MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination - Wikidata - awgldk - TriplyDB

MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

http://www.wikidata.org/entity/Q24598602

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Phenotypic plasticity in prostate cancer: role of intrinsically disordered proteins Tumor Budding: The Name is EMT. Partial EMT Emerging Transcriptional Mechanisms in the Regulation of Epithelial to Mesenchymal Transition and Cellular Plasticity in the Kidney Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis Glioblastoma Circulating Cells: Reality, Trap or Illusion?Tumor invasion optimization by mesenchymal-amoeboid heterogeneity Key nodes of a microRNA network associated with the integrated mesenchymal subtype of high-grade serous ovarian cancer TGF-β-induced epithelial-to-mesenchymal transition proceeds through stepwise activation of multiple feedback loops Advanced fault diagnosis methods in molecular networks Network modeling of TGFβ signaling in hepatocellular carcinoma epithelial-to-mesenchymal transition reveals joint sonic hedgehog and Wnt pathway activation The three-way switch operation of Rac1/RhoA GTPase-based circuit controlling amoeboid-hybrid-mesenchymal transition.Transforming growth factor-β1 regulates epithelial-mesenchymal transition in association with cancer stem-like cells in a breast cancer cell line Inflammatory breast cancer: a model for investigating cluster-based dissemination.Epithelial/mesenchymal plasticity: how have quantitative mathematical models helped improve our understanding?EMT and MET: necessary or permissive for metastasis?Combinatorial interventions inhibit TGFβ-driven epithelial-to-mesenchymal transition and support hybrid cellular phenotypes.Move your microvilli Modeling putative therapeutic implications of exosome exchange between tumor and immune cells.Towards elucidating the connection between epithelial-mesenchymal transitions and stemness Stability of the hybrid epithelial/mesenchymal phenotype.Jagged-Delta asymmetry in Notch signaling can give rise to a Sender/Receiver hybrid phenotype.Mathematical modeling of combinatorial regulation suggests that apparent positive regulation of targets by miRNA could be an artifact resulting from competition for mRNA Stemness of the hybrid Epithelial/Mesenchymal State in Breast Cancer and Its Association with Poor Survival.Preclinical safety and efficacy studies with an affinity-enhanced epithelial junction opener and PEGylated liposomal doxorubicin.Modeling of Cancer Stem Cell State Transitions Predicts Therapeutic Response.The roles of microRNAs in the regulation of tumor metastasis.Mathematical Modelling of Molecular Pathways Enabling Tumour Cell Invasion and Migration.An Ovol2-Zeb1 Mutual Inhibitory Circuit Governs Bidirectional and Multi-step Transition between Epithelial and Mesenchymal States.The role of neural connexins in HeLa cell mobility and intercellular communication through tunneling tubes.Jagged mediates differences in normal and tumor angiogenesis by affecting tip-stalk fate decision.Mutation-induced protein interaction kinetics changes affect apoptotic network dynamic properties and facilitate oncogenesis.Prolonged pemetrexed pretreatment augments persistence of cisplatin-induced DNA damage and eliminates resistant lung cancer stem-like cells associated with EMT Feedbacks, Bifurcations, and Cell Fate Decision-Making in the p53 System OVOL guides the epithelial-hybrid-mesenchymal transition.ΔNp63α Promotes Breast Cancer Cell Motility through the Selective Activation of Components of the Epithelial-to-Mesenchymal Transition Program Population Heterogeneity in the Epithelial to Mesenchymal Transition Is Controlled by NFAT and Phosphorylated Sp1.Interrogating the topological robustness of gene regulatory circuits by randomization.Modeling the Transitions between Collective and Solitary Migration Phenotypes in Cancer Metastasis.Coupling the modules of EMT and stemness: A tunable 'stemness window' model Cell Fate Specification Based on Tristability in the Inner Cell Mass of Mouse Blastocysts.

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

http://www.wikidata.org/entity/Q24598602

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2013 nî lūn-bûn

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2013 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2013年の論文

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name

MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

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Proceedings of the National Academy of Sciences of the United States of America

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MicroRNA-based regulation of epithelial-hybrid-mesenchymal fate determination

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Eshel Ben-Jacob

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Herbert Levine

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Mingyang Lu

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Mohit Kumar Jolly

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P2860

The epithelial-mesenchymal transition (EMT) phenomenon

The ZEB/miR-200 feedback loop--a motor of cellular plasticity in development and cancer?

Epithelial-mesenchymal transition in cancer: parallels between normal development and tumor progression

MicroRNA regulation of a cancer network: consequences of the feedback loops involving miR-17-92, E2F, and Myc

A reciprocal repression between ZEB1 and members of the miR-200 family promotes EMT and invasion in cancer cells

Epithelial-mesenchymal transitions in development and disease

Construction of a genetic toggle switch in Escherichia coli

EMT in developmental morphogenesis

Coupled reversible and irreversible bistable switches underlying TGFβ-induced epithelial to mesenchymal transition

A general model for binary cell fate decision gene circuits with degeneracy: indeterminacy and switch behavior in the absence of cooperativity

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