Protein kinase C-α and arginase I mediate pneumolysin-induced pulmonary endothelial hyperpermeability.
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Pneumococcal hydrogen peroxide-induced stress signaling regulates inflammatory genesThe leading role of microtubules in endothelial barrier dysfunction: disassembly of peripheral microtubules leaves behind the cytoskeletal reorganization.Protective effect of Growth Hormone-Releasing Hormone agonist in bacterial toxin-induced pulmonary barrier dysfunction.A novel tumor necrosis factor-mediated mechanism of direct epithelial sodium channel activationRegulation of NADPH oxidase 5 by protein kinase C isoforms.PKC-dependent phosphorylation of eNOS at T495 regulates eNOS coupling and endothelial barrier function in response to G+ -toxins.A computational study of the oligosaccharide binding sites in the lectin-like domain of Tumor Necrosis Factor and the TNF-derived TIP peptide.Statin-conferred enhanced cellular resistance against bacterial pore-forming toxins in airway epithelial cellsArginase 1: an unexpected mediator of pulmonary capillary barrier dysfunction in models of acute lung injury.Mini-review: novel therapeutic strategies to blunt actions of pneumolysin in the lungsl-Citrulline Protects from Kidney Damage in Type 1 Diabetic Mice.Dynamics of pulmonary endothelial barrier function in acute inflammation: mechanisms and therapeutic perspectives.Inhaled AP301 for treatment of pulmonary edema in mechanically ventilated patients with acute respiratory distress syndrome: a phase IIa randomized placebo-controlled trial.FMS-like tyrosine kinase 3 ligand treatment of mice aggravates acute lung injury in response to Streptococcus pneumoniae: role of pneumolysin.Bacteria and endothelial cells: a toxic relationship.Epithelial Sodium Channel-α Mediates the Protective Effect of the TNF-Derived TIP Peptide in Pneumolysin-Induced Endothelial Barrier Dysfunction.A FIM study to assess safety and exposure of inhaled single doses of AP301-A specific ENaC channel activator for the treatment of acute lung injury.Cytokine-Ion Channel Interactions in Pulmonary Inflammation.The Pulmonary Vascular Barrier: Insights into Structure, Function, and Regulatory Mechanisms.RhoA S-nitrosylation as a regulatory mechanism influencing endothelial barrier function in response to G+-bacterial toxins.Kidney-targeted inhibition of protein kinase C-α ameliorates nephrotoxic nephritis with restoration of mitochondrial dysfunction.Hsp70 Suppresses Mitochondrial Reactive Oxygen Species and Preserves Pulmonary Microvascular Barrier Integrity Following Exposure to Bacterial Toxins.
P2860
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P2860
Protein kinase C-α and arginase I mediate pneumolysin-induced pulmonary endothelial hyperpermeability.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh
2012年學術文章
@zh-hant
name
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@ast
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@en
type
label
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@ast
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@en
prefLabel
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@ast
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@en
P2093
P2860
P50
P356
P1476
Protein kinase C-α and arginas ...... endothelial hyperpermeability.
@en
P2093
Agnieszka Jezierska-Drutel
Alexander D Verin
Bernhard Fischer
Boris A Gorshkov
David J Fulton
Evgeny A Zemskov
Guang Yang
Hamid Hossain
Irina B Alieva
Jean-François Pittet
P2860
P304
P356
10.1165/RCMB.2011-0332OC
P577
2012-05-10T00:00:00Z