Connective tissue growth factor overexpression in cardiomyocytes promotes cardiac hypertrophy and protection against pressure overload
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Transient receptor potential (TRP) channels and cardiac fibrosisConnective tissue growth factor (CCN2, CTGF) and organ fibrosis: lessons from transgenic animalsCardiac endothelium-myocyte interaction: clinical opportunities for new heart failure therapies regardless of ejection fractionRoles for CCN2 in normal physiological processesTaking aim at the extracellular matrix: CCN proteins as emerging therapeutic targetsEffects of stretch and shortening on gene expression in intact myocardium.A mouse strain where basal connective tissue growth factor gene expression can be switched from low to high.Hematopoietic Id Deletion Triggers Endomyocardial Fibrotic and Vascular Defects in the Adult Heart.The Notch pathway controls fibrotic and regenerative repair in the adult heart.Augmented cardiac hypertrophy in response to pressure overload in mice lacking ELTD1.Thyroid Hormone-Regulated Cardiac microRNAs are Predicted to Suppress Pathological Hypertrophic Signaling.Myocardial connective tissue growth factor (CCN2/CTGF) attenuates left ventricular remodeling after myocardial infarction.Desmoglein 2 mutant mice develop cardiac fibrosis and dilation.Long-term in vivo resistin overexpression induces myocardial dysfunction and remodeling in rats.Matricellular proteins in cardiac adaptation and disease.Brain derived neurotrophic factor contributes to the cardiogenic potential of adult resident progenitor cells in failing murine heart.Genetic Analysis of Connective Tissue Growth Factor as an Effector of Transforming Growth Factor β Signaling and Cardiac Remodeling.Simvastatin modulates remodeling of Kv4.3 expression in rat hypertrophied cardiomyocytes.CTGF/CCN2 Postconditioning Increases Tolerance of Murine Hearts towards Ischemia-Reperfusion Injury.Global transcriptome analysis of human bone marrow stromal cells (BMSC) reveals proliferative, mobile and interactive cells that produce abundant extracellular matrix proteins, some of which may affect BMSC potency.Angiotensin II-mediated up-regulation of connective tissue growth factor promotes atrial tissue fibrosis in the canine atrial fibrillation model.Connective tissue growth factor regulates cardiac function and tissue remodeling in a mouse model of dilated cardiomyopathyMatrix metalloproteinase-9 deletion attenuates myocardial fibrosis and diastolic dysfunction in ageing miceCCN2 exerts direct cytoprotective actions in adult cardiac myocytes by activation of the PI3-kinase/Akt/GSK-3β signaling pathway.Muscle ring finger 1 and muscle ring finger 2 are necessary but functionally redundant during developmental cardiac growth and regulate E2F1-mediated gene expression in vivo.Circulating Biomarkers Predictive of Postoperative Atrial Fibrillation.N-Acetylcysteine effects on transforming growth factor-β and tumor necrosis factor-α serum levels as pro-fibrotic and inflammatory biomarkers in patients following ST-segment elevation myocardial infarction.Secondhand Smoke Exposure Enhances Cardiac Fibrosis Effects on the Aging Rat Hearts.ERK1/2 directly acts on CTGF/CCN2 expression to mediate myocardial fibrosis in cardiomyopathy caused by mutations in the lamin A/C geneMolecular control of vascular development by the matricellular proteins CCN1 (Cyr61) and CCN2 (CTGF).Molecular mechanisms that control interstitial fibrosis in the pressure-overloaded heart.Transforming growth factor (TGF)-β signaling in cardiac remodeling.Extracellular matrix alterations in the atria: insights into the mechanisms and perpetuation of atrial fibrillation.Cellular Interplay between Cardiomyocytes and Nonmyocytes in Cardiac Remodeling.The Aging HeartMicroRNA-18 and microRNA-19 regulate CTGF and TSP-1 expression in age-related heart failure.Transforming growth factor β receptor inhibition prevents ventricular fibrosis in a mouse model of progressive cardiac conduction disease.Relevance of mouse models of cardiac fibrosis and hypertrophy in cardiac research.The role of transforming growth factor (TGF)-β in the infarcted myocardium.Unraveling the complexities of cardiac remodeling and hypertrophy - high-content screening and computational modeling
P2860
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P2860
Connective tissue growth factor overexpression in cardiomyocytes promotes cardiac hypertrophy and protection against pressure overload
description
2009 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
article publié dans la revue scientifique PLoS ONE
@fr
artículu científicu espublizáu en 2009
@ast
im August 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/08/25)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/08/25)
@nl
наукова стаття, опублікована в серпні 2009
@uk
name
Connective tissue growth facto ...... tion against pressure overload
@ast
Connective tissue growth facto ...... tion against pressure overload
@en
Connective tissue growth facto ...... tion against pressure overload
@nl
type
label
Connective tissue growth facto ...... tion against pressure overload
@ast
Connective tissue growth facto ...... tion against pressure overload
@en
Connective tissue growth facto ...... tion against pressure overload
@nl
prefLabel
Connective tissue growth facto ...... tion against pressure overload
@ast
Connective tissue growth facto ...... tion against pressure overload
@en
Connective tissue growth facto ...... tion against pressure overload
@nl
P2093
P2860
P1433
P1476
Connective tissue growth facto ...... tion against pressure overload
@en
P2093
Andreas Perrot
Anna N. Panek
Bettina Erdmann
Cemil Ozcelik
Christian Geier
Ingo Morano
Maximilian G. Posch
Natalia Alenina
Rainer Dietz Ingo Morano
Rainer Dietz
P2860
P356
10.1371/JOURNAL.PONE.0006743
P407
P577
2009-08-25T00:00:00Z