous immunity: friend or foe?Computational analysis of multimorbidity between asthma, eczema and rhinitis.TIM-2 is expressed on B cells and in liver and kidney and is a receptor for H-ferritin endocytosis.The regulation of allergy and asthma.Gene-environment interactions in asthma: with apologies to William of Ockham.Integrating genomic and clinical medicine: searching for susceptibility genes in complex lung diseases TIM-1 and TIM-3 proteins in immune regulation.Asthma and atopic dermatitis are associated with increased risk of clinical Plasmodium falciparum malaria.T-cell immunoglobulin and mucin domain 1 deficiency eliminates airway hyperreactivity triggered by the recognition of airway cell death.TIM family proteins promote the lysosomal degradation of the nuclear receptor NUR77.Control and regulation of peripheral tolerance in allergic inflammatory disease: therapeutic consequences

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P2860

Immunology: hepatitis A virus link to atopic disease

http://www.wikidata.org/entity/Q28208230

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2003 nî lūn-bûn

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2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2003 թվականի հոտեմբերին հրատարակված գիտական հոդված

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2003年の論文

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Immunology: hepatitis A virus link to atopic disease

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Immunology: hepatitis A virus link to atopic disease

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Cengiz Cinnioglu

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