Multiple deficiencies in antioxidant enzymes in mice result in a compound increase in sensitivity to oxidative stress
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Is the oxidative stress theory of aging dead?Oxidative stress and diabetes: what can we learn about insulin resistance from antioxidant mutant mouse models?Regulation of the intrinsic apoptosis pathway by reactive oxygen speciesExploring the electrostatic repulsion model in the role of Sirt3 in directing MnSOD acetylation status and enzymatic activityMitochondrial dysfunction and oxidative stress in Parkinson's diseaseVitamin E ameliorates the decremental effect of paraquat on cardiomyocyte contractility in ratsTranscriptome analysis of murine macrophages in response to infection with Streptococcus pyogenes reveals an unusual activation programLong-lived ames dwarf mice are resistant to chemical stressorsUpdate on the oxidative stress theory of aging: does oxidative stress play a role in aging or healthy aging?A drastic superoxide-dependent oxidative stress is prerequisite for the down-regulation of IRP1: Insights from studies on SOD1-deficient mice and macrophages treated with paraquat.Analyses of antioxidant status and nucleotide alterations in genes encoding antioxidant enzymes in patients with benign and malignant thyroid disordersAging and HIV/AIDS: pathogenetic role of therapeutic side effectsProteomic analysis reveals late exercise effects on cardiac remodeling following myocardial infarction.Somatic mtDNA mutations cause aging phenotypes without affecting reactive oxygen species productionTime dependent bladder apoptosis induced by acute bladder outlet obstruction and subsequent emptying is associated with decreased MnSOD expression and Bcl-2/Bax ratioMouse Models of Oxidative Stress Indicate a Role for Modulating Healthy Aging.Life and death: metabolic rate, membrane composition, and life span of animals.Acetylation of MnSOD directs enzymatic activity responding to cellular nutrient status or oxidative stress.Comparative effect of Piper betle, Chlorella vulgaris and tocotrienol-rich fraction on antioxidant enzymes activity in cellular ageing of human diploid fibroblastsIs there more to aging than mitochondrial DNA and reactive oxygen species?Glutathione peroxidase-1 in health and disease: from molecular mechanisms to therapeutic opportunitiesp66Shc aging protein in control of fibroblasts cell fateSOD2 contributes to anti-oxidative capacity in rabbit corneal endothelial cells.Mitochondrial medicine for aging and neurodegenerative diseasesToxin models of mitochondrial dysfunction in Parkinson's disease.Associations between functional polymorphisms in antioxidant defense genes and urinary oxidative stress biomarkers in healthy, premenopausal women.How genetic analysis tests theories of animal aging.CyclinB1/Cdk1 phosphorylates mitochondrial antioxidant MnSOD in cell adaptive response to radiation stress.Testing hypotheses of aging in long-lived mice of the genus Peromyscus: association between longevity and mitochondrial stress resistance, ROS detoxification pathways, and DNA repair efficiencyComparative and alternative approaches and novel animal models for aging research: introduction to special issue.Increased oxidative stress and antioxidant expression in mouse keratinocytes following exposure to paraquat.The Mn-superoxide dismutase single nucleotide polymorphism rs4880 and the glutathione peroxidase 1 single nucleotide polymorphism rs1050450 are associated with aging and longevity in the oldest old.The genomic response of the ipsilateral and contralateral cortex to stroke in aged rats.Overexpression of Twinkle-helicase protects cardiomyocytes from genotoxic stress caused by reactive oxygen speciesOverexpression of Mn superoxide dismutase does not increase life span in mice.Differential contribution of the mitochondrial respiratory chain complexes to reactive oxygen species production by redox cycling agents implicated in parkinsonism.Mice deficient in both Mn superoxide dismutase and glutathione peroxidase-1 have increased oxidative damage and a greater incidence of pathology but no reduction in longevity.Adaptation and sensitization to proteotoxic stress.A positive feedback loop promotes HIF-1α stability through miR-210-mediated suppression of RUNX3 in paraquat-induced EMT.Cardiac-specific overexpression of catalase attenuates paraquat-induced myocardial geometric and contractile alteration: role of ER stress.
P2860
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P2860
Multiple deficiencies in antioxidant enzymes in mice result in a compound increase in sensitivity to oxidative stress
description
2004 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2004
@ast
im Juni 2004 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2004/06/15)
@sk
vědecký článek publikovaný v roce 2004
@cs
wetenschappelijk artikel (gepubliceerd op 2004/06/15)
@nl
наукова стаття, опублікована в червні 2004
@uk
مقالة علمية (نشرت في 15-6-2004)
@ar
name
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@ast
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@en
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@nl
type
label
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@ast
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@en
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@nl
prefLabel
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@ast
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@en
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@nl
P2093
P1476
Multiple deficiencies in antio ...... ensitivity to oxidative stress
@en
P2093
Arlan Richardson
Charles J. Epstein
Gregory Freeman
Larry Estlack
Marian Sabia
Randy Strong
Ting-Ting Huang
P304
P356
10.1016/J.FREERADBIOMED.2004.03.016
P577
2004-06-15T00:00:00Z