Electrogram prolongation and nifedipine-suppressible ventricular arrhythmias in mice following targeted disruption of KCNE1
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Scn3b knockout mice exhibit abnormal ventricular electrophysiological propertiesVariable Na(v)1.5 protein expression from the wild-type allele correlates with the penetrance of cardiac conduction disease in the Scn5a(+/-) mouse modelArrhythmogenic mechanisms in the isolated perfused hypokalaemic murine heart.Atrial Anti-Arrhythmic Effects of Heptanol in Langendorff-Perfused Mouse HeartsNonlinearity between action potential alternans and restitution, which both predict ventricular arrhythmic properties in Scn5a+/- and wild-type murine heartsEffects of potassium channel openers in the isolated perfused hypokalaemic murine heart.BACE1 and presenilin/γ-secretase regulate proteolytic processing of KCNE1 and 2, auxiliary subunits of voltage-gated potassium channels.Physiological consequences of the P2328S mutation in the ryanodine receptor (RyR2) gene in genetically modified murine hearts.Action potential wavelength restitution predicts alternans and arrhythmia in murine Scn5a(+/-) hearts.Atrial arrhythmogenesis in wild-type and Scn5a+/delta murine hearts modelling LQT3 syndrome.Empirical correlation of triggered activity and spatial and temporal re-entrant substrates with arrhythmogenicity in a murine model for Jervell and Lange-Nielsen syndrome.Gap junction inhibition by heptanol increases ventricular arrhythmogenicity by reducing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts.Cardiac electrophysiology in mice: a matter of size.Refractory dispersion promotes conduction disturbance and arrhythmias in a Scn5a (+/-) mouse modelModulation of the QT interval duration in hypertension with antihypertensive treatment.Ventricular anti-arrhythmic effects of heptanol in hypokalaemic, Langendorff-perfused mouse heartsMurine Electrophysiological Models of Cardiac Arrhythmogenesis.Spatial and temporal heterogeneities are localized to the right ventricular outflow tract in a heterozygotic Scn5a mouse model.Criteria for arrhythmogenicity in genetically-modified Langendorff-perfused murine hearts modelling the congenital long QT syndrome type 3 and the Brugada syndrome.Restitution analysis of alternans and its relationship to arrhythmogenicity in hypokalaemic Langendorff-perfused murine hearts.Anti-arrhythmic effects of hypercalcemia in hyperkalemic, Langendorff-perfused mouse heartsArrhythmogenic actions of the Ca2+ channel agonist FPL-64716 in Langendorff-perfused murine hearts.The contribution of refractoriness to arrhythmic substrate in hypokalemic Langendorff-perfused murine hearts.Increased right ventricular repolarization gradients promote arrhythmogenesis in a murine model of Brugada syndrome.The heart requires glycerol as an energy substrate through aquaporin 7, a glycerol facilitator.Alternans in genetically modified langendorff-perfused murine hearts modeling catecholaminergic polymorphic ventricular tachycardia.Pharmacological separation of early afterdepolarizations from arrhythmogenic substrate in DeltaKPQ Scn5a murine hearts modelling human long QT 3 syndrome.Regional variations in action potential alternans in isolated murine Scn5a (+/-) hearts during dynamic pacing.A quantitative analysis of the effect of cycle length on arrhythmogenicity in hypokalaemic Langendorff-perfused murine hearts.Effects of anesthetics on systemic hemodynamics in mice.Caffeine-induced arrhythmias in murine hearts parallel changes in cellular Ca(2+) homeostasis.Computer model of action potential of mouse ventricular myocytes.Effects of L-type Ca2+ channel antagonism on ventricular arrhythmogenesis in murine hearts containing a modification in the Scn5a gene modelling human long QT syndrome 3.Mechanisms of ventricular arrhythmogenesis in mice following targeted disruption of KCNE1 modelling long QT syndrome 5.Transient alterations in transmural repolarization gradients and arrhythmogenicity in hypokalaemic Langendorff-perfused murine hearts.
P2860
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P2860
Electrogram prolongation and nifedipine-suppressible ventricular arrhythmias in mice following targeted disruption of KCNE1
description
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
im Oktober 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/10/15)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/10/15)
@nl
наукова стаття, опублікована в жовтні 2003
@uk
مقالة علمية (نشرت في 15-10-2003)
@ar
name
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@ast
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@en
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@nl
type
label
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@ast
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@en
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@nl
prefLabel
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@ast
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@en
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@nl
P2093
P2860
P3181
P1476
Electrogram prolongation and n ...... g targeted disruption of KCNE1
@en
P2093
Andrew A Grace
Christopher L-H Huang
Richard Balasubramaniam
Richard C Saumarez
P2860
P304
P3181
P356
10.1113/JPHYSIOL.2003.048249
P407
P577
2003-10-01T00:00:00Z