Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
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The Role of Inflammatory Mediators in the Pathogenesis of Alzheimer's DiseaseInflammatory monocytes and the pathogenesis of viral encephalitisTherapeutic effects of glatiramer acetate and grafted CD115⁺ monocytes in a mouse model of Alzheimer's disease.Anti-aggregating effect of the naturally occurring dipeptide carnosine on aβ1-42 fibril formationDealing with Danger in the CNS: The Response of the Immune System to Injury.Angiotensin-converting enzyme overexpression in myelomonocytes prevents Alzheimer's-like cognitive declineAlzheimer's disease: pathophysiology and applications of magnetic nanoparticles as MRI theranostic agents.CCL2 affects β-amyloidosis and progressive neurocognitive dysfunction in a mouse model of Alzheimer's disease.Linking cardiometabolic disorders to sporadic Alzheimer's disease: a perspective on potential mechanisms and mediators.Apolipoprotein E isoform-dependent microglia migrationTyrosine Kinase Inhibition Regulates Early Systemic Immune Changes and Modulates the Neuroimmune Response in α-SynucleinopathyBone marrow-derived microglia infiltrate into the paraventricular nucleus of chronic psychological stress-loaded mice.Telomere length is age-dependent and reduced in monocytes of Alzheimer patients.A panel of nine cerebrospinal fluid biomarkers may identify patients with atypical parkinsonian syndromes.Both Cerebral and Hematopoietic Deficiencies in CCR2 Result in Uncontrolled Herpes Simplex Virus Infection of the Central Nervous System in Mice.Heparanase overexpression impairs inflammatory response and macrophage-mediated clearance of amyloid-β in murine brainThe CCL2 synthesis inhibitor bindarit targets cells of the neurovascular unit, and suppresses experimental autoimmune encephalomyelitis.Elevated plasma MCP-1 concentration following traumatic brain injury as a potential "predisposition" factor associated with an increased risk for subsequent development of Alzheimer's disease.Influence of the physiochemical properties of superparamagnetic iron oxide nanoparticles on amyloid β protein fibrillation in solution.T Lymphocytes and Inflammatory Mediators in the Interplay between Brain and Blood in Alzheimer's Disease: Potential Pools of New Biomarkers.Targeting the pro-inflammatory factor CCL2 (MCP-1) with Bindarit for influenza A (H7N9) treatment.Monocytes and Alzheimer's diseaseThe Potential Role of Chemokines in Alzheimer's Disease Pathogenesis.Microglial Aβ receptors in Alzheimer's disease.Migration of blood cells to β-amyloid plaques in Alzheimer's disease.Microglia-blood vessel interactions: a double-edged sword in brain pathologies.Spontaneous development of autoimmune uveitis Is CCR2 dependent.Dogs with Cognitive Dysfunction as a Spontaneous Model for Early Alzheimer's Disease: A Translational Study of Neuropathological and Inflammatory Markers.There is no correlation between peripheral inflammation and cognitive status at diagnosis in Alzheimer's disease.Inhibition of hypothalamic leukemia inhibitory factor exacerbates diet-induced obesity phenotype.MCP-1/CCR2 signaling-mediated astrocytosis is accelerated in a transgenic mouse model of SOD1-mutated familial ALSPolysaccharides from Ganoderma lucidum attenuate microglia-mediated neuroinflammation and modulate microglial phagocytosis and behavioural response.Chronic stress enhances microglia activation and exacerbates death of nigral dopaminergic neurons under conditions of inflammation.Downregulation of CCL2 induced by the upregulation of microRNA-206 is associated with the severity of HEV71 encephalitis.Association of CXCL1 promoter polymorphism with ischaemic stroke in Korean population.Cerebrospinal Fluid C-C Motif Chemokine Ligand 2 Correlates with Brain Atrophy and Cognitive Impairment in Alzheimer's Disease.Microglial interactions with the neurovascular system in physiology and pathology.Peripheral administration of the soluble TNF inhibitor XPro1595 modifies brain immune cell profiles, decreases beta-amyloid plaque load, and rescues impaired long-term potentiation in 5xFAD mice.Effects of myeloablation, peripheral chimerism, and whole-body irradiation on the entry of bone marrow-derived cells into the brain.Serotonin 2B receptor slows disease progression and prevents degeneration of spinal cord mononuclear phagocytes in amyotrophic lateral sclerosis.
P2860
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P2860
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
description
2010 nî lūn-bûn
@nan
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2010年の論文
@ja
2010年論文
@yue
2010年論文
@zh-hant
2010年論文
@zh-hk
2010年論文
@zh-mo
2010年論文
@zh-tw
2010年论文
@wuu
name
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@ast
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@en
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@nl
type
label
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@ast
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@en
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@nl
prefLabel
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@ast
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@en
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@nl
P2860
P1476
Mechanisms of mononuclear phagocyte recruitment in Alzheimer's disease
@en
P2093
Suzanne E Hickman
P2860
P304
P356
10.2174/187152710791011982
P407
P577
2010-04-01T00:00:00Z