Mice lacking the ski proto-oncogene have defects in neurulation, craniofacial, patterning, and skeletal muscle development
about
Ski is a component of the histone deacetylase complex required for transcriptional repression by Mad and thyroid hormone receptorSki-interacting protein interacts with Smad proteins to augment transforming growth factor-beta-dependent transcriptionThe Ski protein family is required for MeCP2-mediated transcriptional repressionSki regulates muscle terminal differentiation by transcriptional activation of Myog in a complex with Six1 and Eya3Ski is involved in transcriptional regulation by the repressor and full-length forms of Gli3Transforming growth factor-beta-independent regulation of myogenesis by SnoN sumoylationSki interacts with the evolutionarily conserved SNW domain of SkipRegulation of myostatin activity and muscle growthThe Ski oncoprotein interacts with the Smad proteins to repress TGFbeta signalingPalatogenesis and cutaneous repair: A two-headed coinModeling anterior development in mice: diet as modulator of risk for neural tube defectsRepression of TGF-beta signaling by the oncogenic protein SKI in human melanomas: consequences for proliferation, survival, and metastasisA direct intersection between p53 and transforming growth factor beta pathways targets chromatin modification and transcription repression of the alpha-fetoprotein geneThe anaphase-promoting complex coordinates initiation of lens differentiationThe sno gene, which encodes a component of the histone deacetylase complex, acts as a tumor suppressor in miceMisexpression of the eyes absent family triggers the apoptotic programIncreased susceptibility to tumorigenesis of ski-deficient heterozygous miceDefective T-cell activation is associated with augmented transforming growth factor Beta sensitivity in mice with mutations in the Sno gene.Strain-specific modifier genes governing craniofacial phenotypes.An allelic series of mice reveals a role for RERE in the development of multiple organs affected in chromosome 1p36 deletions.Synergistic regulation of vertebrate muscle development by Dach2, Eya2, and Six1, homologs of genes required for Drosophila eye formation.Modular regulation of the MLC1F/3F gene and striated muscle diversity.The Max network gone mad.Regulation of mandibular growth and morphogenesis.The SMAD-binding domain of SKI: a hotspot for de novo mutations causing Shprintzen-Goldberg syndrome.Regulation of myogenic differentiation in the developing limb bud.SnoN in mammalian development, function and diseases.Novel regulation of Ski protein stability and endosomal sorting by actin cytoskeleton dynamics in hepatocytesPeroxisome proliferator-activated receptor γ (PPARγ) mediates a Ski oncogene-induced shift from glycolysis to oxidative energy metabolism.Ski acts as a co-repressor with Smad2 and Smad3 to regulate the response to type beta transforming growth factorProtooncogene Ski cooperates with the chromatin-remodeling factor Satb2 in specifying callosal neurons.Ski represses bone morphogenic protein signaling in Xenopus and mammalian cellsOverexpression of SKI oncoprotein leads to p53 degradation through regulation of MDM2 protein sumoylation.Generation of Ski-knockdown mice by expressing a long double-strand RNA from an RNA polymerase II promoter.Kinesin-2 controls development and patterning of the vertebrate skeleton by Hedgehog- and Gli3-dependent mechanisms.Transformation of hematopoietic cells by the Ski oncoprotein involves repression of retinoic acid receptor signalingDoes the cranial mesenchyme contribute to neural fold elevation during neurulation?Differential role of Sloan-Kettering Institute (Ski) protein in Nodal and transforming growth factor-beta (TGF-β)-induced Smad signaling in prostate cancer cells.Tyrosine phosphatase MEG2 modulates murine development and platelet and lymphocyte activation through secretory vesicle functionMutations in the TGF-β repressor SKI cause Shprintzen-Goldberg syndrome with aortic aneurysm
P2860
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P2860
Mice lacking the ski proto-oncogene have defects in neurulation, craniofacial, patterning, and skeletal muscle development
description
1997 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1997
@ast
im August 1997 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1997/08/15)
@sk
vědecký článek publikovaný v roce 1997
@cs
wetenschappelijk artikel (gepubliceerd op 1997/08/15)
@nl
наукова стаття, опублікована в серпні 1997
@uk
научни чланак (објављен 1997/08/15)
@sr
name
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@ast
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@en
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@nl
type
label
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@ast
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@en
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@nl
prefLabel
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@ast
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@en
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@nl
P2093
P2860
P356
P1433
P1476
Mice lacking the ski proto-onc ...... nd skeletal muscle development
@en
P2093
C. Colmenares
H. C. Heyman
S. Y. Desai
P2860
P304
P356
10.1101/GAD.11.16.2029
P577
1997-08-15T00:00:00Z