Akt activation is responsible for enhanced migratory and invasive behavior of arsenic-transformed human bronchial epithelial cells
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The mechanistic basis of arsenicosis: pathogenesis of skin cancerThe microRNA-200 family: small molecules with novel roles in cancer development, progression and therapyJNK-dependent Stat3 phosphorylation contributes to Akt activation in response to arsenic exposureJNK and STAT3 signaling pathways converge on Akt-mediated phosphorylation of EZH2 in bronchial epithelial cells induced by arsenicChronic inorganic arsenic exposure in vitro induces a cancer cell phenotype in human peripheral lung epithelial cellsMolecular features in arsenic-induced lung tumorsPersistent Exposure to Porphyromonas gingivalis Promotes Proliferative and Invasion Capabilities, and Tumorigenic Properties of Human Immortalized Oral Epithelial Cells.Chronic arsenic exposure in nanomolar concentrations compromises wound response and intercellular signaling in airway epithelial cellsMicroRNA-200b suppresses arsenic-transformed cell migration by targeting protein kinase Cα and Wnt5b-protein kinase Cα positive feedback loop and inhibiting Rac1 activationAkt and phospholipase Cγ are involved in the regulation of growth and migration of MDA-MB-468 breast cancer and SW480 colon cancer cells when cultured with diabetogenic levels of glucose and insulin.MicroRNA-200b targets protein kinase Cα and suppresses triple-negative breast cancer metastasisFeedback regulations of miR-21 and MAPKs via Pdcd4 and Spry1 are involved in arsenite-induced cell malignant transformationP38/NF-κB/snail pathway is involved in caffeic acid-induced inhibition of cancer stem cells-like properties and migratory capacity in malignant human keratinocyte.Xiaoji Decoction inhibited cell proliferation and induced apoptosis through Akt signaling pathway in human lung cancer A549 cells.Epithelial to mesenchymal transition in arsenic-transformed cells promotes angiogenesis through activating β-catenin-vascular endothelial growth factor pathway.Regulation of the microRNA 200b (miRNA-200b) by transcriptional regulators PEA3 and ELK-1 protein affects expression of Pin1 protein to control anoikisFunctional role of inorganic trace elements in angiogenesis part III: (Ti, Li, Ce, As, Hg, Va, Nb and Pb).Carcinogenic metalloid arsenic induces expression of mdig oncogene through JNK and STAT3 activation.Acetylated Sp1 inhibits PTEN expression through binding to PTEN core promoter and recruitment of HDAC1 and promotes cancer cell migration and invasion.17β-Estradiol Inhibites Tumor Necrosis Factor-α Induced Apoptosis of Human Nucleus Pulposus Cells via the PI3K/Akt Pathway.The role of microRNAs in metal carcinogen-induced cell malignant transformation and tumorigenesis.TG-interacting factor mediates arsenic-induced malignant transformation of keratinocytes via c-Src/EGFR/AKT/FOXO3A and redox signalings.EMT and CSC-like properties mediated by the IKKβ/IκBα/RelA signal pathway via the transcriptional regulator, Snail, are involved in the arsenite-induced neoplastic transformation of human keratinocytes.MicroRNAs - Important Molecules in Lung Cancer Research.Alterations of miRNAs and Their Potential Roles in Arsenite-Induced Transformation of Human Bronchial Epithelial Cells.Gestational Exposure to Sidestream (Secondhand) Cigarette Smoke Promotes Transgenerational Epigenetic Transmission of Exacerbated Allergic Asthma and Bronchopulmonary Dysplasia.PI3K/Akt/mTOR Signaling Pathway and the Biphasic Effect of Arsenic in Carcinogenesis.
P2860
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P2860
Akt activation is responsible for enhanced migratory and invasive behavior of arsenic-transformed human bronchial epithelial cells
description
2012 nî lūn-bûn
@nan
2012 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2012 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
name
Akt activation is responsible ...... man bronchial epithelial cells
@ast
Akt activation is responsible ...... man bronchial epithelial cells
@en
Akt activation is responsible ...... man bronchial epithelial cells
@nl
type
label
Akt activation is responsible ...... man bronchial epithelial cells
@ast
Akt activation is responsible ...... man bronchial epithelial cells
@en
Akt activation is responsible ...... man bronchial epithelial cells
@nl
prefLabel
Akt activation is responsible ...... man bronchial epithelial cells
@ast
Akt activation is responsible ...... man bronchial epithelial cells
@en
Akt activation is responsible ...... man bronchial epithelial cells
@nl
P2093
P2860
P356
P1476
Akt activation is responsible ...... man bronchial epithelial cells
@en
P2093
Chengfeng Yang
Junling Yang
Theresa Fisher
Zhishan Wang
P2860
P356
10.1289/EHP.1104061
P407
P50
P577
2011-09-27T00:00:00Z