Over-expression of antioxidant enzymes protects cultured hippocampal and cortical neurons from necrotic insults
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Rosuvastatin induces delayed preconditioning against L-glutamate excitotoxicity in cultured cortical neuronsChronic Oxidative Stress, Mitochondrial Dysfunction, Nrf2 Activation and Inflammation in the Hippocampus Accompany Heightened Systemic Inflammation and Oxidative Stress in an Animal Model of Gulf War IllnessInhibition of activated NR2B gene- and caspase-3 protein-expression by glutathione following traumatic brain injury in a rat model.A1 adenosine receptors play an essential role in protecting the embryo against hypoxia.HSV-1-based amplicon vectors: design and applications.HSV-1 amplicon vectors: a promising and versatile tool for gene delivery.Glutathione metabolism and Parkinson's disease.The mitochondrial impairment, oxidative stress and neurodegeneration connection: reality or just an attractive hypothesis?Neuroprotective effect of adenoviral catalase gene transfer in cortical neuronal cultures.Genetic and pharmacologic manipulation of oxidative stress after neonatal hypoxia-ischemia.Going ape as an approach to cancer therapeutics.Therapeutic potential of targeting hydrogen peroxide metabolism in the treatment of brain ischaemia.Prohibitin reduces mitochondrial free radical production and protects brain cells from different injury modalities.Suppression of reactive oxygen species production enhances neuronal survival in vitro and in vivo in the anoxia-tolerant turtle Trachemys scripta.Evidence for caspase effects on release of cytochrome c and AIF in a model of ischemia in cortical neurons.Gene therapy using SOD1 protects striatal neurons from experimental stroke.The protective role of catalase against cerebral ischemia in vitro and in vivo.Glutamate-mediated influx of extracellular Ca2+ is coupled with reactive oxygen species generation in cultured hippocampal neurons but not in astrocytes.The effect of nuclear factor erythroid 2-related factor/antioxidant response element signalling pathway in the lanthanum chloride-induced impairment of learning and memory in rats.Hypoxic preconditioning reverses protection after neonatal hypoxia-ischemia in glutathione peroxidase transgenic murine brain.Diphenyl diselenide protects neuronal cells against oxidative stress and mitochondrial dysfunction: Involvement of the glutathione-dependent antioxidant systemPrevention of progression in Parkinson's disease
P2860
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P2860
Over-expression of antioxidant enzymes protects cultured hippocampal and cortical neurons from necrotic insults
description
2003 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
scientific journal article
@en
vedecký článok (publikovaný 2003/12/01)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/12/01)
@nl
наукова стаття, опублікована в грудні 2003
@uk
مقالة علمية (نشرت في ديسمبر 2003)
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name
Over-expression of antioxidant ...... neurons from necrotic insults
@ast
Over-expression of antioxidant ...... neurons from necrotic insults
@en
Over-expression of antioxidant ...... neurons from necrotic insults
@nl
type
label
Over-expression of antioxidant ...... neurons from necrotic insults
@ast
Over-expression of antioxidant ...... neurons from necrotic insults
@en
Over-expression of antioxidant ...... neurons from necrotic insults
@nl
prefLabel
Over-expression of antioxidant ...... neurons from necrotic insults
@ast
Over-expression of antioxidant ...... neurons from necrotic insults
@en
Over-expression of antioxidant ...... neurons from necrotic insults
@nl
P2093
P2860
P1476
Over-expression of antioxidant ...... neurons from necrotic insults
@en
P2093
Elise Cheng
Pearl Chang
Robert Sapolsky
Sheila Brooke
P2860
P304
P356
10.1046/J.1471-4159.2003.02123.X
P407
P577
2003-12-01T00:00:00Z