EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells - Wikidata - awgldk - TriplyDB

EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells

EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells

http://www.wikidata.org/entity/Q28360903

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Recent advances on tea polyphenols Regulation of survival, proliferation, invasion, angiogenesis, and metastasis of tumor cells through modulation of inflammatory pathways by nutraceuticals Dietary antioxidants protect gut epithelial cells from oxidant-induced apoptosis Novel Investigations of Flavonoids as Chemopreventive Agents for Hepatocellular Carcinoma Broad targeting of angiogenesis for cancer prevention and therapy Inhibitory effect of arsenic trioxide on angiogenesis and expression of vascular endothelial growth factor in gastric cancer Green tea extract inhibits proliferation of uterine leiomyoma cells in vitro and in nude mice Inhibitory effect of epigallocatechin gallate (EGCG), resveratrol, and curcumin on proliferation of human retinal pigment epithelial cells in vitro.Steep your genes in health: drink tea.Epigallocatechin-3-gallate suppresses proinflammatory cytokines and chemokines induced by Toll-like receptor 9 agonists in prostate cancer cells.Pro-oxidative activities and dose-response relationship of (-)-epigallocatechin-3-gallate in the inhibition of lung cancer cell growth: a comparative study in vivo and in vitro Analysis of the anti-tumor effect of cetuximab using protein kinetics and mouse xenograft models.Hypoxia-inducible factor-1 activation by (-)-epicatechin gallate: potential adverse effects of cancer chemoprevention with high-dose green tea extracts.(-)-Epigallocatechin-3-gallate inhibits growth of gastric cancer by reducing VEGF production and angiogenesis.Survivin knockdown increased anti-cancer effects of (-)-epigallocatechin-3-gallate in human malignant neuroblastoma SK-N-BE2 and SH-SY5Y cells Inhibition of tumour invasion and angiogenesis by epigallocatechin gallate (EGCG), a major component of green tea.Epicatechins Purified from Green Tea (Camellia sinensis) Differentially Suppress Growth of Gender-Dependent Human Cancer Cell Lines.(-)-Epigallocatechin-3-gallate inhibits VEGF expression induced by IL-6 via Stat3 in gastric cancer.Potential of the bioflavonoids in the prevention/treatment of ocular disorders Implications of Green Tea and Its Constituents in the Prevention of Cancer via the Modulation of Cell Signalling Pathway.Green tea polyphenols as proteasome inhibitors: implication in chemoprevention.Chemoprevention in gastrointestinal physiology and disease. Anti-inflammatory approaches for colorectal cancer chemoprevention Epigenetic modulation of the retinoid X receptor alpha by green tea in the azoxymethane-Apc Min/+ mouse model of intestinal cancer.Campomanesia adamantium Peel Extract in Antidiarrheal Activity: The Ability of Inhibition of Heat-Stable Enterotoxin by Polyphenols.Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer The HMG-CoA reductase pathway, statins and angioprevention.Theaflavin-3, 3'-digallate decreases human ovarian carcinoma OVCAR-3 cell-induced angiogenesis via Akt and Notch-1 pathways, not via MAPK pathways.Overview of major classes of plant-derived anticancer drugs EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1α and NFκB, and VEGF expression.Green tea catechins inhibit angiogenesis through suppression of STAT3 activation.Predicting the physiological relevance of in vitro cancer preventive activities of phytochemicals.Cancer preventive mechanisms of the green tea polyphenol (-)-epigallocatechin-3-gallate.Therapeutic efficacy of topical epigallocatechin gallate in murine dry eye.Plants and their active compounds: natural molecules to target angiogenesis.Tea polyphenols, their biological effects and potential molecular targets.The protective effects of green tea extract against L-arginine toxicity to cultured human mesangial cells Expanding circle of inhibition: small-molecule inhibitors of Bcl-2 as anticancer cell and antiangiogenic agents.Natural products and colon cancer: current status and future prospects.Molecular mechanisms of green tea polyphenols Crystal engineering of green tea epigallocatechin-3-gallate (EGCg) cocrystals and pharmacokinetic modulation in rats.

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EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells

http://www.wikidata.org/entity/Q28360903

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2001 nî lūn-bûn

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British Journal of Cancer

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B A Shin

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B W Ahn

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C D Bucana

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Y D Jung

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P2860

Blockade of the MAP kinase pathway suppresses growth of colon tumors in vivo

How MAP kinases are regulated

Angiogenesis in cancer, vascular, rheumatoid and other disease

The mitogen-activated protein kinase signal transduction pathway

Regulation by vascular endothelial growth factor of human colon cancer tumorigenesis in a mouse model of experimental liver metastasis

Polyphenols as cancer chemopreventive agents.

Epigallocathechin-3 gallate selectively inhibits the PDGF-BB-induced intracellular signaling transduction pathway in vascular smooth muscle cells and inhibits transformation of sis-transfected NIH 3T3 fibroblasts and human glioblastoma cells (A172).

Expression of vascular endothelial growth factor and its receptor, KDR, correlates with vascularity, metastasis, and proliferation of human colon cancer.

Tea and cancer.

Extracellular signal-regulated kinase activation is required for up-regulation of vascular endothelial growth factor by serum starvation in human colon carcinoma cells.

P2888

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844-50

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2885130

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10.1054/BJOC.2000.1691

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6

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84

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12073746

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1019321821

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11259102

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2363808

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