Coordinated transport of phosphorylated amyloid-beta precursor protein and c-Jun NH2-terminal kinase-interacting protein-1
about
The amyloid precursor protein: beyond amyloidAmyloid-Beta Induced Changes in Vesicular Transport of BDNF in Hippocampal NeuronsNebula/DSCR1 upregulation delays neurodegeneration and protects against APP-induced axonal transport defects by restoring calcineurin and GSK-3β signalingSpatial and temporal characteristics of normal and perturbed vesicle transportCo-operative versus independent transport of different cargoes by Kinesin-1Axonal transport and neurodegenerative disease: can we see the elephant?A peptide zipcode sufficient for anterograde transport within amyloid precursor protein.The amyloid-beta precursor protein is phosphorylated via distinct pathways during differentiation, mitosis, stress, and degeneration.The cleavage products of amyloid-beta precursor protein are sorted to distinct carrier vesicles that are independently transported within neurites.Loss of c-Jun N-terminal kinase-interacting protein-1 does not affect axonal transport of the amyloid precursor protein or Aβ production.Dual-tagged amyloid-β precursor protein reveals distinct transport pathways of its N- and C-terminal fragmentsGRIP1 interlinks N-cadherin and AMPA receptors at vesicles to promote combined cargo transport into dendrites.LC3 binding to the scaffolding protein JIP1 regulates processive dynein-driven transport of autophagosomes.Clues to neuro-degeneration in Niemann-Pick type C disease from global gene expression profilingPhosphorylation of amyloid precursor protein at threonine 668 is essential for its copper-responsive trafficking in SH-SY5Y neuroblastoma cells.Tyr(682) in the intracellular domain of APP regulates amyloidogenic APP processing in vivo.Calsyntenin-1 mediates axonal transport of the amyloid precursor protein and regulates Aβ productionThe intracellular threonine of amyloid precursor protein that is essential for docking of Pin1 is dispensable for developmental function.Neuritic deposits of amyloid-beta peptide in a subpopulation of central nervous system-derived neuronal cells.Functional interaction between amyloid-β precursor protein and peripherin neurofilaments: a shared pathway leading to Alzheimer's disease and amyotrophic lateral sclerosis?Amyloid-β precursor protein: Multiple fragments, numerous transport routes and mechanismsUnconventional functions of microtubule motors.A persistent stress response to impeded axonal transport leads to accumulation of amyloid-β in the endoplasmic reticulum, and is a probable cause of sporadic Alzheimer's disease.Interaction of ASK1 and the beta-amyloid precursor protein in a stress-signaling complex.Shared Molecular Mechanisms in Alzheimer's Disease and Amyotrophic Lateral Sclerosis: Neurofilament-Dependent Transport of sAPP, FUS, TDP-43 and SOD1, with Endoplasmic Reticulum-Like Tubules.Do axonal defects in tau and amyloid precursor protein transgenic animals model axonopathy in Alzheimer's disease?APP Receptor? To Be or Not To Be.Seeding neuritic plaques from the distance: a possible role for brainstem neurons in the development of Alzheimer's disease pathology.Potential role of PCTAIRE-2, PCTAIRE-3 and P-Histone H4 in amyloid precursor protein-dependent Alzheimer pathology.Independent pathways downstream of the Wnd/DLK MAPKKK regulate synaptic structure, axonal transport, and injury signalingJIP1 regulates the directionality of APP axonal transport by coordinating kinesin and dynein motors.The interactome of the amyloid beta precursor protein family members is shaped by phosphorylation of their intracellular domains.Biology and pathophysiology of the amyloid precursor protein.Integrated regulation of motor-driven organelle transport by scaffolding proteins.Axonal transport defects in Alzheimer's disease.FE65 proteins regulate NMDA receptor activation-induced amyloid precursor protein processing.Activation of Ras-ERK Signaling and GSK-3 by Amyloid Precursor Protein and Amyloid Beta Facilitates Neurodegeneration in Alzheimer's Disease.Serum-induced neurite retraction in CAD cells--involvement of an ATP-actin retractile system and the lack of microtubule-associated proteins.Regulation of dynein-dynactin-driven vesicular transport.Is abnormal axonal transport a cause, a contributing factor or a consequence of the neuronal pathology in Alzheimer's disease?
P2860
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P248
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P2860
Coordinated transport of phosphorylated amyloid-beta precursor protein and c-Jun NH2-terminal kinase-interacting protein-1
description
2005 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
@ast
im November 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/11/21)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/11/21)
@nl
наукова стаття, опублікована в листопаді 2005
@uk
مقالة علمية (نشرت في 21-11-2005)
@ar
name
Coordinated transport of phosp ...... l kinase-interacting protein-1
@ast
Coordinated transport of phosp ...... l kinase-interacting protein-1
@en
Coordinated transport of phosp ...... l kinase-interacting protein-1
@nl
type
label
Coordinated transport of phosp ...... l kinase-interacting protein-1
@ast
Coordinated transport of phosp ...... l kinase-interacting protein-1
@en
Coordinated transport of phosp ...... l kinase-interacting protein-1
@nl
prefLabel
Coordinated transport of phosp ...... l kinase-interacting protein-1
@ast
Coordinated transport of phosp ...... l kinase-interacting protein-1
@en
Coordinated transport of phosp ...... l kinase-interacting protein-1
@nl
P2860
P921
P356
P1476
Coordinated transport of phosp ...... l kinase-interacting protein-1
@en
P2093
Virgil Muresan
Zoia Muresan
P2860
P304
P356
10.1083/JCB.200502043
P407
P577
2005-11-21T00:00:00Z