about
Diva, a Bcl-2 homologue that binds directly to Apaf-1 and induces BH3-independent cell deathBoo, a novel negative regulator of cell death, interacts with Apaf-1.Ordering the cytochrome c-initiated caspase cascade: hierarchical activation of caspases-2, -3, -6, -7, -8, and -10 in a caspase-9-dependent mannerThe CED-4-homologous protein FLASH is involved in Fas-mediated activation of caspase-8 during apoptosisBtf, a novel death-promoting transcriptional repressor that interacts with Bcl-2-related proteins.F1Aalpha, a death receptor-binding protein homologous to the Caenorhabditis elegans sex-determining protein, FEM-1, is a caspase substrate that mediates apoptosisInvolvement of 5'-flanking kB-like sites within bcl-x gene in silica-induced bcl-x expressionMAP-1, a novel proapoptotic protein containing a BH3-like motif that associates with Bax through its Bcl-2 homology domainsBcl-rambo, a novel Bcl-2 homologue that induces apoptosis via its unique C-terminal extensionThe Caenorhabditis elegans sex-determining protein FEM-2 and its human homologue, hFEM-2, are Ca2+/calmodulin-dependent protein kinase phosphatases that promote apoptosisIdentification of the hypoxia-inducible factor 1 alpha-responsive HGTD-P gene as a mediator in the mitochondrial apoptotic pathwayBOK and NOXA are essential mediators of p53-dependent apoptosisNF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosisWISP-1 attenuates p53-mediated apoptosis in response to DNA damage through activation of the Akt kinaseSuppression of apoptosis signal-regulating kinase 1-induced cell death by 14-3-3 proteinsMitochondrial release of caspase-2 and -9 during the apoptotic processBH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell deathA surface groove essential for viral Bcl-2 function during chronic infection in vivo.Interaction of Alzheimer's presenilin-1 and presenilin-2 with Bcl-X(L). A potential role in modulating the threshold of cell deathInduction of apoptosis in prostate carcinoma cells by BH3 peptides which inhibit Bak/Bcl-2 interactionsHealth and cellular impacts of air pollutants: from cytoprotection to cytotoxicityAutomatic morphological subtyping reveals new roles of caspases in mitochondrial dynamicsThe anti-apoptotic Bcl-x(L) protein, a new piece in the puzzle of cytochrome c interactomeDifferences in bcl-2- and bax-independent function in regulating apoptosis in sensory neuron populationsThe mitochondrial permeability transition pore and its role in cell deathDNA IR-Double Strand Breaks (DSBs) and cellular response via ATMLithium inhibits aluminum-induced apoptosis in rabbit hippocampus, by preventing cytochrome c translocation, Bcl-2 decrease, Bax elevation and caspase-3 activation.Formation of nuclear Bax/p53 complexes is associated with chemotherapy induced apoptosis.Analysis of the composition, assembly kinetics and activity of native Apaf-1 apoptosomes.Pro-apoptotic apoptosis protease-activating factor 1 (Apaf-1) has a cytoplasmic localization distinct from Bcl-2 or Bcl-x(L).Apoptosis induced by cadmium in human lymphoma U937 cells through Ca2+-calpain and caspase-mitochondria- dependent pathways.Hypoxia induces apoptosis by caspase activation accompanying cytochrome C release from mitochondria in MC3T3E1 osteoblasts. p38 MAPK is related in hypoxia-induced apoptosis.The N-terminus and alpha-5, alpha-6 helices of the pro-apoptotic protein Bax, modulate functional interactions with the anti-apoptotic protein Bcl-xL.Atorvastatin induces apoptosis by a caspase-9-dependent pathway: an in vitro study on activated rat hepatic stellate cells.Targeted disruption of caspase genes in mice: what they tell us about the functions of individual caspases in apoptosis.Anticancer activity of Nelumbo nucifera stamen extract in human colon cancer HCT-116 cells in vitro.Regulation of caspase activation in apoptosis: implications in pathogenesis and treatment of disease.The potential for monocyte-mediated immunotherapy during infection and malignancy. Part I: apoptosis induction and cytotoxic mechanisms.Mechanisms of apoptosis by c-Myc.Immune evasion by adenoviruses.
P2860
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P2860
description
1998 nî lūn-bûn
@nan
1998 թուականի Մարտին հրատարակուած գիտական յօդուած
@hyw
1998 թվականի մարտին հրատարակված գիտական հոդված
@hy
1998年の論文
@ja
1998年論文
@yue
1998年論文
@zh-hant
1998年論文
@zh-hk
1998年論文
@zh-mo
1998年論文
@zh-tw
1998年论文
@wuu
name
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@ast
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@en
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@nl
type
label
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@ast
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@en
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@nl
prefLabel
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@ast
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@en
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@nl
P2093
P356
P1476
Caspase-9, Bcl-XL, and Apaf-1 form a ternary complex
@en
P2093
P304
P356
10.1074/JBC.273.10.5841
P407
P577
1998-03-06T00:00:00Z