Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits - Wikidata - awgldk - TriplyDB

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

http://www.wikidata.org/entity/Q28246677

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Clinical and Neurobiological Relevance of Current Animal Models of Autism Spectrum Disorders.The Crossroads of Synaptic Growth Signaling, Membrane Traffic and Neurological Disease: Insights from Drosophila Characterizing autism spectrum disorders by key biochemical pathways.The neurology of mTOR Development of thalamocortical connections between the mediodorsal thalamus and the prefrontal cortex and its implication in cognition Dendritic spine dysgenesis in autism related disorders On the Teneurin track: a new synaptic organization molecule emerges The Indispensable Roles of Microglia and Astrocytes during Brain Development A fly's view of neuronal remodeling Advancing the understanding of autism disease mechanisms through genetics LTD-like molecular pathways in developmental synaptic pruning A Subset of Autism-Associated Genes Regulate the Structural Stability of Neurons.From High Intellectual Potential to Asperger Syndrome: Evidence for Differences and a Fundamental Overlap-A Systematic Review The neuroanatomy of autism - a developmental perspective FOXG1-Dependent Dysregulation of GABA/Glutamate Neuron Differentiation in Autism Spectrum Disorders Maternal immune activation: Implications for neuropsychiatric disorders.Tricornered Kinase Regulates Synapse Development by Regulating the Levels of Wiskott-Aldrich Syndrome Protein Reelin Regulates the Maturation of Dendritic Spines, Synaptogenesis and Glial Ensheathment of Newborn Granule Cells.Hyperconnectivity of prefrontal cortex to amygdala projections in a mouse model of macrocephaly/autism syndrome Piecing together a different picture: A host of new studies on autism have begun decoding the longstanding puzzle of its causes.Activity-Dependent Synaptic Refinement: New Insights from Drosophila.Diversity and Versatility of Phagocytosis: Roles in Innate Immunity, Tissue Remodeling, and Homeostasis.Genetic variants in the transcription regulatory region of MEGF10 are associated with autism in Chinese Han population.The Pathophysiological Role of Microglia in Dynamic Surveillance, Phagocytosis and Structural Remodeling of the Developing CNS.Folinic acid improves verbal communication in children with autism and language impairment: a randomized double-blind placebo-controlled trial.Developmental changes in large-scale network connectivity in autism.Dendrite pathology and neurodegeneration: focus on mTOR Tuberous Sclerosis: A New Frontier in Targeted Treatment of Autism Evidence for contribution of common genetic variants within chromosome 8p21.2-8p21.1 to restricted and repetitive behaviors in autism spectrum disorders.An Autism-Linked Mutation Disables Phosphorylation Control of UBE3A.Early Behavioral Abnormalities and Perinatal Alterations of PTEN/AKT Pathway in Valproic Acid Autism Model Mice.Standardization of capillary electrophoresis for diagnosis of fragile X syndrome in the Brazilian public health system.Disruption of an Evolutionarily Novel Synaptic Expression Pattern in Autism PACAP Protects Adult Neural Stem Cells from the Neurotoxic Effect of Ketamine Associated with Decreased Apoptosis, ER Stress and mTOR Pathway Activation.Abnormal glutamate release in aged BTBR mouse model of autism.Local brain connectivity across development in autism spectrum disorder: A cross-sectional investigation.Corticospinal tract anatomy and functional connectivity of primary motor cortex in autism.Modeling developmental neuropsychiatric disorders with iPSC technology: challenges and opportunities Control of spine maturation and pruning through proBDNF synthesized and released in dendrites.Collybistin binds and inhibits mTORC1 signaling: a potential novel mechanism contributing to intellectual disability and autism.

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P2860

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

http://www.wikidata.org/entity/Q28246677

description

2014 nî lūn-bûn

@nan

2014 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած

@hyw

2014 թվականի սեպտեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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type

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Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

@en

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

@nl

prefLabel

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

@ast

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

@en

Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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J.NEURON.2014.07.040

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Loss of mTOR-dependent macroautophagy causes autistic-like synaptic pruning deficits

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Ai Yamamoto

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Alexander Sosunov

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Andrew J Dwork

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Bradley S Peterson

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Candace Castagna

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Ellen Kanter

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Gorazd Rosoklija

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Guomei Tang

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James Goldman

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Kathryn Gudsnuk

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P2860

Trajectory of development in adolescents and adults with autism

Homeostatic levels of p62 control cytoplasmic inclusion body formation in autophagy-deficient mice

Multiple autism-linked genes mediate synapse elimination via proteasomal degradation of a synaptic scaffold PSD-95

mTOR signaling: at the crossroads of plasticity, memory and disease

Reversal of learning deficits in a Tsc2+/- mouse model of tuberous sclerosis

Neuroligins/LRRTMs prevent activity- and Ca2+/calmodulin-dependent synapse elimination in cultured neurons

Common circuit defect of excitatory-inhibitory balance in mouse models of autism.

Cellular and synaptic network defects in autism

Suppression of basal autophagy in neural cells causes neurodegenerative disease in mice

Loss of autophagy in the central nervous system causes neurodegeneration in mice

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