An examination of the secretion-like coupling model for the activation of the Ca2+ release-activated Ca2+ current I(CRAC) in RBL-1 cells
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Coupling of STIM1 to store-operated Ca2+ entry through its constitutive and inducible movement in the endoplasmic reticulum.Adenophostin A and ribophostin, but not inositol 1,4,5-trisphosphate or manno-adenophostin, activate the Ca2+ release-activated Ca2+ current, I(CRAC), in weak intracellular Ca2+ bufferArf-1 (ADP-ribosylation factor-1) is involved in the activation of a mammalian Na+-selective currentCa2+ store depletion causes STIM1 to accumulate in ER regions closely associated with the plasma membrane.Some assembly required: constructing the elementary units of store-operated Ca2+ entry.STIMulating store-operated Ca(2+) entry.Molecular basis of calcium signaling in lymphocytes: STIM and ORAIA zinc-sensing receptor triggers the release of intracellular Ca2+ and regulates ion transport.Regulation of Ca2+ signaling with particular focus on mast cells.Pharmacology of store-operated calcium channelsCa2+ waves initiate antigen-stimulated Ca2+ responses in mast cellsMitofusin 2 regulates STIM1 migration from the Ca2+ store to the plasma membrane in cells with depolarized mitochondria.STIM and Orai: the long-awaited constituents of store-operated calcium entryKey role for store-operated Ca2+ channels in activating gene expression in human airway bronchial epithelial cells.Regulation of store-operated calcium entry during cell division.Recent breakthroughs in the molecular mechanism of capacitative calcium entry (with thoughts on how we got here)New molecular players in capacitative Ca2+ entry.Herpes simplex virus triggers activation of calcium-signaling pathwaysRole of the microtubule cytoskeleton in the function of the store-operated Ca2+ channel activator STIM1.Complex actions of 2-aminoethyldiphenyl borate on store-operated calcium entry.Methods for studying store-operated calcium entry.Pancreatic protease activation by alcohol metabolite depends on Ca2+ release via acid store IP3 receptorsPharmacology of ORAI channels as a tool to understand their physiological functions.Cytoskeleton in mast cell signaling.Calcium microdomains at the immunological synapse: how ORAI channels, mitochondria and calcium pumps generate local calcium signals for efficient T-cell activation.Store-operated Ca2+ entry depends on mitochondrial Ca2+ uptake2-Aminoethoxydiphenyl borate directly facilitates and indirectly inhibits STIM1-dependent gating of CRAC channels.Local Ca2+ influx through Ca2+ release-activated Ca2+ (CRAC) channels stimulates production of an intracellular messenger and an intercellular pro-inflammatory signal.Close functional coupling between Ca2+ release-activated Ca2+ channels, arachidonic acid release, and leukotriene C4 secretion.Activation of the store-operated calcium current ICRAC can be dissociated from regulated exocytosis in rat basophilic leukaemia (RBL-1) cells.Store depletion-activated CaT1 currents in rat basophilic leukemia mast cells are inhibited by 2-aminoethoxydiphenyl borate. Evidence for a regulatory component that controls activation of both CaT1 and CRAC (Ca(2+) release-activated Ca(2+) channel)Effects of inhibitors of the lipo-oxygenase family of enzymes on the store-operated calcium current I(CRAC) in rat basophilic leukaemia cells.Heterotrimeric G-proteins activate Cl- channels through stimulation of a cyclooxygenase-dependent pathway in a model liver cell line.Localized Ca2+ uncaging reveals polarized distribution of Ca2+-sensitive Ca2+ release sites: mechanism of unidirectional Ca2+ waves.STIM1 as a key regulator for Ca2+ homeostasis in skeletal-muscle development and function.NAADP mobilizes Ca2+ from a thapsigargin-sensitive store in the nuclear envelope by activating ryanodine receptors.Evidence that Ca2+-release-activated Ca2+ channels in rat hepatocytes are required for the maintenance of hormone-induced Ca2+ oscillations.Stabilization of cortical actin induces internalization of transient receptor potential 3 (Trp3)-associated caveolar Ca2+ signaling complex and loss of Ca2+ influx without disruption of Trp3-inositol trisphosphate receptor association.Dissociation of the store-operated calcium current I(CRAC) and the Mg-nucleotide-regulated metal ion current MagNuM.Inhibition of SERCA Ca2+ pumps by 2-aminoethoxydiphenyl borate (2-APB). 2-APB reduces both Ca2+ binding and phosphoryl transfer from ATP, by interfering with the pathway leading to the Ca2+-binding sites.
P2860
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P2860
An examination of the secretion-like coupling model for the activation of the Ca2+ release-activated Ca2+ current I(CRAC) in RBL-1 cells
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2001 nî lūn-bûn
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2001 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
2001年の論文
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2001年論文
@yue
2001年論文
@zh-hant
2001年論文
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2001年論文
@zh-mo
2001年論文
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2001年论文
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name
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@ast
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@en
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@nl
type
label
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@ast
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@en
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@nl
prefLabel
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@ast
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@en
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@nl
P2093
P2860
P1476
An examination of the secretio ...... current I(CRAC) in RBL-1 cells
@en
P2093
A B Parekh
D Bakowski
M D Glitsch
P2860
P356
10.1111/J.1469-7793.2001.0055G.X
P407
P577
2001-04-01T00:00:00Z