Defective transcription-coupled repair of oxidative base damage in Cockayne syndrome patients from XP group G
about
Conserved residues of human XPG protein important for nuclease activity and function in nucleotide excision repairHuman exonuclease 1 functionally complements its yeast homologues in DNA recombination, RNA primer removal, and mutation avoidanceThe ATPase domain but not the acidic region of Cockayne syndrome group B gene product is essential for DNA repairOxidative Decay of DNA3-Methyladenine-DNA glycosylase (MPG protein) interacts with human RAD23 proteinsStrong functional interactions of TFIIH with XPC and XPG in human DNA nucleotide excision repair, without a preassembled repairosomeCooperation of the Cockayne syndrome group B protein and poly(ADP-ribose) polymerase 1 in the response to oxidative stress.Cockayne syndrome group B protein enhances elongation by RNA polymerase IIIn vitro repair of oxidative DNA damage by human nucleotide excision repair system: possible explanation for neurodegeneration in xeroderma pigmentosum patientsIdentification and characterization of a human DNA glycosylase for repair of modified bases in oxidatively damaged DNACockayne syndrome: defective repair of transcription?Long-patch DNA repair synthesis during base excision repair in mammalian cellsMolecular analysis of mutations in the CSB (ERCC6) gene in patients with Cockayne syndromeXeroderma pigmentosum group C protein interacts physically and functionally with thymine DNA glycosylaseRecruitment of the putative transcription-repair coupling factor CSB/ERCC6 to RNA polymerase II elongation complexesMethylated DNA-binding domain 1 and methylpurine-DNA glycosylase link transcriptional repression and DNA repair in chromatinA common mutational pattern in Cockayne syndrome patients from xeroderma pigmentosum group G: implications for a second XPG functionIncreased apoptosis, p53 up-regulation, and cerebellar neuronal degeneration in repair-deficient Cockayne syndrome miceNucleotide Excision Repair, Genome Stability, and Human Disease: New Insight from Model SystemsMitochondrial inhibition of uracil-DNA glycosylase is not mutagenicStructure and potential mutagenicity of new hydantoin products from guanosine and 8-oxo-7,8-dihydroguanine oxidation by transition metals.Somatic mutations in aging, cancer and neurodegenerationRepair of oxidized bases in DNA bubble structures by human DNA glycosylases NEIL1 and NEIL2RNA polymerase II elongation complexes containing the Cockayne syndrome group B protein interact with a molecular complex containing the transcription factor IIH components xeroderma pigmentosum B and p62A multistep damage recognition mechanism for global genomic nucleotide excision repairEffect of thymine glycol on transcription elongation by T7 RNA polymerase and mammalian RNA polymerase IIIdentification of the XPG region that causes the onset of Cockayne syndrome by using Xpg mutant mice generated by the cDNA-mediated knock-in methodDifferent effects of CSA and CSB deficiency on sensitivity to oxidative DNA damagePostnatal growth failure, short life span, and early onset of cellular senescence and subsequent immortalization in mice lacking the xeroderma pigmentosum group G geneComplexities of the DNA base excision repair pathway for repair of oxidative DNA damageDetection and determination of oligonucleotide triplex formation-mediated transcription-coupled DNA repair in HeLa nuclear extracts.Early postnatal ataxia and abnormal cerebellar development in mice lacking Xeroderma pigmentosum Group A and Cockayne syndrome Group B DNA repair genesOxidative damage-induced PCNA complex formation is efficient in xeroderma pigmentosum group A but reduced in Cockayne syndrome group B cells.Xeroderma pigmentosum group A protein loads as a separate factor onto DNA lesions.Use of DNA repair-deficient XPA transgenic mice in short-term carcinogenicity testing.Xeroderma pigmentosum group G with severe neurological involvement and features of Cockayne syndrome in infancy.Requirement for yeast RAD26, a homolog of the human CSB gene, in elongation by RNA polymerase IIDifferential requirement for the ATPase domain of the Cockayne syndrome group B gene in the processing of UV-induced DNA damage and 8-oxoguanine lesions in human cells.Transcription-coupled repair of DNA damage: unanticipated players, unexpected complexities.Rad26p, a transcription-coupled repair factor, is recruited to the site of DNA lesion in an elongating RNA polymerase II-dependent manner in vivo
P2860
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P2860
Defective transcription-coupled repair of oxidative base damage in Cockayne syndrome patients from XP group G
description
1997 nî lūn-bûn
@nan
1997 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
1997年の論文
@ja
1997年学术文章
@wuu
1997年学术文章
@zh-cn
1997年学术文章
@zh-hans
1997年学术文章
@zh-my
1997年学术文章
@zh-sg
1997年學術文章
@yue
name
Defective transcription-couple ...... drome patients from XP group G
@ast
Defective transcription-couple ...... drome patients from XP group G
@en
Defective transcription-couple ...... drome patients from XP group G
@nl
type
label
Defective transcription-couple ...... drome patients from XP group G
@ast
Defective transcription-couple ...... drome patients from XP group G
@en
Defective transcription-couple ...... drome patients from XP group G
@nl
prefLabel
Defective transcription-couple ...... drome patients from XP group G
@ast
Defective transcription-couple ...... drome patients from XP group G
@en
Defective transcription-couple ...... drome patients from XP group G
@nl
P2093
P1433
P1476
Defective transcription-couple ...... drome patients from XP group G
@en
P2093
P K Cooper
S A Leadon
S G Clarkson
T Nouspikel
P356
10.1126/SCIENCE.275.5302.990
P407
P577
1997-02-14T00:00:00Z