A general model for binary cell fate decision gene circuits with degeneracy: indeterminacy and switch behavior in the absence of cooperativity
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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determinationImplications of the Hybrid Epithelial/Mesenchymal Phenotype in MetastasisDriving Cells to the Desired State in a Bimodal Distribution through Manipulation of Internal Noise with Biologically Practicable ApproachesClassification of transient behaviours in a time-dependent toggle switch modelThe three-way switch operation of Rac1/RhoA GTPase-based circuit controlling amoeboid-hybrid-mesenchymal transition.BTR: training asynchronous Boolean models using single-cell expression data.Reshaping the epigenetic landscape during early flower development: induction of attractor transitions by relative differences in gene decay rates.Mathematical modeling of GATA-switching for regulating the differentiation of hematopoietic stem cell.Inhibitors Alter the Stochasticity of Regulatory Proteins to Force Cells to Switch to the Other State in the Bistable System.A transcriptional dynamic network during Arabidopsis thaliana pollen development.Towards elucidating the connection between epithelial-mesenchymal transitions and stemnessFrom understanding the development landscape of the canonical fate-switch pair to constructing a dynamic landscape for two-step neural differentiation.Hybrid T-helper cells: stabilizing the moderate center in a polarized system.Bifurcation dynamics and determination of alternate cell fates in bipotent progenitor cellsIncreased robustness of early embryogenesis through collective decision-making by key transcription factorsPluripotency, Differentiation, and Reprogramming: A Gene Expression Dynamics Model with Epigenetic Feedback Regulation.Coupling the modules of EMT and stemness: A tunable 'stemness window' modelA bistable genetic switch based on designable DNA-binding domains.The AhR agonist VAF347 augments retinoic acid-induced differentiation in leukemia cells.Nanog Fluctuations in Embryonic Stem Cells Highlight the Problem of Measurement in Cell Biology.Boolean network model for GPR142 against Type 2 diabetes and relative dynamic change ratio analysis using systems and biological circuits approach.Single-cell transcriptional analysis to uncover regulatory circuits driving cell fate decisions in early mouse development.Transcriptional bursting diversifies the behaviour of a toggle switch: hybrid simulation of stochastic gene expression.CD4+ T Cell Fate Decisions Are Stochastic, Precede Cell Division, Depend on GITR Co-Stimulation, and Are Associated With Uropodium Development.
P2860
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P2860
A general model for binary cell fate decision gene circuits with degeneracy: indeterminacy and switch behavior in the absence of cooperativity
description
2011 nî lūn-bûn
@nan
2011 թուականին հրատարակուած գիտական յօդուած
@hyw
2011 թվականին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
A general model for binary cel ...... n the absence of cooperativity
@ast
A general model for binary cel ...... n the absence of cooperativity
@en
A general model for binary cel ...... n the absence of cooperativity
@nl
type
label
A general model for binary cel ...... n the absence of cooperativity
@ast
A general model for binary cel ...... n the absence of cooperativity
@en
A general model for binary cel ...... n the absence of cooperativity
@nl
prefLabel
A general model for binary cel ...... n the absence of cooperativity
@ast
A general model for binary cel ...... n the absence of cooperativity
@en
A general model for binary cel ...... n the absence of cooperativity
@nl
P2093
P2860
P3181
P1433
P1476
A general model for binary cel ...... n the absence of cooperativity
@en
P2093
Julianne D Halley
Mircea Andrecut
P2860
P304
P3181
P356
10.1371/JOURNAL.PONE.0019358
P407
P577
2011-01-01T00:00:00Z