Arsenic-specific stem cell selection during malignant transformation. - Wikidata - awgldk - TriplyDB

Arsenic-specific stem cell selection during malignant transformation.

Arsenic-specific stem cell selection during malignant transformation.

http://www.wikidata.org/entity/Q33828693

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Carcinogenic effects of "whole-life" exposure to inorganic arsenic in CD1 mice Arsenic Disruption of DNA Damage Responses-Potential Role in Carcinogenesis and Chemotherapy EMT and stem cell-like properties associated with HIF-2α are involved in arsenite-induced transformation of human bronchial epithelial cells EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Arsenic-induced sub-lethal stress reprograms human bronchial epithelial cells to CD61¯ cancer stem cells Arsenic transformation predisposes human skin keratinocytes to UV-induced DNA damage yet enhances their survival apparently by diminishing oxidant response Chronic inorganic arsenic exposure in vitro induces a cancer cell phenotype in human peripheral lung epithelial cells Overabundance of putative cancer stem cells in human skin keratinocyte cells malignantly transformed by arsenic Methylarsonous acid causes oxidative DNA damage in cells independent of the ability to biomethylate inorganic arsenic Prenatal arsenic exposure alters gene expression in the adult liver to a proinflammatory state contributing to accelerated atherosclerosis Early life inorganic lead exposure induces testicular teratoma and renal and urinary bladder preneoplasia in adult metallothionein-knockout mice but not in wild type mice.Targeting miRNAs involved in cancer stem cell and EMT regulation: An emerging concept in overcoming drug resistance.Arsenic, stem cells, and the developmental basis of adult cancer.P38/NF-κB/snail pathway is involved in caffeic acid-induced inhibition of cancer stem cells-like properties and migratory capacity in malignant human keratinocyte.Arsenic immunotoxicity: a review.Multiple drug resistance due to resistance to stem cells and stem cell treatment progress in cancer (Review).Side populations from cervical-cancer-derived cell lines have stem-cell-like properties.Clonogenicity: holoclones and meroclones contain stem cells Enrichment of Human Stem-Like Prostate Cells with s-SHIP Promoter Activity Uncovers a Role in Stemness for the Long Noncoding RNA H19.Arsenic toxicology: translating between experimental models and human pathology.Global gene expression changes in human urothelial cells exposed to low-level monomethylarsonous acid.Differential DNA methylation profile of key genes in malignant prostate epithelial cells transformed by inorganic arsenic or cadmium Cross-regulations among NRFs and KEAP1 and effects of their silencing on arsenic-induced antioxidant response and cytotoxicity in human keratinocytes Arsenic-transformed malignant prostate epithelia can convert noncontiguous normal stem cells into an oncogenic phenotype.Regulatory role of KEAP1 and NRF2 in PPARγ expression and chemoresistance in human non-small-cell lung carcinoma cells.Chronic cadmium exposure in vitro causes acquisition of multiple tumor cell characteristics in human pancreatic epithelial cells.Mechanisms Underlying Latent Disease Risk Associated with Early-Life Arsenic Exposure: Current Research Trends and Scientific Gaps.Hedgehog pathway is involved in nitidine chloride induced inhibition of epithelial-mesenchymal transition and cancer stem cells-like properties in breast cancer cells Deconstructing the mechanisms and consequences of TGF-β-induced EMT during cancer progression.Recruitment of normal stem cells to an oncogenic phenotype by noncontiguous carcinogen-transformed epithelia depends on the transforming carcinogen.Inorganic Arsenic-Related Changes in the Stromal Tumor Microenvironment in a Prostate Cancer Cell-Conditioned Media Model.Chronic exposure of renal stem cells to inorganic arsenic induces a cancer phenotype.Aberrant microRNA expression likely controls RAS oncogene activation during malignant transformation of human prostate epithelial and stem cells by arsenic.Drug treatment of cancer cell lines: a way to select for cancer stem cells?MicroRNAs and drug resistance in prostate cancers.Mitigation of arsenic-induced acquired cancer phenotype in prostate cancer stem cells by miR-143 restoration.Chronic oxidative stress leads to malignant transformation along with acquisition of stem cell characteristics, and epithelial to mesenchymal transition in human renal epithelial cells.Oxidative DNA damage after acute exposure to arsenite and monomethylarsonous acid in biomethylation-deficient human cells.EMT and CSC-like properties mediated by the IKKβ/IκBα/RelA signal pathway via the transcriptional regulator, Snail, are involved in the arsenite-induced neoplastic transformation of human keratinocytes.A systematic approach to cancer: evolution beyond selection

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Arsenic-specific stem cell selection during malignant transformation.

http://www.wikidata.org/entity/Q33828693

description

2010 nî lūn-bûn

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2010 թուականի Մարտին հրատարակուած գիտական յօդուած

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2010 թվականի մարտին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Arsenic-specific stem cell selection during malignant transformation.

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Arsenic-specific stem cell selection during malignant transformation.

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type

label

Arsenic-specific stem cell selection during malignant transformation.

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Arsenic-specific stem cell selection during malignant transformation.

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prefLabel

Arsenic-specific stem cell selection during malignant transformation.

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Arsenic-specific stem cell selection during malignant transformation.

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Journal of the National Cancer Institute

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Arsenic-specific stem cell selection during malignant transformation.

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Erik J Tokar

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James M Phang

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Jie Liu

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Michael P Waalkes

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Mukta M Webber

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Wei Liu

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Wei Qu

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P2860

The Hallmarks of Cancer

Evidence for induction of oxidative stress caused by chronic exposure of Chinese residents to arsenic contained in drinking water

Clonogenic multiple myeloma progenitors, stem cell properties, and drug resistance

The role of PTEN/Akt/PI3K signaling in the maintenance and viability of prostate cancer stem-like cell populations

Multidrug resistance in cancer: role of ATP-dependent transporters

Regulation and function of proline oxidase under nutrient stress

Association of arsenic-induced malignant transformation with DNA hypomethylation and aberrant gene expression

Inorganic arsenic and human prostate cancer

Arsenic exposure in utero exacerbates skin cancer response in adulthood with contemporaneous distortion of tumor stem cell dynamics

Increased mortality from lung cancer and bronchiectasis in young adults after exposure to arsenic in utero and in early childhood

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20339138

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