GLIS3, a novel member of the GLIS subfamily of Krüppel-like zinc finger proteins with repressor and activation functions
about
Genome-wide association study identifies five new susceptibility loci for primary angle closure glaucomaThe Krüppel-like zinc finger protein Glis2 functions as a negative modulator of the Wnt/beta-catenin signaling pathwayTIP27: a novel repressor of the nuclear orphan receptor TAK1/TR4Regulatory role for Krüppel-like zinc-finger protein Gli-similar 1 (Glis1) in PMA-treated and psoriatic epidermis.Gli-similar (Glis) Krüppel-like zinc finger proteins: insights into their physiological functions and critical roles in neonatal diabetes and cystic renal diseaseKrüppel-like zinc finger protein Gli-similar 2 (Glis2) represses transcription through interaction with C-terminal binding protein 1 (CtBP1).New genetic loci implicated in fasting glucose homeostasis and their impact on type 2 diabetes riskThe Krüppel-like zinc finger protein Glis3 directly and indirectly activates insulin gene transcriptionKruppel-like zinc finger protein Glis2 is essential for the maintenance of normal renal functionsIdentification of nuclear localization, DNA binding, and transactivating mechanisms of Kruppel-like zinc finger protein Gli-similar 2 (Glis2)Polycystic kidney disease in the medaka (Oryzias latipes) pc mutant caused by a mutation in the Gli-Similar3 (glis3) gene.TRANSCRIPTION FACTOR GLI-SIMILAR 3 (GLIS3): IMPLICATIONS FOR THE DEVELOPMENT OF CONGENITAL HYPOTHYROIDISM.Zfra is a small wizard in the mitochondrial apoptosisModulation of the transactivation function and stability of Krüppel-like zinc finger protein Gli-similar 3 (Glis3) by Suppressor of Fused.The Krüppel-like zinc finger protein GLIS3 transactivates neurogenin 3 for proper fetal pancreatic islet differentiation in mice.4D MRI of polycystic kidneys from rapamycin-treated Glis3-deficient miceHECT E3 Ubiquitin Ligase Itch Functions as a Novel Negative Regulator of Gli-Similar 3 (Glis3) Transcriptional Activity.The Spatiotemporal Pattern of Glis3 Expression Indicates a Regulatory Function in Bipotent and Endocrine Progenitors during Early Pancreatic Development and in Beta, PP and Ductal CellsExpanding the Clinical Spectrum Associated With GLIS3 Mutations.Extended clinical features associated with novel Glis3 mutation: a case report.Glis3 regulates neurogenin 3 expression in pancreatic β-cells and interacts with its activator, Hnf6.Functional analysis of the zinc finger and activation domains of Glis3 and mutant Glis3(NDH1)Sustained expression of the transcription factor GLIS3 is required for normal beta cell function in adults.Systematic evaluation of validated type 2 diabetes and glycaemic trait loci for association with insulin clearanceMonogenic Diabetes: What It Teaches Us on the Common Forms of Type 1 and Type 2 Diabetes.Addiction genetics and pleiotropic effects of common haplotypes that make polygenic contributions to vulnerability to substance dependence.Glis3 is associated with primary cilia and Wwtr1/TAZ and implicated in polycystic kidney disease.Gli-similar proteins: their mechanisms of action, physiological functions, and roles in disease.Molecular genetics of addiction and related heritable phenotypes: genome-wide association approaches identify "connectivity constellation" and drug target genes with pleiotropic effects.Transcription factor Glis3, a novel critical player in the regulation of pancreatic beta-cell development and insulin gene expression.Four pediatric patients with autosomal recessive polycystic kidney disease developed new-onset diabetes after renal transplantation.The Potential Role of Krüppel-Like Zinc-Finger Protein Glis3 in Genetic Diseases and Cancers.Identifying and targeting determinants of melanoma cellular invasion.Transcription Factor GLIS3: A New and Critical Regulator of Postnatal Stages of Mouse Spermatogenesis.Expression Profile of Sonic Hedgehog Pathway Members in the Developing Human Fetal Brain.A murine model of neonatal diabetes mellitus in Glis3-deficient mice.Glis family proteins are differentially implicated in the cellular reprogramming of human somatic cells.GLIS1-3: emerging roles in reprogramming, stem and progenitor cell differentiation and maintenance.Mutations in GLIS3 are responsible for a rare syndrome with neonatal diabetes mellitus and congenital hypothyroidism.GLIS3 rs7020673 and rs10758593 polymorphisms interact in the susceptibility for type 1 diabetes mellitus.
P2860
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P2860
GLIS3, a novel member of the GLIS subfamily of Krüppel-like zinc finger proteins with repressor and activation functions
description
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
im Oktober 2003 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2003/10/01)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd op 2003/10/01)
@nl
наукова стаття, опублікована в жовтні 2003
@uk
مقالة علمية (نشرت في أكتوبر 2003)
@ar
name
GLIS3, a novel member of the G ...... essor and activation functions
@ast
GLIS3, a novel member of the G ...... essor and activation functions
@en
GLIS3, a novel member of the G ...... essor and activation functions
@nl
type
label
GLIS3, a novel member of the G ...... essor and activation functions
@ast
GLIS3, a novel member of the G ...... essor and activation functions
@en
GLIS3, a novel member of the G ...... essor and activation functions
@nl
prefLabel
GLIS3, a novel member of the G ...... essor and activation functions
@ast
GLIS3, a novel member of the G ...... essor and activation functions
@en
GLIS3, a novel member of the G ...... essor and activation functions
@nl
P2093
P2860
P3181
P356
P1476
GLIS3, a novel member of the G ...... essor and activation functions
@en
P2093
Anton M. Jetten
Gen Nakanishi
Mark Lewandoski
Yong-Sik Kim
P2860
P304
P3181
P356
10.1093/NAR/GKG776
P407
P577
2003-10-01T00:00:00Z