about
Nuclear factor-kappaB motif and interferon-alpha-stimulated response element co-operate in the activation of guanylate-binding protein-1 expression by inflammatory cytokines in endothelial cellsThe helical domain of GBP-1 mediates the inhibition of endothelial cell proliferation by inflammatory cytokines.Human guanylate binding protein-1 is a secreted GTPase present in increased concentrations in the cerebrospinal fluid of patients with bacterial meningitisThe guanylate binding protein-1 GTPase controls the invasive and angiogenic capability of endothelial cells through inhibition of MMP-1 expressionGuanylate-binding protein-1 expression is selectively induced by inflammatory cytokines and is an activation marker of endothelial cells during inflammatory diseasesEGF receptor is required for KRAS-induced pancreatic tumorigenesisMembranous CD24 drives the epithelial phenotype of pancreatic cancer.Notch signaling is required for exocrine regeneration after acute pancreatitis.Oncogenic KRas-induced Increase in Fluid-phase Endocytosis is Dependent on N-WASP and is Required for the Formation of Pancreatic Preneoplastic LesionsEarly requirement of Rac1 in a mouse model of pancreatic cancer.Context-Dependent Epigenetic Regulation of Nuclear Factor of Activated T Cells 1 in Pancreatic Plasticity.
P50
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P50
description
hulumtuese
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researcher
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ricercatrice
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wetenschapper
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հետազոտող
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name
Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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type
label
Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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prefLabel
Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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Clara Lubeseder-Martellato
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P214
P1053
A-8659-2017
P106
P21
P213
0000 0000 1553 7506
P214
P31
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0000-0002-8360-5800
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P7859
viaf-10895448