Arsenic exposure in utero exacerbates skin cancer response in adulthood with contemporaneous distortion of tumor stem cell dynamics - Wikidata - awgldk - TriplyDB

Arsenic exposure in utero exacerbates skin cancer response in adulthood with contemporaneous distortion of tumor stem cell dynamics

Arsenic exposure in utero exacerbates skin cancer response in adulthood with contemporaneous distortion of tumor stem cell dynamics

http://www.wikidata.org/entity/Q28387727

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Renal, hepatic, pulmonary and adrenal tumors induced by prenatal inorganic arsenic followed by dimethylarsinic acid in adulthood in CD1 mice Carcinogenic effects of "whole-life" exposure to inorganic arsenic in CD1 mice Arsenic Disruption of DNA Damage Responses-Potential Role in Carcinogenesis and Chemotherapy EMT and stem cell-like properties associated with HIF-2α are involved in arsenite-induced transformation of human bronchial epithelial cells Arsenic-induced sub-lethal stress reprograms human bronchial epithelial cells to CD61¯ cancer stem cells Cancer in experimental animals exposed to arsenic and arsenic compounds The impact of low-dose carcinogens and environmental disruptors on tissue invasion and metastasis In utero and early life arsenic exposure in relation to long-term health and disease Arsenic transformation predisposes human skin keratinocytes to UV-induced DNA damage yet enhances their survival apparently by diminishing oxidant response Arsenic exposure and toxicology: a historical perspective Overabundance of putative cancer stem cells in human skin keratinocyte cells malignantly transformed by arsenic Cancer Stem-Like Cells Accumulated in Nickel-Induced Malignant Transformation Methylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenic Arsenic Induces p62 Expression to Form a Positive Feedback Loop with Nrf2 in Human Epidermal Keratinocytes: Implications for Preventing Arsenic-Induced Skin Cancer Arsenite suppression of involucrin transcription through AP1 promoter sites in cultured human keratinocytes.Arsenic exposure transforms human epithelial stem/progenitor cells into a cancer stem-like phenotype.Effect of prenatal arsenic exposure on DNA methylation and leukocyte subpopulations in cord blood.Arsenic-specific stem cell selection during malignant transformation.MicroRNA-200b suppresses arsenic-transformed cell migration by targeting protein kinase Cα and Wnt5b-protein kinase Cα positive feedback loop and inhibiting Rac1 activation Early life inorganic lead exposure induces testicular teratoma and renal and urinary bladder preneoplasia in adult metallothionein-knockout mice but not in wild type mice.Evaluation of the carcinogenicity of inorganic arsenic.Arsenic, stem cells, and the developmental basis of adult cancer.Effect of Sodium Arsenite on Mouse Skin Carcinogenesis.Arsenic toxicology: translating between experimental models and human pathology.Induction of human squamous cell-type carcinomas by arsenic.Arsenic-transformed malignant prostate epithelia can convert noncontiguous normal stem cells into an oncogenic phenotype.Placental arsenic concentrations in relation to both maternal and infant biomarkers of exposure in a US cohort.Tumors and proliferative lesions in adult offspring after maternal exposure to methylarsonous acid during gestation in CD1 mice Mechanisms Underlying Latent Disease Risk Associated with Early-Life Arsenic Exposure: Current Research Trends and Scientific Gaps.The epigenome as a potential mediator of cancer and disease prevention in prenatal development.Geography of non-melanoma skin cancer and ecological associations with environmental risk factors in England.Recruitment of normal stem cells to an oncogenic phenotype by noncontiguous carcinogen-transformed epithelia depends on the transforming carcinogen.Aberrant cytokeratin expression during arsenic-induced acquired malignant phenotype in human HaCaT keratinocytes consistent with epidermal carcinogenesis.Chronic exposure of renal stem cells to inorganic arsenic induces a cancer phenotype.Arsenic-induced cancer cell phenotype in human breast epithelia is estrogen receptor-independent but involves aromatase activation.Aberrant microRNA expression likely controls RAS oncogene activation during malignant transformation of human prostate epithelial and stem cells by arsenic.Mitigation of arsenic-induced acquired cancer phenotype in prostate cancer stem cells by miR-143 restoration.Is Nickel Chloride really a Non-Genotoxic Carcinogen?Microenvironmental Scenario of the Bone Marrow of Inorganic Arsenic-Exposed Experimental Mice.Opposing actions of insulin and arsenite converge on PKCdelta to alter keratinocyte proliferative potential and differentiation

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Arsenic exposure in utero exacerbates skin cancer response in adulthood with contemporaneous distortion of tumor stem cell dynamics

http://www.wikidata.org/entity/Q28387727

description

2008 nî lūn-bûn

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2008 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

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2008年の論文

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2008年論文

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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0008-5472.CAN-08-2099

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Cancer Research

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Arsenic exposure in utero exac ...... on of tumor stem cell dynamics

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Bhalchandra A Diwan

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Carol S Trempus

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Dori R Germolec

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Erik J Tokar

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Jerrold M Ward

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Jie Liu

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Michael P Waalkes

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Raymond W Tennant

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Ronald E Cannon

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P2860

Arsenic can mediate skin neoplasia by chronic stimulation of keratinocyte-derived growth factors.

Arsenic enhancement of skin neoplasia by chronic stimulation of growth factors

Tissue repair and stem cell renewal in carcinogenesis

Association of arsenic-induced malignant transformation with DNA hypomethylation and aberrant gene expression

Increased mortality from lung cancer and bronchiectasis in young adults after exposure to arsenic in utero and in early childhood

Arsenite and insulin exhibit opposing effects on epidermal growth factor receptor and keratinocyte proliferative potential

Transplacental arsenic carcinogenesis in mice

The origins of the developmental origins theory

Critical windows of exposure for children's health: cancer in human epidemiological studies and neoplasms in experimental animal models

c-Myc activation in transgenic mouse epidermis results in mobilization of stem cells and differentiation of their progeny.

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8278-85

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10.1158/0008-5472.CAN-08-2099

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20

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18922899

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2652700

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