EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1α and NFκB, and VEGF expression. - Wikidata - awgldk - TriplyDB

EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1α and NFκB, and VEGF expression.

EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1α and NFκB, and VEGF expression.

http://www.wikidata.org/entity/Q36831607

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Epigallocatechin Gallate Nanodelivery Systems for Cancer Therapy Chemoprevention of Breast Cancer by Dietary Polyphenols Nuclear receptors and epigenetic regulation: opportunities for nutritional targeting and disease prevention Meroxest improves the prognosis of immunocompetent C57BL/6 mice with allografts of E0771 mouse breast tumor cells.Effects of physiological levels of the green tea extract epigallocatechin-3-gallate on breast cancer cells Breast cancer chemopreventive and chemotherapeutic effects of Camellia Sinensis (green tea): an updated review Green tea compounds in breast cancer prevention and treatment HIF-1 alpha overexpression correlates with poor overall survival and disease-free survival in gastric cancer patients post-gastrectomy.Implications of Green Tea and Its Constituents in the Prevention of Cancer via the Modulation of Cell Signalling Pathway.Insight into the mechanism of polyphenols on the activity of HMGR by molecular docking.Green Tea Polyphenol Induces Changes in Cancer-Related Factors in an Animal Model of Bladder Cancer Theaflavin-3, 3'-digallate decreases human ovarian carcinoma OVCAR-3 cell-induced angiogenesis via Akt and Notch-1 pathways, not via MAPK pathways.Epigallocatechin Gallate-Mediated Alteration of the MicroRNA Expression Profile in 5α-Dihydrotestosterone-Treated Human Dermal Papilla Cells Green tea polyphenol tailors cell adhesivity of RGD displaying surfaces: multicomponent models monitored optically.Emerging importance of dietary phytochemicals in fight against cancer: Role in targeting cancer stem cells.Epigallocatechin gallate sensitizes cisplatin-resistant oral cancer CAR cell apoptosis and autophagy through stimulating AKT/STAT3 pathway and suppressing multidrug resistance 1 signaling.Targeting cancer stem cells and signaling pathways by phytochemicals: Novel approach for breast cancer therapy Screening of multi-targeted natural compounds for receptor tyrosine kinases inhibitors and biological evaluation on cancer cell lines, in silico and in vitro.Biophysical characteristics of proteins and living cells exposed to the green tea polyphenol epigallocatechin-3-gallate (EGCg): review of recent advances from molecular mechanisms to nanomedicine and clinical trials.Development of novel radiochemotherapy approaches targeting prostate tumor progenitor cells using nanohybrids.The combined use of Camellia sinensis and metronomic zoledronic acid in a breast cancer-induced osteolysis mouse model.Role of angiogenic factors of herbal origin in regulation of molecular pathways that control tumor angiogenesis.Epigenetic approach for angiostatic therapy: promising combinations for cancer treatment.Changes in the oxidative stress/anti-oxidant system after exposure to sulfur mustard and antioxidant strategies in the therapy, a review.Enhanced uptake and transport of (+)-catechin and (-)-epigallocatechin gallate in niosomal formulation by human intestinal Caco-2 cells.Therapeutic Potential of Epigallocatechin Gallate Nanodelivery Systems Effect of antioxidants supplementation on aging and longevity Combination curcumin and (-)-epigallocatechin-3-gallate inhibits colorectal carcinoma microenvironment-induced angiogenesis by JAK/STAT3/IL-8 pathway.Translational opportunities for broad-spectrum natural phytochemicals and targeted agent combinations in breast cancer.Consumption of green tea epigallocatechin-3-gallate enhances systemic immune response, antioxidative capacity and HPA axis functions in aged male swiss albino mice.Antiangiogenic Effect of Flavonoids and Chalcones: An Update.Epigallocatechin-3-Gallate Inhibition of Myeloperoxidase and Its Counter-Regulation by Dietary Iron and Lipocalin 2 in Murine Model of Gut Inflammation.EGCG inhibited bladder cancer T24 and 5637 cell proliferation and migration via PI3K/AKT pathway.Phytochemicals: Current strategies for treating breast cancer.High-Resolution Adhesion Kinetics of EGCG-Exposed Tumor Cells on Biomimetic Interfaces: Comparative Monitoring of Cell Viability Using Label-Free Biosensor and Classic End-Point Assays.Matcha green tea (MGT) inhibits the propagation of cancer stem cells (CSCs), by targeting mitochondrial metabolism, glycolysis and multiple cell signalling pathways The Methylation Status of the Epigenome: Its Emerging Role in the Regulation of Tumor Angiogenesis and Tumor Growth, and Potential for Drug Targeting Dietary phytochemicals in breast cancer research: anticancer effects and potential utility for effective chemoprevention

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EGCG, a major green tea catechin suppresses breast tumor angiogenesis and growth via inhibiting the activation of HIF-1α and NFκB, and VEGF expression.

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EGCG, a major green tea catech ...... and NFκB, and VEGF expression.

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Edmund Chinchar

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Emily Y Thomas

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Ivy Pei

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Jian-Wei Gu

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Hypoxia and oxidative stress in breast cancer. Oxidative stress: its effects on the growth, metastatic potential and response to therapy of breast cancer.

Tumor angiogenesis: therapeutic implications

The biology of VEGF and its receptors

Green tea and risk of breast cancer in Asian Americans

Ethanol stimulates tumor progression and expression of vascular endothelial growth factor in chick embryos

EGCG, a major component of green tea, inhibits tumour growth by inhibiting VEGF induction in human colon carcinoma cells

Production of large amounts of hydrogen peroxide by human tumor cells

Inhibition of prostate carcinogenesis in TRAMP mice by oral infusion of green tea polyphenols.

High tumor levels of vascular endothelial growth factor predict poor response to systemic therapy in advanced breast cancer.

The effect of menopause and hysterectomy on systemic vascular endothelial growth factor in women undergoing surgery for breast cancer.

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