Reactive oxygen species are not required for an arsenic trioxide-induced antioxidant response or apoptosis - Wikidata - awgldk - TriplyDB

Reactive oxygen species are not required for an arsenic trioxide-induced antioxidant response or apoptosis

Reactive oxygen species are not required for an arsenic trioxide-induced antioxidant response or apoptosis

http://www.wikidata.org/entity/Q28388476

about

TrxR1 as a potent regulator of the Nrf2-Keap1 response system Arsenic-mediated activation of the Nrf2-Keap1 antioxidant pathway Retinoic acid synergizes ATO-mediated cytotoxicity by precluding Nrf2 activity in AML cells Alterations in glutathione levels and apoptotic regulators are associated with acquisition of arsenic trioxide resistance in multiple myeloma Immunotoxicity and biodistribution analysis of arsenic trioxide in C57Bl/6 mice following a 2-week inhalation exposure Arsenic trioxide downregulates specificity protein (Sp) transcription factors and inhibits bladder cancer cell and tumor growth Chemistry of the cysteine sensors in Kelch-like ECH-associated protein 1 Autophagic degradation of the BCR-ABL oncoprotein and generation of antileukemic responses by arsenic trioxide Activation of the Nrf2 pathway by inorganic arsenic in human hepatocytes and the role of transcriptional repressor Bach1 Anticancer activity of metal complexes: involvement of redox processes.Synthetic Lethal Approaches Exploiting DNA Damage in Aggressive Myeloma.Arsenic trioxide induces unfolded protein response in vascular endothelial cells.Ethacrynic acid and a derivative enhance apoptosis in arsenic trioxide-treated myeloid leukemia and lymphoma cells: the role of glutathione S-transferase p1-1.Downregulation of Mcl-1 through GSK-3β activation contributes to arsenic trioxide-induced apoptosis in acute myeloid leukemia cells.Arsenic modulates heme oxygenase-1, interleukin-6, and vascular endothelial growth factor expression in endothelial cells: roles of ROS, NF-κB, and MAPK pathways.Selective apoptosis of multiple myeloma cells in primary samples induced by arsenic trioxide.In Vivo and In Vitro Arsenic Exposition Induces Oxidative Stress in Anterior Pituitary Gland.Realgar nanoparticles versus ATO arsenic compounds induce in vitro and in vivo activity against multiple myeloma.Arsenic trioxide-mediated suppression of miR-182-5p is associated with potent anti-oxidant effects through up-regulation of SESN2.

P2860

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P2860

Reactive oxygen species are not required for an arsenic trioxide-induced antioxidant response or apoptosis

http://www.wikidata.org/entity/Q28388476

description

2009 nî lūn-bûn

@nan

2009 թուականի Մայիսին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի մայիսին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

@zh-hk

2009年論文

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2009年論文

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2009年论文

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name

Reactive oxygen species are no ...... ioxidant response or apoptosis

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Reactive oxygen species are no ...... ioxidant response or apoptosis

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Reactive oxygen species are no ...... ioxidant response or apoptosis

@nl

type

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Reactive oxygen species are no ...... ioxidant response or apoptosis

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Reactive oxygen species are no ...... ioxidant response or apoptosis

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Reactive oxygen species are no ...... ioxidant response or apoptosis

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prefLabel

Reactive oxygen species are no ...... ioxidant response or apoptosis

@ast

Reactive oxygen species are no ...... ioxidant response or apoptosis

@en

Reactive oxygen species are no ...... ioxidant response or apoptosis

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P1433

Journal of Biological Chemistry

P1476

Reactive oxygen species are no ...... ioxidant response or apoptosis

@en

P2093

Alejo A Morales

http://www.w3.org/2001/XMLSchema#string

Delia Gutman

http://www.w3.org/2001/XMLSchema#string

Kelvin P Lee

http://www.w3.org/2001/XMLSchema#string

Lawrence H Boise

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Pedro J Cejas

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P2860

Induction of murine NAD(P)H:quinone oxidoreductase by 2,3,7,8-tetrachlorodibenzo-p-dioxin requires the CNC (cap n collar) basic leucine zipper transcription factor Nrf2 (nuclear factor erythroid 2-related factor 2)

Accelerated ovarian failure induced by 4-vinyl cyclohexene diepoxide in Nrf2 null mice

Multiorgan autoimmune inflammation, enhanced lymphoproliferation, and impaired homeostasis of reactive oxygen species in mice lacking the antioxidant-activated transcription factor Nrf2

Biliverdin reductase: a major physiologic cytoprotectant

Interactions of arsenic with human metallothionein-2

An Nrf2/small Maf heterodimer mediates the induction of phase II detoxifying enzyme genes through antioxidant response elements

Bilirubin is an antioxidant of possible physiological importance

Arsenic induces and enhances rat hepatic metallothionein production in vivo

Cell survival responses to environmental stresses via the Keap1-Nrf2-ARE pathway

Sensitivity to carcinogenesis is increased and chemoprotective efficacy of enzyme inducers is lost in nrf2 transcription factor-deficient mice

P304

12886-95

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scholarly article

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10.1074/JBC.M806546200

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19

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284

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2009-05-08T00:00:00Z

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19279006

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P932

2676019

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