Mouse HORMAD1 and HORMAD2, two conserved meiotic chromosomal proteins, are depleted from synapsed chromosome axes with the help of TRIP13 AAA-ATPase
about
Identification of novel mitosis regulators through data mining with human centromere/kinetochore proteins as group queriesPolo-like kinase is required for synaptonemal complex disassembly and phosphorylation in mouse spermatocytesShugoshin is a Mad1/Cdc20-like interactor of Mad2RAD21L, a novel cohesin subunit implicated in linking homologous chromosomes in mammalian meiosisGene expression in the fetal mouse ovary is altered by exposure to low doses of bisphenol AEctopic Expression of Testis Germ Cell Proteins in Cancer and Its Potential Role in Genomic InstabilityThe multifaceted roles of the HORMA domain in cellular signalingGenetic studies of IgA nephropathy: what have we learned from genome-wide association studiesDouble-strand break repair on sex chromosomes: challenges during male meiotic prophaseTRIP13PCH-2 promotes Mad2 localization to unattached kinetochores in the spindle checkpoint responseMeiosis-specific cohesin component, Stag3 is essential for maintaining centromere chromatid cohesion, and required for DNA repair and synapsis between homologous chromosomesSPO11-independent DNA repair foci and their role in meiotic silencingHAL-2 promotes homologous pairing during Caenorhabditis elegans meiosis by antagonizing inhibitory effects of synaptonemal complex precursorsHormad1 mutation disrupts synaptonemal complex formation, recombination, and chromosome segregation in mammalian meiosisPch2 is a hexameric ring ATPase that remodels the chromosome axis protein Hop1Pch2 acts through Xrs2 and Tel1/ATM to modulate interhomolog bias and checkpoint function during meiosisImplementation of meiosis prophase I programme requires a conserved retinoid-independent stabilizer of meiotic transcriptsRecombination, Pairing, and Synapsis of Homologs during MeiosisThe cohesion protein SOLO associates with SMC1 and is required for synapsis, recombination, homolog bias and cohesion and pairing of centromeres in Drosophila MeiosisMeiotic DNA double-strand breaks and chromosome asynapsis in mice are monitored by distinct HORMAD2-independent and -dependent mechanismsFunctional conservation of Mei4 for meiotic DNA double-strand break formation from yeasts to miceMEIOB targets single-strand DNA and is necessary for meiotic recombinationMeiotic DNA break formation requires the unsynapsed chromosome axis-binding protein IHO1 (CCDC36) in miceHORMAD2 is essential for synapsis surveillance during meiotic prophase via the recruitment of ATR activityMouse TRIP13/PCH2 is required for recombination and normal higher-order chromosome structure during meiosisMeiotic homologue alignment and its quality surveillance are controlled by mouse HORMAD1.HORMAD1-dependent checkpoint/surveillance mechanism eliminates asynaptic oocytesHomologue engagement controls meiotic DNA break number and distributionAlignment of Homologous Chromosomes and Effective Repair of Programmed DNA Double-Strand Breaks during Mouse Meiosis Require the Minichromosome Maintenance Domain Containing 2 (MCMDC2) ProteinThe CSN/COP9 signalosome regulates synaptonemal complex assembly during meiotic prophase I of Caenorhabditis elegans.Coordinating cohesion, co-orientation, and congression during meiosis: lessons from holocentric chromosomesPolo-like kinase-dependent phosphorylation of the synaptonemal complex protein SYP-4 regulates double-strand break formation through a negative feedback loop.Chromosome axis defects induce a checkpoint-mediated delay and interchromosomal effect on crossing over during Drosophila meiosis.Modulating Crossover Frequency and Interference for Obligate Crossovers in Saccharomyces cerevisiae Meiosis.BRCA1 establishes DNA damage signaling and pericentric heterochromatin of the X chromosome in male meiosis.Superresolution microscopy reveals the three-dimensional organization of meiotic chromosome axes in intact Caenorhabditis elegans tissue.Mouse HORMAD1 is a meiosis i checkpoint protein that modulates DNA double- strand break repair during female meiosis.Genetic study of Hormad1 and Hormad2 with non-obstructive azoospermia patients in the male Chinese population.TRIP13 promotes error-prone nonhomologous end joining and induces chemoresistance in head and neck cancerDisassembly of mitotic checkpoint complexes by the joint action of the AAA-ATPase TRIP13 and p31(comet).
P2860
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P2860
Mouse HORMAD1 and HORMAD2, two conserved meiotic chromosomal proteins, are depleted from synapsed chromosome axes with the help of TRIP13 AAA-ATPase
description
2009 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2009 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2009
@ast
im Oktober 2009 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2009/10/01)
@sk
vědecký článek publikovaný v roce 2009
@cs
wetenschappelijk artikel (gepubliceerd op 2009/10/01)
@nl
наукова стаття, опублікована в жовтні 2009
@uk
مقالة علمية (نشرت في أكتوبر 2009)
@ar
name
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@ast
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@en
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@nl
type
label
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@ast
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@en
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@nl
prefLabel
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@ast
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@en
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@nl
P2093
P2860
P3181
P1433
P1476
Mouse HORMAD1 and HORMAD2, two ...... the help of TRIP13 AAA-ATPase
@en
P2093
Attila Toth
Christian R. Eckmann
Ewelina Bolcun-Filas
Howard J. Cooke
Huiling Xu
Ignasi Roig
Katrin Daniel
Lukasz Wojtasz
Michael J. McKay
Verawan Boonsanay
P2860
P304
P3181
P356
10.1371/JOURNAL.PGEN.1000702
P577
2009-10-01T00:00:00Z