Microarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversion - Wikidata - awgldk - TriplyDB

Microarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversion

Microarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversion

http://www.wikidata.org/entity/Q28473308

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MiR-155 induction by F. novicida but not the virulent F. tularensis results in SHIP down-regulation and enhanced pro-inflammatory cytokine response Interactions between Autophagy and Bacterial Toxins: Targets for Therapy?Differential effects of type I and II interferons on myeloid cells and resistance to intracellular bacterial infections Subversion of host recognition and defense systems by Francisella spp Comparative review of Francisella tularensis and Francisella novicida The involvement of IL-17A in the murine response to sub-lethal inhalational infection with Francisella tularensis Autophagy, Immunity, and Microbial Adaptations Brugia malayi gene expression in response to the targeting of the Wolbachia endosymbiont by tetracycline treatment Gender-associated genes in filarial nematodes are important for reproduction and potential intervention targets Fine tuning inflammation at the front door: macrophage complement receptor 3-mediates phagocytosis and immune suppression for Francisella tularensis Akt and SHIP modulate Francisella escape from the phagosome and induction of the Fas-mediated death pathway.Caspase-7 activation by the Nlrc4/Ipaf inflammasome restricts Legionella pneumophila infection.The presence of CD14 overcomes evasion of innate immune responses by virulent Francisella tularensis in human dendritic cells in vitro and pulmonary cells in vivo.Host factors required for modulation of phagosome biogenesis and proliferation of Francisella tularensis within the cytosol.Coxiella burnetii Nine Mile II proteins modulate gene expression of monocytic host cells during infection From mouth to macrophage: mechanisms of innate immune subversion by Mycobacterium avium subsp. paratuberculosis.Reciprocal regulation of activating and inhibitory Fc{gamma} receptors by TLR7/8 activation: implications for tumor immunotherapy.A Francisella novicida pdpA mutant exhibits limited intracellular replication and remains associated with the lysosomal marker LAMP-1 Directed screen of Francisella novicida virulence determinants using Drosophila melanogaster.Eradication of intracellular Francisella tularensis in THP-1 human macrophages with a novel autophagy inducing agent Francisella tularensis antioxidants harness reactive oxygen species to restrict macrophage signaling and cytokine production Generation of a convalescent model of virulent Francisella tularensis infection for assessment of host requirements for survival of tularemia The impact of "omic" and imaging technologies on assessing the host immune response to biodefence agents.Differential expression of microRNAs in Francisella tularensis-infected human macrophages: miR-155-dependent downregulation of MyD88 inhibits the inflammatory response.Mechanisms of microbial escape from phagocyte killing.Genetic identification of unique immunological responses in mice infected with virulent and attenuated Francisella tularensis Francisella tularensis subsp. tularensis induces a unique pulmonary inflammatory response: role of bacterial gene expression in temporal regulation of host defense responses Innate immune recognition of francisella tularensis: activation of type-I interferons and the inflammasome The francisella intracellular life cycle: toward molecular mechanisms of intracellular survival and proliferation.Secrets of a successful pathogen: legionella resistance to progression along the autophagic pathway.ModuleBlast: identifying activated sub-networks within and across species Akt-mediated proinflammatory response of mononuclear phagocytes infected with Burkholderia cenocepacia occurs by a novel GSK3β-dependent, IκB kinase-independent mechanism.Eating the strangers within: host control of intracellular bacteria via xenophagy.Francisella tularensis reveals a disparity between human and mouse NLRP3 inflammasome activation.Inflammasome priming is similar for francisella species that differentially induce inflammasome activation.Prior infection with Type A Francisella tularensis antagonizes the pulmonary transcriptional response to an aerosolized Toll-like receptor 4 agonist.Development of tolerogenic dendritic cells and regulatory T cells favors exponential bacterial growth and survival during early respiratory tularemia.Autophagy stimulation by rapamycin suppresses lung inflammation and infection by Burkholderia cenocepacia in a model of cystic fibrosis.Proteomic analysis of bronchoalveolar lavage fluid proteins from mice infected with Francisella tularensis ssp. novicida.Cytosolic clearance of replication-deficient mutants reveals Francisella tularensis interactions with the autophagic pathway

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Microarray analysis of human monocytes infected with Francisella tularensis identifies new targets of host response subversion

http://www.wikidata.org/entity/Q28473308

description

2008 nî lūn-bûn

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2008 թուականի Օգոստոսին հրատարակուած գիտական յօդուած

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2008 թվականի օգոստոսին հրատարակված գիտական հոդված

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2008年の論文

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2008年論文

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2008年论文

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Microarray analysis of human m ...... ts of host response subversion

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Clay B Marsh

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Corey D Clay

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Jonathan P Butchar

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Larry S Schlesinger

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Mark D Wewers

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Mikhail A Gavrilin

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Susheela Tridandapani

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Thomas J Cremer

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P2860

Bioconductor: open software development for computational biology and bioinformatics

The inflammasome: a molecular platform triggering activation of inflammatory caspases and processing of proIL-beta

Cluster analysis and display of genome-wide expression patterns

Gene Expression Omnibus: NCBI gene expression and hybridization array data repository

Linear models and empirical bayes methods for assessing differential expression in microarray experiments

The suppressor of cytokine signaling (SOCS) 1 and SOCS3 but not SOCS2 proteins inhibit interferon-mediated antiviral and antiproliferative activities

MyD88 is an adaptor protein in the hToll/IL-1 receptor family signaling pathways

Open source software for the analysis of microarray data.

Macrophage pro-inflammatory response to Francisella novicida infection is regulated by SHIP.

The role of the activated macrophage in clearing Listeria monocytogenes infection

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