Ca(2+) signalling and PKCalpha activate increased endothelial permeability by disassembly of VE-cadherin junctions
about
A presenilin-1/gamma-secretase cleavage releases the E-cadherin intracellular domain and regulates disassembly of adherens junctions.Protease-activated receptor-1 activation of endothelial cells induces protein kinase Calpha-dependent phosphorylation of syntaxin 4 and Munc18c: role in signaling p-selectin expressionCaveolin-1 scaffold domain interacts with TRPC1 and IP3R3 to regulate Ca2+ store release-induced Ca2+ entry in endothelial cellsTezosentan-induced attenuation of lung injury in endotoxemic sheep is associated with reduced activation of protein kinase C.Activation of protein kinase C and disruption of endothelial monolayer integrity by sodium arsenite--Potential mechanism in the development of atherosclerosisReactive oxygen species in inflammation and tissue injuryOxidized phospholipids reduce vascular leak and inflammation in rat model of acute lung injuryBiomimetic carriers mimicking leukocyte plasma membrane to increase tumor vasculature permeability.Bone marrow-derived progenitor cells prevent thrombin-induced increase in lung vascular permeability.Ca2+ influx via TRPC channels induces NF-kappaB-dependent A20 expression to prevent thrombin-induced apoptosis in endothelial cells.The NO cascade, eNOS location, and microvascular permeability.Role of protein kinase Czeta in thrombin-induced RhoA activation and inter-endothelial gap formation of human dermal microvessel endothelial cell monolayersPDGF receptor activation induces p120-catenin phosphorylation at serine 879 via a PKCalpha-dependent pathway.Role of CPI-17 in the regulation of endothelial cytoskeleton.Regulation of endothelial permeability by Src kinase signaling: vascular leakage versus transcellular transport of drugs and macromolecules.Pyk2 phosphorylation of VE-PTP downstream of STIM1-induced Ca2+ entry regulates disassembly of adherens junctions.Sirolimus-FKBP12.6 impairs endothelial barrier function through protein kinase C-α activation and disruption of the p120-vascular endothelial cadherin interaction.Is thrombin a key player in the 'coagulation-atherogenesis' maze?Protein kinase C modulates pulmonary endothelial permeability: a paradigm for acute lung injury.Actinomyosin contraction, phosphorylation of VE-cadherin, and actin remodeling enable melanoma-induced endothelial cell-cell junction disassembly.Store-operated Ca2+ entry plays a role in HMGB1-induced vascular endothelial cell hyperpermeability.p120-catenin expressed in alveolar type II cells is essential for the regulation of lung innate immune response.Agonist of growth hormone-releasing hormone reduces pneumolysin-induced pulmonary permeability edema.Bixa orellana leaf extract suppresses histamine-induced endothelial hyperpermeability via the PLC-NO-cGMP signaling cascade.Intracellular Ascorbate Prevents Endothelial Barrier Permeabilization by Thrombin.Protein kinase C-α and arginase I mediate pneumolysin-induced pulmonary endothelial hyperpermeability.PKCα activation of p120-catenin serine 879 phospho-switch disassembles VE-cadherin junctions and disrupts vascular integrity.Mutant B-Raf(V600E) Promotes Melanoma Paracellular Transmigration by Inducing Thrombin-mediated Endothelial Junction BreakdownArginase 1: an unexpected mediator of pulmonary capillary barrier dysfunction in models of acute lung injury.Phosphorylation of E-cadherin at threonine 790 by protein kinase Cδ reduces β-catenin binding and suppresses the function of E-cadherin.Molecular mechanisms of endothelial hyperpermeability: implications in inflammation.The VE-cadherin cytoplasmic domain undergoes proteolytic processing during endocytosis.Protein kinase Calpha: disease regulator and therapeutic target.Interplay between FAK, PKCδ, and p190RhoGAP in the regulation of endothelial barrier function.Barrier enhancing signals in pulmonary edema.The effect of nanoparticle uptake on cellular behavior: disrupting or enabling functions?TRPV4: physiological role and therapeutic potential in respiratory diseases.Specific miRNA and its target in neutrophils after traumatic injury.The pathophysiology of pulmonary hypertension in left heart disease.Thrombin-unique coagulation system protein with multifaceted impacts on cancer and metastasis.
P2860
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P2860
Ca(2+) signalling and PKCalpha activate increased endothelial permeability by disassembly of VE-cadherin junctions
description
2001 nî lūn-bûn
@nan
2001 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի հունիսին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@ast
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@en
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@nl
type
label
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@ast
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@en
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@nl
prefLabel
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@ast
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@en
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@nl
P2093
P2860
P3181
P1476
Ca(2+) signalling and PKCalpha ...... embly of VE-cadherin junctions
@en
P2093
C Tiruppathi
R D Minshall
R Sandoval
P2860
P304
P3181
P356
10.1111/J.1469-7793.2001.0433A.X
P407
P577
2001-06-01T00:00:00Z