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Deciphering Subtype-Selective Modulations in TRPA1 Biosensor ChannelsTRPA1 as a drug target--promise and challengesDiabetic neuropathic pain: Physiopathology and treatmentTRP Channels in Skin Biology and PathophysiologyAcute cardiopulmonary toxicity of inhaled aldehydes: role of TRPA1New Horizons in Diabetic Neuropathy: Mechanisms, Bioenergetics, and Pain.Painful Diabetic Neuropathy: Prevention or Suppression?Glo1 inhibitors for neuropsychiatric and anti-epileptic drug developmentAre sensory TRP channels biological alarms for lipid peroxidation?Methylglyoxal, the dark side of glycolysisPioglitazone rapidly reduces neuropathic pain through astrocyte and nongenomic PPARγ mechanismsStreptozotocin Stimulates the Ion Channel TRPA1 Directly: INVOLVEMENT OF PEROXYNITRITEPathogenesis of painful diabetic neuropathy.The role of sodium channels in painful diabetic and idiopathic neuropathy.ThermoTRPs and Pain.The role of methylglyoxal and the glyoxalase system in diabetes and other age-related diseases.Transient receptor potential ankyrin 1 (TRPA1) antagonists.Effects of methylglyoxal on human cardiac fibroblast: roles of transient receptor potential ankyrin 1 (TRPA1) channels.Nociceptor plasticity: A closer look.Transient receptor potential A1 channels regulate epithelial cell barriers formed by MDCK cells.A Caenorhabditis elegans Model Elucidates a Conserved Role for TRPA1-Nrf Signaling in Reactive α-Dicarbonyl Detoxification.Methylglyoxal Requires AC1 and TRPA1 to Produce Pain and Spinal Neuron Activation.Long-lasting antinociceptive effects of green light in acute and chronic pain in rats.Targeting nociceptive transient receptor potential channels to treat chronic pain: current state of the field.Regulation of Pain and Itch by TRP Channels.TRPC proteins contribute to development of diabetic retinopathy and regulate glyoxalase 1 activity and methylglyoxal accumulation.Impact of intensive treatment on serum methylglyoxal levels among individuals with screen-detected type 2 diabetes: the ADDITION-Denmark study.Accumulation of methylglyoxal increases the advanced glycation end-product levels in DRG and contributes to lumbar disk herniation-induced persistent pain.Methylglyoxal Disrupts Paranodal Axoglial Junctions via Calpain Activation.Reducing CXCR4-mediated nociceptor hyperexcitability reverses painful diabetic neuropathy.
P2860
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P2860
description
2013 nî lūn-bûn
@nan
2013 թուականին հրատարակուած գիտական յօդուած
@hyw
2013 թվականին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Methylglyoxal evokes pain by stimulating TRPA1
@ast
Methylglyoxal evokes pain by stimulating TRPA1
@en
Methylglyoxal evokes pain by stimulating TRPA1
@nl
type
label
Methylglyoxal evokes pain by stimulating TRPA1
@ast
Methylglyoxal evokes pain by stimulating TRPA1
@en
Methylglyoxal evokes pain by stimulating TRPA1
@nl
prefLabel
Methylglyoxal evokes pain by stimulating TRPA1
@ast
Methylglyoxal evokes pain by stimulating TRPA1
@en
Methylglyoxal evokes pain by stimulating TRPA1
@nl
P2093
P2860
P50
P1433
P1476
Methylglyoxal evokes pain by stimulating TRPA1
@en
P2093
Angelika Bierhaus
Clive Gentry
Emily Light
Julie Vallortigara
Thomas Fleming
P2860
P304
P356
10.1371/JOURNAL.PONE.0077986
P407
P577
2013-01-01T00:00:00Z