ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
about
Population genomics of parallel adaptation in threespine stickleback using sequenced RAD tagsEBV tegument protein BNRF1 disrupts DAXX-ATRX to activate viral early gene transcriptionDifferential expression of the miR-200 family microRNAs in epithelial and B cells and regulation of Epstein-Barr virus reactivation by the miR-200 family member miR-429Keeping it quiet: chromatin control of gammaherpesvirus latencyRegulation of the latent-lytic switch in Epstein-Barr virusInvolvement of Jun dimerization protein 2 (JDP2) in the maintenance of Epstein-Barr virus latencyThe B-cell specific transcription factor, Oct-2, promotes Epstein-Barr virus latency by inhibiting the viral immediate-early protein, BZLF1A role for the nucleosome assembly proteins TAF-Iβ and NAP1 in the activation of BZLF1 expression and Epstein-Barr virus reactivationAP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.Epigenetic regulation of EBV persistence and oncogenesis.Murine gammaherpesvirus 68 has evolved gamma interferon and stat1-repressible promoters for the lytic switch gene 50.Sumoylation of the Epstein-Barr virus BZLF1 protein inhibits its transcriptional activity and is regulated by the virus-encoded protein kinase.Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus's natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter.Either ZEB1 or ZEB2/SIP1 can play a central role in regulating the Epstein-Barr virus latent-lytic switch in a cell-type-specific manner.Activation and repression of Epstein-Barr Virus and Kaposi's sarcoma-associated herpesvirus lytic cycles by short- and medium-chain fatty acidsMicroRNA miR-BART20-5p stabilizes Epstein-Barr virus latency by directly targeting BZLF1 and BRLF1Cellular microRNAs 200b and 429 regulate the Epstein-Barr virus switch between latency and lytic replication.Histone deacetylases and the nuclear receptor corepressor regulate lytic-latent switch gene 50 in murine gammaherpesvirus 68-infected macrophagesTegument protein control of latent herpesvirus establishment and animation.The ZIIR element of the Epstein-Barr virus BZLF1 promoter plays a central role in establishment and maintenance of viral latencyTransforming growth factor beta-induced reactivation of Epstein-Barr virus involves multiple Smad-binding elements cooperatively activating expression of the latent-lytic switch BZLF1 gene.Cellular transcription factor Oct-1 interacts with the Epstein-Barr virus BRLF1 protein to promote disruption of viral latencyEBV and human microRNAs co-target oncogenic and apoptotic viral and human genes during latencyAn Epstein-Barr Virus (EBV) mutant with enhanced BZLF1 expression causes lymphomas with abortive lytic EBV infection in a humanized mouse model.Transcriptome changes induced by Epstein-Barr virus LMP1 and LMP2A in transgenic lymphocytes and lymphoma.Epstein-Barr virus-induced epigenetic alterations following transient infection.Secreted Oral Epithelial Cell Membrane Vesicles Induce Epstein-Barr Virus Reactivation in Latently Infected B Cells.Valpromide Inhibits Lytic Cycle Reactivation of Epstein-Barr Virus.The B-cell-specific transcription factor and master regulator Pax5 promotes Epstein-Barr virus latency by negatively regulating the viral immediate early protein BZLF1.The role of microRNAs in Epstein-Barr virus latency and lytic reactivation.Contribution of myocyte enhancer factor 2 family transcription factors to BZLF1 expression in Epstein-Barr virus reactivation from latency.Repression of ESR1 through actions of estrogen receptor alpha and Sin3A at the proximal promoter.Epstein-Barr virus utilizes Ikaros in regulating its latent-lytic switch in B cells.Epstein-Barr virus genetics: talking about the BAC generation.Epigenetic modification of the Epstein-Barr virus BZLF1 promoter regulates viral reactivation from latencySwitching of EBV cycles between latent and lytic states.Is murine gammaherpesvirus-68 (MHV-68) a suitable immunotoxicological model for examining immunomodulatory drug-associated viral recrudescence?Regulation of Epstein-Barr virus reactivation from latency.Epstein-Barr virus infection and persistence in nasopharyngeal epithelial cells.Viral Ubiquitin Ligase Stimulates Selective Host MicroRNA Expression by Targeting ZEB Transcriptional Repressors.
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P2860
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
description
2007 nî lūn-bûn
@nan
2007 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@ast
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@en
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@nl
type
label
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@ast
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@en
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@nl
prefLabel
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@ast
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@en
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@nl
P2093
P2860
P1433
P1476
ZEB1 regulates the latent-lytic switch in infection by Epstein-Barr virus
@en
P2093
Janet E Mertz
Xianming Yu
Zhenxun Wang
P2860
P356
10.1371/JOURNAL.PPAT.0030194
P407
P577
2007-12-01T00:00:00Z