APRIL-deficient mice have normal immune system development - Wikidata - awgldk - TriplyDB

APRIL-deficient mice have normal immune system development

APRIL-deficient mice have normal immune system development

http://www.wikidata.org/entity/Q28511017

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Atacicept: targeting B cells in multiple sclerosis The intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the games The current status of targeting BAFF/BLyS for autoimmune diseases Profile of atacicept and its potential in the treatment of systemic lupus erythematosus Historical perspectives on tumor necrosis factor and its superfamily: 25 years later, a golden journey Selective APRIL blockade delays systemic lupus erythematosus in mouse.APRIL mediates peritoneal B-1 cell homeostasis.BLyS and APRIL in rheumatoid arthritis.Similarities and differences between selective and nonselective BAFF blockade in murine SLE No evidence that soluble TACI induces signalling via membrane-expressed BAFF and APRIL in myeloid cells.Signaling by the tumor necrosis factor receptor superfamily in B-cell biology and disease The discovery and development of belimumab: the anti-BLyS-lupus connection Targeting B lymphocyte stimulator in systemic lupus erythematosus and other autoimmune rheumatic disorders.Dispensability of APRIL to the development of systemic lupus erythematosus in NZM 2328 mice.Nurselike cells express BAFF and APRIL, which can promote survival of chronic lymphocytic leukemia cells via a paracrine pathway distinct from that of SDF-1alpha Combined loss of the BH3-only proteins Bim and Bmf restores B-cell development and function in TACI-Ig transgenic mice The role of BAFF in immune function and implications for autoimmunity.BLyS antagonists and peptide tolerance induction.Requirement for BAFF and APRIL during B cell development in GALT B cells as a therapeutic target in autoimmune diseases.Serum BAFF and APRIL levels in patients with IgG4-related disease and their clinical significance.Inhibition of B cell activating factor (BAFF) in the management of systemic lupus erythematosus (SLE).The TNF family member APRIL promotes colorectal tumorigenesis Multiple signaling pathways promote B lymphocyte stimulator dependent B-cell growth and survival.An APRIL to remember: novel TNF ligands as therapeutic targets.APRIL secreted by neutrophils binds to heparan sulfate proteoglycans to create plasma cell niches in human mucosa.Clinical targeting of the TNF and TNFR superfamilies Apoptosis in the development of the immune system.Raised intrathecal levels of APRIL and BAFF in patients with systemic lupus erythematosus: relationship to neuropsychiatric symptoms.APRIL and BCMA promote human multiple myeloma growth and immunosuppression in the bone marrow microenvironment.The role of B lymphocyte stimulator (BLyS) in systemic lupus erythematosus.Synergisic effect of APRIL knockdown and Jiedu Xiaozheng Yin, a Chinese medicinal recipe, on the inhibition of hepatocellular carcinoma cell proliferation.Functions of Runx in IgA class switch recombination.Induction of gut IgA production through T cell-dependent and T cell-independent pathways.Biologic differences between various inhibitors of the BLyS/BAFF pathway: should we expect differences between belimumab and other inhibitors in development?Roles for BLyS family members in meeting the distinct homeostatic demands of innate and adaptive B cells.The role of B lymphocyte stimulator in B cell biology: implications for the treatment of lupus.Therapeutic targeting of the BAFF/APRIL axis in systemic lupus erythematosus.Mutation of the BAFF furin cleavage site impairs B-cell homeostasis and antibody responses.BAFF receptor and TACI in B-1b cell maintenance and antibacterial responses.

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APRIL-deficient mice have normal immune system development

http://www.wikidata.org/entity/Q28511017

description

2004 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի փետրվարին հրատարակված գիտական հոդված

@hy

artículu científicu espublizáu en 2004

@ast

im Februar 2004 veröffentlichter wissenschaftlicher Artikel

@de

scientific journal article

@en

vedecký článok (publikovaný 2004/02/01)

@sk

vědecký článek publikovaný v roce 2004

@cs

wetenschappelijk artikel (gepubliceerd op 2004/02/01)

@nl

наукова стаття, опублікована в лютому 2004

@uk

مقالة علمية (نشرت في فبراير 2004)

@ar

name

APRIL-deficient mice have normal immune system development

@ast

APRIL-deficient mice have normal immune system development

@en

APRIL-deficient mice have normal immune system development

@nl

type

label

APRIL-deficient mice have normal immune system development

@ast

APRIL-deficient mice have normal immune system development

@en

APRIL-deficient mice have normal immune system development

@nl

prefLabel

APRIL-deficient mice have normal immune system development

@ast

APRIL-deficient mice have normal immune system development

@en

APRIL-deficient mice have normal immune system development

@nl

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P356

MCB.24.3.997-1006.2004

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Molecular and Cellular Biology

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APRIL-deficient mice have normal immune system development

@en

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Avi Ashkenazi

http://www.w3.org/2001/XMLSchema#string

Christine Olsson

http://www.w3.org/2001/XMLSchema#string

David Lawrence

http://www.w3.org/2001/XMLSchema#string

Dhaya Seshasayee

http://www.w3.org/2001/XMLSchema#string

Dorothy French

http://www.w3.org/2001/XMLSchema#string

Eugene Varfolomeev

http://www.w3.org/2001/XMLSchema#string

Flavius Martin

http://www.w3.org/2001/XMLSchema#string

Frank Kischkel

http://www.w3.org/2001/XMLSchema#string

Hua Wang

http://www.w3.org/2001/XMLSchema#string

Iqbal S. Grewal

http://www.w3.org/2001/XMLSchema#string

P2860

BAFF, a novel ligand of the tumor necrosis factor family, stimulates B cell growth

TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease

TACI is a TRAF-interacting receptor for TALL-1, a tumor necrosis factor family member involved in B cell regulation

Tumor necrosis factor (TNF) receptor superfamily member TACI is a high affinity receptor for TNF family members APRIL and BLyS

Identification of a ligand for the death-domain-containing receptor Apo3

NF-AT activation induced by a CAML-interacting member of the tumor necrosis factor receptor superfamily

APRIL, a new ligand of the tumor necrosis factor family, stimulates tumor cell growth

Biologically active APRIL is secreted following intracellular processing in the Golgi apparatus by furin convertase

An endogenous hybrid mRNA encodes TWE-PRIL, a functional cell surface TWEAK-APRIL fusion protein

APRIL modulates B and T cell immunity

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997–1006

http://www.w3.org/2001/XMLSchema#string

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scholarly article

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10.1128/MCB.24.3.997-1006.2004

http://www.w3.org/2001/XMLSchema#string

P407

P433

3

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24

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2004-02-01T00:00:00Z

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14729948

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321448

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