Catechol-O-methyltransferase inhibition increases pain sensitivity through activation of both beta2- and beta3-adrenergic receptors - Wikidata - awgldk - TriplyDB

Catechol-O-methyltransferase inhibition increases pain sensitivity through activation of both beta2- and beta3-adrenergic receptors

Catechol-O-methyltransferase inhibition increases pain sensitivity through activation of both beta2- and beta3-adrenergic receptors

http://www.wikidata.org/entity/Q28578391

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Pain genes Catechol-O-methyltransferase inhibition alters pain and anxiety-related volitional behaviors through activation of β-adrenergic receptors in the rat Comt1 genotype and expression predicts anxiety and nociceptive sensitivity in inbred strains of mice The val158met polymorphism of human catechol-O-methyltransferase (COMT) affects anterior cingulate cortex activation in response to painful laser stimulation.Dysregulation of the autonomous nervous system in patients with temporomandibular disorder: a pupillometric study.Low enzymatic activity haplotypes of the human catechol-O-methyltransferase gene: enrichment for marker SNPs.Beta2-adrenoceptor agonists alleviate neuropathic allodynia in mice after chronic treatment.Genetic variation in the beta-2 adrenergic receptor (ADRB2) predicts functional gastrointestinal diagnoses and poorer health-related quality of life Pathophysiology of fibromyalgia.The ACTTION-APS-AAPM Pain Taxonomy (AAAPT) Multidimensional Approach to Classifying Acute Pain Conditions.Chronic CRF1 receptor blockade reduces heroin intake escalation and dependence-induced hyperalgesia In search of analgesia: emerging roles of GPCRs in pain.Epigenetic modification of DRG neuronal gene expression subsequent to nerve injury: etiological contribution to complex regional pain syndromes (Part I).β2- and β3-adrenergic receptors drive COMT-dependent pain by increasing production of nitric oxide and cytokines Effect of catechol-O-methyltransferase polymorphism on response to propranolol therapy in chronic musculoskeletal pain: a randomized, double-blind, placebo-controlled, crossover pilot study.A hotspot of inactivation: The A22S and V108M polymorphisms individually destabilize the active site structure of catechol O-methyltransferase.Potential autonomic risk factors for chronic TMD: descriptive data and empirically identified domains from the OPPERA case-control study Genetics of pain perception, COMT and postoperative pain management in children.Catechol O-methyltransferase haplotype predicts immediate musculoskeletal neck pain and psychological symptoms after motor vehicle collision.Pharmacogenomic considerations in opioid analgesia.Results of a pilot multicenter genotype-based randomized placebo-controlled trial of propranolol to reduce pain after major thermal burn injury Genetics and Gene Expression Involving Stress and Distress Pathways in Fibromyalgia with and without Comorbid Chronic Fatigue Syndrome.The potential contribution of stress systems to the transition to chronic whiplash-associated disorders.Injury-Dependent and Disability-Specific Lumbar Spinal Gene Regulation following Sciatic Nerve Injury in the Rat Translating the human genome to manage pediatric postoperative pain.Molecular genetic mechanisms of allelic specific regulation of murine Comt expression COMT gene locus: new functional variants Catechol-O-methyltransferase genotype predicts pain severity in hospitalized burn patients.COMT Diplotype Amplifies Effect of Stress on Risk of Temporomandibular Pain.Anti-inflammatory and antinociceptive effects of salbutamol on acute and chronic models of inflammation in rats: involvement of an antioxidant mechanism.Human pain and genetics: some basics Nuclear factor-kappa B regulates pain and COMT expression in a rodent model of inflammation.Serotonin-induced hypersensitivity via inhibition of catechol O-methyltransferase activity Adding Genetic Testing to Evidence-Based Guidelines to Determine the Safest and Most Effective Chronic Pain Treatment for Injured Workers Modification of COMT-dependent pain sensitivity by psychological stress and sex.Biopsychosocial influence on shoulder pain: risk subgroups translated across preclinical and clinical prospective cohorts.Chronic pain after lower abdominal surgery: do catechol-O-methyl transferase/opioid receptor μ-1 polymorphisms contribute?Stress induces a switch of intracellular signaling in sensory neurons in a model of generalized pain Pharmacogenetics of chronic pain and its treatment.Janus molecule I: dichotomous effects of COMT in neuropathic vs nociceptive pain modalities.

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Catechol-O-methyltransferase inhibition increases pain sensitivity through activation of both beta2- and beta3-adrenergic receptors

http://www.wikidata.org/entity/Q28578391

description

2007 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2007 թվականի ապրիլին հրատարակված գիտական հոդված

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artículu científicu espublizáu en 2007

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im November 2006 veröffentlichter wissenschaftlicher Artikel

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scientific journal article

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vedecký článok (publikovaný 2007/04/01)

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vědecký článek publikovaný v roce 2007

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wetenschappelijk artikel (gepubliceerd op 2007/04/01)

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наукова стаття, опублікована у квітні 2007

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مقالة علمية (نشرت في أبريل 2007)

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name

Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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J.PAIN.2006.09.022

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Catechol-O-methyltransferase i ...... and beta3-adrenergic receptors

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Andrea Gail Nackley

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Karamarie Fecho

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Luda Diatchenko

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Patrick Flood

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William Maixner

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cDNA for the human beta 2-adrenergic receptor: a protein with multiple membrane-spanning domains and encoded by a gene whose chromosomal location is shared with that of the receptor for platelet-derived growth factor

MAPK-dependent regulation of IL-1- and beta-adrenoreceptor-induced inflammatory cytokine production from mast cells: implications for the stress response

Comparison of the effects of antidepressants and their metabolites on reuptake of biogenic amines and on receptor binding

Quantitative assessment of tactile allodynia in the rat paw

Ethical guidelines for investigations of experimental pain in conscious animals

A new and sensitive method for measuring thermal nociception in cutaneous hyperalgesia

Direct binding of activated c-Src to the beta 3-adrenergic receptor is required for MAP kinase activation.

Effect of drugs used for neuropathic pain management on tetrodotoxin-resistant Na(+) currents in rat sensory neurons.

The beta3-adrenergic receptor activates mitogen-activated protein kinase in adipocytes through a Gi-dependent mechanism.

Catechol-O-methyltransferase (COMT): biochemistry, molecular biology, pharmacology, and clinical efficacy of the new selective COMT inhibitors.

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