Murine Jagged1/Notch signaling in the second heart field orchestrates Fgf8 expression and tissue-tissue interactions during outflow tract development
about
Coordinating tissue interactions: Notch signaling in cardiac development and diseaseThe neural crest in cardiac congenital anomaliesCranial neural crest ablation of Jagged1 recapitulates the craniofacial phenotype of Alagille syndrome patientsIslet1 derivatives in the heart are of both neural crest and second heart field originEndothelial deletion of murine Jag1 leads to valve calcification and congenital heart defects associated with Alagille syndromeWnt/β-catenin and Bmp signals control distinct sets of transcription factors in cardiac progenitor cellsHes1 expression is reduced in Tbx1 null cells and is required for the development of structures affected in 22q11 deletion syndromeA Tbx1-Six1/Eya1-Fgf8 genetic pathway controls mammalian cardiovascular and craniofacial morphogenesisThe neural crest-enriched microRNA miR-452 regulates epithelial-mesenchymal signaling in the first pharyngeal archEvolutionary and developmental origins of the cardiac neural crest: building a divided outflow tractCardiac neural crest orchestrates remodeling and functional maturation of mouse semilunar valves.Outflow tract septation and the aortic arch system in reptiles: lessons for understanding the mammalian heart.The molecular genetics of congenital heart disease: a review of recent developments.Overexpression of angiopoietin-1 increases CD133+/c-kit+ cells and reduces myocardial apoptosis in db/db mouse infarcted hearts.Heart fields and cardiac morphogenesisCommon variations in BMP4 confer genetic susceptibility to sporadic congenital heart disease in a Han Chinese population.Notch signaling in descending thoracic aortic aneurysm and dissectionJun is required in Isl1-expressing progenitor cells for cardiovascular developmentJagged1 is essential for osteoblast development during maxillary ossification.Nfatc1 coordinates valve endocardial cell lineage development required for heart valve formation.Loss of Wnt5a disrupts second heart field cell deployment and may contribute to OFT malformations in DiGeorge syndromeHistone deacetylase 3 regulates smooth muscle differentiation in neural crest cells and development of the cardiac outflow tractThe secondary heart field is a new site of calcineurin/Nfatc1 signaling for semilunar valve development.Persistent expression of activated notch in the developing hypothalamus affects survival of pituitary progenitors and alters pituitary structure.Notch signaling and cardiac repair.Signaling in cell differentiation and morphogenesisNeural crest-derived SEMA3C activates endothelial NRP1 for cardiac outflow tract septation.Numb family proteins: novel players in cardiac morphogenesis and cardiac progenitor cell differentiation.Partitioning the heart: mechanisms of cardiac septation and valve developmentHistone Deacetylase 3 Coordinates Deacetylase-independent Epigenetic Silencing of Transforming Growth Factor-β1 (TGF-β1) to Orchestrate Second Heart Field DevelopmentApelin-13 increases myocardial progenitor cells and improves repair postmyocardial infarction.Endothelial Notch1 Is Required for Proper Development of the Semilunar Valves and Cardiac Outflow Tract.Rac1 Signaling Is Required for Anterior Second Heart Field Cellular Organization and Cardiac Outflow Tract Development.FRS2α-mediated FGF signals suppress premature differentiation of cardiac stem cells through regulating autophagy activity.The pathology and pathobiology of bicuspid aortic valve: State of the art and novel research perspectives.The role of secondary heart field in cardiac development.Fibronectin signals through integrin α5β1 to regulate cardiovascular development in a cell type-specific manner.Tissue-tissue interactions during morphogenesis of the outflow tract.The Notch1 transcriptional activation domain is required for development and reveals a novel role for Notch1 signaling in fetal hematopoietic stem cells.Notch and cardiac outflow tract development.
P2860
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P2860
Murine Jagged1/Notch signaling in the second heart field orchestrates Fgf8 expression and tissue-tissue interactions during outflow tract development
description
2009 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Juli 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/07/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/07/01)
@nl
наукова стаття, опублікована в липні 2009
@uk
مقالة علمية (نشرت في يوليو 2009)
@ar
name
Murine Jagged1/Notch signaling ...... ring outflow tract development
@ast
Murine Jagged1/Notch signaling ...... ring outflow tract development
@en
Murine Jagged1/Notch signaling ...... ring outflow tract development
@nl
type
label
Murine Jagged1/Notch signaling ...... ring outflow tract development
@ast
Murine Jagged1/Notch signaling ...... ring outflow tract development
@en
Murine Jagged1/Notch signaling ...... ring outflow tract development
@nl
prefLabel
Murine Jagged1/Notch signaling ...... ring outflow tract development
@ast
Murine Jagged1/Notch signaling ...... ring outflow tract development
@en
Murine Jagged1/Notch signaling ...... ring outflow tract development
@nl
P2093
P2860
P3181
P356
P1476
Murine Jagged1/Notch signaling ...... ring outflow tract development
@en
P2093
Frances A. High
Jason Z. Stoller
Jonathan A. Epstein
Kathleen M. Loomes
Klaus H. Kaestner
Min Min Lu
Nicole B. Antonucci
Rajan Jain
Warren S. Pear
P2860
P304
P3181
P356
10.1172/JCI38922
P407
P577
2009-07-01T00:00:00Z